Yadav Vivek R相关文献与解析，生知库 | 链接生命科学的每一步

Celastrol, a triterpene, enhances TRAIL-induced apoptosis through the down-regulation of cell survival proteins and up-regulation of death receptors

雷公藤内酯醇是一种三萜类化合物，它通过下调细胞存活蛋白和上调死亡受体来增强TRAIL诱导的细胞凋亡。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.A109.090209

Sung, Bokyung; Park, Byoungduck; Yadav, Vivek R; Aggarwal, Bharat B

γ-Tocotrienol but not γ-tocopherol blocks STAT3 cell signaling pathway through induction of protein-tyrosine phosphatase SHP-1 and sensitizes tumor cells to chemotherapeutic agents

γ-生育三烯酚（而非γ-生育酚）通过诱导蛋白酪氨酸磷酸酶SHP-1阻断STAT3细胞信号通路，并使肿瘤细胞对化疗药物更加敏感。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.A110.158378

Kannappan, Ramaswamy; Yadav, Vivek R; Aggarwal, Bharat B

Ursolic acid, a pentacyclin triterpene, potentiates TRAIL-induced apoptosis through p53-independent up-regulation of death receptors. EVIDENCE FOR THE ROLE OF REACTIVE OXYGEN SPECIES AND JNK

熊果酸是一种五环三萜，它通过不依赖于p53的死亡受体上调作用增强TRAIL诱导的细胞凋亡。活性氧和JNK的作用证据

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.A110.183699

Prasad, Sahdeo; Yadav, Vivek R; Kannappan, Ramaswamy; Aggarwal, Bharat B

Ursolic acid inhibits the growth of human pancreatic cancer and enhances the antitumor potential of gemcitabine in an orthotopic mouse model through suppression of the inflammatory microenvironment.

熊果酸通过抑制炎症微环境，抑制人类胰腺癌的生长，并增强吉西他滨在原位小鼠模型中的抗肿瘤潜力

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.7537

Prasad Sahdeo, Yadav Vivek R, Sung Bokyung, Gupta Subash C, Tyagi Amit K, Aggarwal Bharat B

The brain metabolic activity after resuscitation with liposome-encapsulated hemoglobin in a rat model of hypovolemic shock

在低血容量性休克大鼠模型中，使用脂质体包裹的血红蛋白进行复苏后，脑代谢活动的变化

期刊:Journal of Cerebral Blood Flow and Metabolism

影响因子:4.5

doi:10.1038/jcbfm.2015.82

Rao, Geeta; Hedrick, Andria F; Yadav, Vivek R; Xie, Jun; Hussain, Alamdar; Awasthi, Vibhudutta

Conformational Dynamics Play an Important Role in the Autoinhibition and Activation of Hyperpolarization-activated Cyclic Nucleotide-modulated (HCN) Ion Channels(♦): Role of Dynamics in the Autoinhibition and Activation of the Hyperpolarization-activated Cyclic Nucleotide-modulated (HCN) Ion Channels

构象动力学在超极化激活环核苷酸调节（HCN）离子通道的自身抑制和激活中起重要作用(♦):动力学在超极化激活环核苷酸调节（HCN）离子通道的自身抑制和激活中的作用

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.A110.121061

Prasad, Sahdeo; Yadav, Vivek R; Sundaram, Chitra; Reuter, Simone; Hema, Padmanabhan S; Nair, Mangalam S; Chaturvedi, Madan M; Aggarwal, Bharat B

发表日期：2015

文献求助收藏

Liposomes modified with superhydrophilic polymer linked to a nonphospholipid anchor exhibit reduced complement activation and enhanced circulation

用超亲水聚合物修饰的脂质体，通过与非磷脂锚连接，可降低补体激活并增强循环。

期刊:Journal of Pharmaceutical Sciences

影响因子:3.8

doi:10.1002/jps.24254

Nag, Okhil K; Yadav, Vivek R; Croft, Brandon; Hedrick, Andria; Awasthi, Vibhudutta

