Xu Ming-Yi相关文献与解析，生知库 | 链接生命科学的每一步

Mertk(+) Liver Sinusoidal Endothelial Cells Negatively Regulate PINK1 Related Mitophagy and Accelerate MASH

Mertk(+)肝窦内皮细胞负调控PINK1相关线粒体自噬并加速MASH

期刊:Immunity Inflammation and Disease

影响因子:2.7

doi:10.1002/iid3.70256

Gao, Yu-Xuan; Weng, Zhong; Tang, Long; Xu, Ming-Yi; Luo, Sheng-Zheng

Single-cell transcriptome reveals a novel mechanism of C-Kit(+)-liver sinusoidal endothelial cells in NASH

单细胞转录组揭示了C-Kit(+)肝窦内皮细胞在非酒精性脂肪性肝炎中的一种新机制

期刊:Cell and Bioscience

影响因子:6.2

doi:10.1186/s13578-024-01215-7

Li, Hui-Yi; Gao, Yu-Xuan; Wu, Jun-Cheng; Li, Jing-Ze; Fu, Seng-Wang; Xu, Ming-Yi

Causal relationship between gut microbiota and risk of esophageal cancer: evidence from Mendelian randomization study

肠道菌群与食管癌风险之间的因果关系：来自孟德尔随机化研究的证据

期刊:Aging-Us

影响因子:3.9

doi:10.18632/aging.205547

Wang, Kui; Wang, Jiawei; Chen, Yuhua; Long, Huan; Pan, Wei; Liu, Yunfei; Xu, Ming-Yi; Guo, Qiang

Correction to: Linc-SCRG1 accelerates progression of hepatocellular carcinoma as a ceRNA of miR26a to derepress SKP2

更正：Linc-SCRG1作为miR26a的ceRNA，通过解除SKP2的抑制作用，加速肝细胞癌的进展。

期刊:Journal of Experimental & Clinical Cancer Research

影响因子:12.8

doi:10.1186/s13046-021-01975-x

Hu, Jun-Jie; Zhou, Cui; Luo, Xin; Luo, Sheng-Zheng; Li, Zheng-Hong; Xu, Zi-Xin; Xu, Ming-Yi

Linc-SCRG1 accelerates progression of hepatocellular carcinoma as a ceRNA of miR26a to derepress SKP2

Linc-SCRG1 作为 miR26a 的 ceRNA 来解除 SKP2 的抑制，从而加速肝细胞癌的进展

期刊:Journal of Experimental & Clinical Cancer Research

影响因子:11.4

doi:10.1186/s13046-020-01825-2

Jun-Jie Hu #, Cui Zhou #, Xin Luo #, Sheng-Zheng Luo, Zheng-Hong Li, Zi-Xin Xu, Ming-Yi Xu

Hepatocyte-derived exosomal miR-27a activateshepatic stellate cells through the inhibitionof PINK1-mediated mitophagy in MAFLD

肝细胞来源的外泌体 miR-27a 通过抑制 PINK1 介导的线粒体自噬激活 MAFLD 中的肝星状细胞。

期刊:Molecular Therapy-Nucleic Acids

影响因子:6.1

doi:10.1016/j.omtn.2021.10.022

Luo, Xin; Xu, Zi-Xin; Wu, Jun-Cheng; Luo, Sheng-Zheng; Xu, Ming-Yi

Lipotoxic hepatocyte-derived exosomal miR-1297 promotes hepatic stellate cell activation through the PTEN signaling pathway in metabolic-associated fatty liver disease

脂毒性肝细胞来源的外泌体 miR-1297 通过 PTEN 信号通路促进代谢相关性脂肪肝疾病中肝星状细胞的活化。

期刊:World Journal of Gastroenterology

影响因子:5.4

doi:10.3748/wjg.v27.i14.1419

Luo, Xin; Luo, Sheng-Zheng; Xu, Zi-Xin; Zhou, Cui; Li, Zheng-Hong; Zhou, Xiao-Yan; Xu, Ming-Yi

Zinc-α2-glycoprotein 1 attenuates non-alcoholic fatty liver disease by negatively regulating tumour necrosis factor-α

锌-α2-糖蛋白1通过负调控肿瘤坏死因子-α来减轻非酒精性脂肪性肝病。

期刊:World Journal of Gastroenterology

影响因子:5.4

doi:10.3748/wjg.v25.i36.5451

Liu, Ting; Luo, Xin; Li, Zheng-Hong; Wu, Jun-Cheng; Luo, Sheng-Zheng; Xu, Ming-Yi

Single-cell transcriptomic landscape of intrahepatic B Cells in MASH

MASH中肝内B细胞的单细胞转录组图谱

Application of stem cells in the precise diagnosis and treatment of liver diseases

干细胞在肝脏疾病精准诊断和治疗中的应用

Mertk(+) Liver Sinusoidal Endothelial Cells Negatively Regulate PINK1 Related Mitophagy and Accelerate MASH

Mertk(+)肝窦内皮细胞负调控PINK1相关线粒体自噬并加速MASH

Single-cell transcriptome reveals a novel mechanism of C-Kit(+)-liver sinusoidal endothelial cells in NASH

单细胞转录组揭示了C-Kit(+)肝窦内皮细胞在非酒精性脂肪性肝炎中的一种新机制

Causal relationship between gut microbiota and risk of esophageal cancer: evidence from Mendelian randomization study

肠道菌群与食管癌风险之间的因果关系：来自孟德尔随机化研究的证据

Correction to: Linc-SCRG1 accelerates progression of hepatocellular carcinoma as a ceRNA of miR26a to derepress SKP2

更正：Linc-SCRG1作为miR26a的ceRNA，通过解除SKP2的抑制作用，加速肝细胞癌的进展。

Linc-SCRG1 accelerates progression of hepatocellular carcinoma as a ceRNA of miR26a to derepress SKP2

Linc-SCRG1 作为 miR26a 的 ceRNA 来解除 SKP2 的抑制，从而加速肝细胞癌的进展

Hepatocyte-derived exosomal miR-27a activateshepatic stellate cells through the inhibitionof PINK1-mediated mitophagy in MAFLD

肝细胞来源的外泌体 miR-27a 通过抑制 PINK1 介导的线粒体自噬激活 MAFLD 中的肝星状细胞。

Lipotoxic hepatocyte-derived exosomal miR-1297 promotes hepatic stellate cell activation through the PTEN signaling pathway in metabolic-associated fatty liver disease

脂毒性肝细胞来源的外泌体 miR-1297 通过 PTEN 信号通路促进代谢相关性脂肪肝疾病中肝星状细胞的活化。

Zinc-α2-glycoprotein 1 attenuates non-alcoholic fatty liver disease by negatively regulating tumour necrosis factor-α

锌-α2-糖蛋白1通过负调控肿瘤坏死因子-α来减轻非酒精性脂肪性肝病。