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Occupancy of human EPCR by protein C induces β-arrestin-2 biased PAR1 signaling by both APC and thrombin

蛋白C与人EPCR结合可诱导APC和凝血酶介导的β-arrestin-2偏向性PAR1信号传导

期刊:Blood

影响因子:23.1

doi:10.1182/blood-2016-06-720581

Roy, Ram Vinod; Ardeshirylajimi, Abdolreza; Dinarvand, Peyman; Yang, Likui; Rezaie, Alireza R

Molecular basis of the clotting defect in a bleeding patient missing the Asp-185 codon in the factor X gene

因子X基因中Asp-185密码子缺失导致出血患者凝血缺陷的分子基础

期刊:Thrombosis Research

影响因子:3.4

doi:10.1016/j.thromres.2014.08.004

Lu, Qiuya; Yang, Likui; Manithody, Chandrashekhara; Wang, Xuefeng; Rezaie, Alireza R

发表日期：2014

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Characterization of the protein Z-dependent protease inhibitor interactive-sites of protein Z

蛋白质 Z 依赖性蛋白酶抑制剂相互作用位点的表征

期刊:

影响因子:

doi:10.1016/j.bbapap.2014.06.011

Qureshi, Shabir H; Lu, Qiuya; Manithody, Chandrashekhara; Yang, Likui; Rezaie, Alireza R

发表日期：2014

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Expression and functional characterisation of natural R147W and K150del variants of protein C in the Chinese population

中国人群中天然存在的R147W和K150del蛋白C变体的表达和功能特征

期刊:Thrombosis and Haemostasis

影响因子:4.3

doi:10.1160/TH12-10-0760

Ding, Qiulan; Yang, Likui; Hassanian, Seyed Mahdi; Rezaie, A R

发表日期：2013

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Determinants of the specificity of protease-activated receptors 1 and 2 signaling by factor Xa and thrombin

因子 Xa 和凝血酶介导的蛋白酶激活受体 1 和 2 信号传导特异性的决定因素

期刊:Journal of Cellular Biochemistry

影响因子:2.8

doi:10.1002/jcb.23427

Rana, Soumendra; Yang, Likui; Hassanian, Seyed Mahdi; Rezaie, Alireza R

信号转导

发表日期：2012

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Factor X/Xa elicits protective signaling responses in endothelial cells directly via PAR-2 and indirectly via endothelial protein C receptor-dependent recruitment of PAR-1.

因子 X/Xa 可直接通过 PAR-2 在内皮细胞中引发保护性信号反应，也可间接通过内皮蛋白 C 受体依赖性募集 PAR-1 来引发保护性信号反应

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M110.163642

Bae Jong-Sup, Yang Likui, Rezaie Alireza R

Characterization of a heparin-binding site on the catalytic domain of factor XIa: mechanism of heparin acceleration of factor XIa inhibition by the serpins antithrombin and C1-inhibitor

XIa因子催化结构域上肝素结合位点的表征：肝素加速丝氨酸蛋白酶抑制剂抗凝血酶和C1抑制剂对XIa因子抑制的机制

期刊:Biochemistry

影响因子:3

doi:10.1021/bi802298r

Yang, Likui; Sun, Mao-fu; Gailani, David; Rezaie, Alireza R

发表日期：2009

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Protein Z-dependent protease inhibitor binds to the C-terminal domain of protein Z

蛋白Z依赖性蛋白酶抑制剂与蛋白Z的C端结构域结合。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M802639200

Rezaie, Alireza R; Bae, Jong-Sup; Manithody, Chandrashekhara; Qureshi, Shabir H; Yang, Likui

发表日期：2008

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Nonlinear vibration of a buckled/damaged BNC nanobeam transversally impacted by a high-speed C(60)

受高速 C(60) 横向冲击的屈曲/损伤 BNC 纳米梁的非线性振动

Activated protein C protects against pressure overload-induced hypertrophy through AMPK signaling

活化蛋白C通过AMPK信号通路保护细胞免受压力超负荷诱导的肥大。

Occupancy of human EPCR by protein C induces β-arrestin-2 biased PAR1 signaling by both APC and thrombin

蛋白C与人EPCR结合可诱导APC和凝血酶介导的β-arrestin-2偏向性PAR1信号传导

Molecular basis of the clotting defect in a bleeding patient missing the Asp-185 codon in the factor X gene

因子X基因中Asp-185密码子缺失导致出血患者凝血缺陷的分子基础

Characterization of the protein Z-dependent protease inhibitor interactive-sites of protein Z

蛋白质 Z 依赖性蛋白酶抑制剂相互作用位点的表征

Expression and functional characterisation of natural R147W and K150del variants of protein C in the Chinese population

中国人群中天然存在的R147W和K150del蛋白C变体的表达和功能特征

Determinants of the specificity of protease-activated receptors 1 and 2 signaling by factor Xa and thrombin

因子 Xa 和凝血酶介导的蛋白酶激活受体 1 和 2 信号传导特异性的决定因素

Factor X/Xa elicits protective signaling responses in endothelial cells directly via PAR-2 and indirectly via endothelial protein C receptor-dependent recruitment of PAR-1.

因子 X/Xa 可直接通过 PAR-2 在内皮细胞中引发保护性信号反应，也可间接通过内皮蛋白 C 受体依赖性募集 PAR-1 来引发保护性信号反应

Characterization of a heparin-binding site on the catalytic domain of factor XIa: mechanism of heparin acceleration of factor XIa inhibition by the serpins antithrombin and C1-inhibitor

XIa因子催化结构域上肝素结合位点的表征：肝素加速丝氨酸蛋白酶抑制剂抗凝血酶和C1抑制剂对XIa因子抑制的机制

Protein Z-dependent protease inhibitor binds to the C-terminal domain of protein Z

蛋白Z依赖性蛋白酶抑制剂与蛋白Z的C端结构域结合。