免疫/内分泌

发表日期：2015

文献求助收藏

Biological evaluation of liposome-encapsulated hemoglobin surface-modified with a novel PEGylated nonphospholipid amphiphile

对用新型聚乙二醇化非磷脂两亲分子修饰的脂质体包裹的血红蛋白进行生物学评价

期刊:Artificial Organs

影响因子:2.3

doi:10.1111/aor.12304

Yadav, Vivek R; Nag, Okhil; Awasthi, Vibhudutta

发表日期：2014

文献求助收藏

Hemorrhage-induced interleukin-1 receptor pathway in lung is suppressed by 3,5-bis(2-fluorobenzylidene)-4-piperidone in a rat model of hypovolemic shock

在低血容量性休克大鼠模型中，3,5-双(2-氟亚苄基)-4-哌啶酮可抑制肺部出血诱导的白细胞介素-1受体通路。

期刊:Artificial Organs

影响因子:2.3

doi:10.1111/aor.12305

Yadav, Vivek R; Vilekar, Prachi; Awasthi, Shanjana; Awasthi, Vibhudutta

Integrating Lifestyle Medicine and the Geriatric 5Ms for Sustained Behavior Change in Older Adults

将生活方式医学与老年人5M原则相结合，以实现老年人持续的行为改变

Celastrol, a triterpene, enhances TRAIL-induced apoptosis through the down-regulation of cell survival proteins and up-regulation of death receptors

雷公藤内酯醇是一种三萜类化合物，它通过下调细胞存活蛋白和上调死亡受体来增强TRAIL诱导的细胞凋亡。

γ-Tocotrienol but not γ-tocopherol blocks STAT3 cell signaling pathway through induction of protein-tyrosine phosphatase SHP-1 and sensitizes tumor cells to chemotherapeutic agents

γ-生育三烯酚（而非γ-生育酚）通过诱导蛋白酪氨酸磷酸酶SHP-1阻断STAT3细胞信号通路，并使肿瘤细胞对化疗药物更加敏感。

Ursolic acid, a pentacyclin triterpene, potentiates TRAIL-induced apoptosis through p53-independent up-regulation of death receptors. EVIDENCE FOR THE ROLE OF REACTIVE OXYGEN SPECIES AND JNK

熊果酸是一种五环三萜，它通过不依赖于p53的死亡受体上调作用增强TRAIL诱导的细胞凋亡。活性氧和JNK的作用证据

Ursolic acid inhibits the growth of human pancreatic cancer and enhances the antitumor potential of gemcitabine in an orthotopic mouse model through suppression of the inflammatory microenvironment.

熊果酸通过抑制炎症微环境，抑制人类胰腺癌的生长，并增强吉西他滨在原位小鼠模型中的抗肿瘤潜力

The brain metabolic activity after resuscitation with liposome-encapsulated hemoglobin in a rat model of hypovolemic shock

在低血容量性休克大鼠模型中，使用脂质体包裹的血红蛋白进行复苏后，脑代谢活动的变化

构象动力学在超极化激活环核苷酸调节（HCN）离子通道的自身抑制和激活中起重要作用(♦):动力学在超极化激活环核苷酸调节（HCN）离子通道的自身抑制和激活中的作用

Liposomes modified with superhydrophilic polymer linked to a nonphospholipid anchor exhibit reduced complement activation and enhanced circulation

用超亲水聚合物修饰的脂质体，通过与非磷脂锚连接，可降低补体激活并增强循环。

Biological evaluation of liposome-encapsulated hemoglobin surface-modified with a novel PEGylated nonphospholipid amphiphile

对用新型聚乙二醇化非磷脂两亲分子修饰的脂质体包裹的血红蛋白进行生物学评价

Hemorrhage-induced interleukin-1 receptor pathway in lung is suppressed by 3,5-bis(2-fluorobenzylidene)-4-piperidone in a rat model of hypovolemic shock

在低血容量性休克大鼠模型中，3,5-双(2-氟亚苄基)-4-哌啶酮可抑制肺部出血诱导的白细胞介素-1受体通路。