日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Autism spectrum disorder-associated Sema5A p.Arg676Cys drives Arf6/FE65 signaling and aberrant cell morphogenesis

与自闭症谱系障碍相关的Sema5A p.Arg676Cys突变驱动Arf6/FE65信号通路和异常细胞形态发生

Takahashi, Mikito; Yako, Hideji; Miyamoto, Yuki; Kukimoto-Niino, Mutsuko; Shirouzu, Mikako; Yamauchi, Junji

Alpha-1 Antitrypsin Deficiency-Associated Chronic Obstructive Pulmonary Disease

α1-抗胰蛋白酶缺乏症相关慢性阻塞性肺疾病

Grijalva-Flores, Dagoberto; Resendiz, Marino J E; Takahashi, Mikito; Tanaka, Mei; Yako, Hideji; Miyamoto, Yuki; Yamauchi, Junji; Choi, Jae-Yeon; Singh, Pallavi; Ben Mamoun, Choukri; Albalawi, Yazeed; Zubair, Mohammad; Fouka, Evangelia; Vrouvaki, Argyro; Moustaka Christodoulou, Marina; Loukides, Stelios; Hillas, Georgios

Pyruvate Administration Restores Impaired Nociception by Enhancing Neurite Outgrowth in Streptozotocin-Induced Diabetic Mice

丙酮酸给药可通过促进链脲佐菌素诱导的糖尿病小鼠的神经突生长来恢复受损的伤害感受能力

Yako, Hideji; Suzuki, Mari; Takaku, Shizuka; Niimi, Naoko; Kato, Ayako; Kato, Koichi; Yamauchi, Junji; Sango, Kazunori

Knocking Down FRMD4A, a Factor Associated with the Brain Development Disorder and a Risk Factor for Alzheimer's Disease, Using RNA-Targeting CRISPR/Cas13 Reveals Its Role in Cell Morphogenesis

利用RNA靶向CRISPR/Cas13技术敲除FRMD4A(一种与脑发育障碍相关的因子,也是阿尔茨海默病风险因子),揭示了其在细胞形态发生中的作用

Honjo, Asahi; Yako, Hideji; Miyamoto, Yuki; Yagi, Moeri; Yamamoto, Masahiro; Nishi, Akinori; Sakagami, Hiroyuki; Yamauchi, Junji

The ErbB2-Dock7 Signaling Axis Mediates Excessive Cell Morphogenesis Induced by Autism Spectrum Disorder- and Intellectual Disability-Associated Sema5A p.Arg676Cys

ErbB2-Dock7信号轴介导自闭症谱系障碍和智力障碍相关Sema5A p.Arg676Cys诱导的过度细胞形态发生

Takahashi, Mikito; Yako, Hideji; Suzuki, Ayaka; Isa, Ryuma; Miyamoto, Yuki; Yamauchi, Junji

Claudin-11, a hypomyelinating leukodystrophy 22 (HLD22)-responsible protein, uniquely interacts with shroom-2 to change cell phenotypes

Claudin-11 是一种与髓鞘形成不足的脑白质营养不良 22 (HLD22) 相关的蛋白质,它与 shroom-2 具有独特的相互作用,从而改变细胞表型。

Kobayashi, Sakurako; Yokoi, Takanori; Omata, Takeru; Yako, Hideji; Miyamoto, Yuki; Yamauchi, Junji

Imeglimin improves hyperglycemia and hypoglycemia-induced cell death and mitochondrial dysfunction in immortalized adult mouse Schwann IMS32 cells.

伊美格列明可改善永生化成年小鼠雪旺氏 IMS32 细胞中高血糖和低血糖引起的细胞死亡和线粒体功能障碍

Kato Ayako, Nihei Wataru, Yako Hideji, Tatsumi Yasuaki, Himeno Tatsuhito, Kondo Masaki, Kato Yoshiro, Nakamura Jiro, Kamiya Hideki, Sango Kazunori, Kato Koichi

Immortalized Schwann cell lines as useful tools for pathogenesis-based therapeutic approaches to diabetic peripheral neuropathy

永生化雪旺细胞系作为基于发病机制的糖尿病周围神经病变治疗方法的有效工具

Sango, Kazunori; Yako, Hideji; Niimi, Naoko; Takaku, Shizuka

Advantages of omics approaches for elucidating metabolic changes in diabetic peripheral neuropathy

组学方法在阐明糖尿病周围神经病变代谢变化方面的优势

Yako, Hideji; Niimi, Naoko; Takaku, Shizuka; Sango, Kazunori

Docosahexaenoic Acid Suppresses Oxidative Stress-Induced Autophagy and Cell Death via the AMPK-Dependent Signaling Pathway in Immortalized Fischer Rat Schwann Cells 1.

二十二碳六烯酸通过 AMPK 依赖性信号通路抑制永生化 Fischer 大鼠雪旺细胞中氧化应激诱导的自噬和细胞死亡 1.

Tatsumi Yasuaki, Kato Ayako, Niimi Naoko, Yako Hideji, Himeno Tatsuhito, Kondo Masaki, Tsunekawa Shin, Kato Yoshiro, Kamiya Hideki, Nakamura Jiro, Higai Koji, Sango Kazunori, Kato Koichi