日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Residual inflammatory risk in ischemic stroke: incremental prognostic value of MHR and NPAR beyond the Essen stroke risk score

缺血性卒中残余炎症风险:MHR 和 NPAR 在埃森卒中风险评分基础上的增量预后价值

Yang, Zizhao; Qiao, Pengyan; Zhang, Meiqi; Liang, Yue; Zhao, Liangyuan; Wang, Pingzhi

Epigenetic regulation-mediated disorders in dopamine transporter endocytosis: A novel mechanism for the pathogenesis of Parkinson's disease

表观遗传调控介导的多巴胺转运体内吞作用障碍:帕金森病发病机制的新机制

Liang, Ziqi; Liu, Wanqing; Cao, Mian; Cui, Jiajun; Lan, Jinshuai; Ding, Yue; Zhang, Tong; Yang, Zizhao

Utilization of natural products in diverse pathogeneses of diseases associated with single or double DNA strand damage repair

利用天然产物修复与单链或双链DNA损伤相关的多种疾病的发病机制

Liang, Jiali; Liu, Wanqing; Zhang, Tong; Guo, Dean; Gong, Jiyu; Yang, Zizhao

Notoginsenoside R1 mitigates UVB-induced skin sunburn injury through modulation of N(4)-acetylcytidine and autophagy.

三七皂苷R1通过调节N(4)-乙酰胞苷和自噬来减轻UVB引起的皮肤晒伤损伤。

Liang Shuyun, Liu Xiaokang, Yang Yuting, Zhang Fangyuan, Sun Xiaobo, Zhang Tong, Guo Dean, Gong Jiyu, Yang Zizhao

Targeting the m(6)A RNA demethylase FTO enhances UVB-induced DNA damage repair and suppresses skin tumor growth.

靶向 m(6)A RNA 去甲基化酶 FTO 可增强 UVB 诱导的 DNA 损伤修复并抑制皮肤肿瘤生长。

Yang Zizhao, Verghese Michelle, Cui Yan-Hong, Wei Jiangbo, Yang Seungwon, He Chuan, He Yu-Ying

The role of dsRNA A-to-I editing catalyzed by ADAR family enzymes in the pathogeneses

ADAR家族酶催化的dsRNA A-to-I编辑在发病机制中的作用

Liu, Wanqing; Wu, Yufan; Zhang, Tong; Sun, Xiaobo; Guo, Dean; Yang, Zizhao

The m(6)A reader YTHDC2 regulates UVB-induced DNA damage repair and histone modification.

m(6)A 阅读器 YTHDC2 调节 UVB 诱导的 DNA 损伤修复和组蛋白修饰

Yang Zizhao, Verghese Michelle, Yang Seungwon, Shah Palak, He Yu-Ying

Cellular Immune Signal Exchange From Ischemic Stroke to Intestinal Lesions Through Brain-Gut Axis

缺血性中风通过脑-肠轴向肠道病变传递细胞免疫信号

Yang, Zizhao; Wei, Fei; Zhang, Bin; Luo, Yun; Xing, Xiaoyan; Wang, Min; Chen, Rongchang; Sun, Guibo; Sun, Xiaobo

Myostatin inhibition using mRK35 produces skeletal muscle growth and tubular aggregate formation in wild type and TgACTA1D286G nemaline myopathy mice

使用 mRK35 抑制肌生长抑制素可导致野生型和 TgACTA1D286G 线虫肌病小鼠的骨骼肌生长和管状聚集体形成

Jennifer A Tinklenberg, Emily M Siebers, Margaret J Beatka, Hui Meng, Lin Yang, Zizhao Zhang, Jacob A Ross, Julien Ochala, Carl Morris, Jane M Owens, Nigel G Laing, Kristen J Nowak, Michael W Lawlor