Yohei Ichimura相关文献与解析，生知库 | 链接生命科学的每一步

Ishikawa, Takaaki; Tsurubuchi, Takao; Nagatomo, Kumie; Hosaka, Sho; Fukushima, Hiroko; Sakamoto, Noriaki; Hirato, Junko; Inoue, Yohei; Ichimura, Koichi; Mizumoto, Masashi; Muroi, Ai; Takada, Hidetoshi; Ishikawa, Eiichi

肿瘤

肿瘤免疫

发表日期：2026

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Increased expression of aquaporin-1 in dermal fibroblasts and dermal microvascular endothelial cells possibly contributes to skin fibrosis and edema in patients with systemic sclerosis

真皮成纤维细胞和真皮微血管内皮细胞中水通道蛋白-1表达增加可能导致系统性硬化症患者的皮肤纤维化和水肿

期刊:Journal of Dermatological Science

影响因子:3.8

doi:10.1016/j.jdermsci.2018.09.007

Takashi Yamashita, Yoshihide Asano, Ryosuke Saigusa, Takashi Taniguchi, Kouki Nakamura, Shunsuke Miura, Tetsuo Toyama, Takehiro Takahashi, Yohei Ichimura, Megumi Hirabayashi, Ayumi Yoshizaki, Tomomitsu Miyagaki, Makoto Sugaya, Shinichi Sato

Fli1-haploinsufficient dermal fibroblasts promote skin-localized transdifferentiation of Th2-like regulatory T cells

Fli1单倍体不足的真皮成纤维细胞促进皮肤局部Th2样调节性T细胞的转分化

期刊:Arthritis Research & Therapy

影响因子:4.4

doi:10.1186/s13075-018-1521-3

Ryosuke Saigusa ，Yoshihide Asano ，Takashi Taniguchi ，Megumi Hirabayashi ，Kouki Nakamura ，Shunsuke Miura ，Takashi Yamashita ，Takehiro Takahashi ，Yohei Ichimura ，Tetsuo Toyama ，Ayumi Yoshizaki ，Maria Trojanowska ，Shinichi Sato

Epithelial Fli1 deficiency drives systemic autoimmunity and fibrosis: Possible roles in scleroderma

上皮细胞Fli1缺陷驱动系统性自身免疫和纤维化：可能在硬皮病中发挥作用

期刊:Journal of Experimental Medicine

影响因子:12.6

doi:10.1084/jem.20160247

Takehiro Takahashi ，Yoshihide Asano ，Koji Sugawara ，Takashi Yamashita ，Kouki Nakamura ，Ryosuke Saigusa ，Yohei Ichimura ，Tetsuo Toyama ，Takashi Taniguchi ，Kaname Akamata ，Shinji Noda ，Ayumi Yoshizaki ，Daisuke Tsuruta ，Maria Trojanowska ，Shinichi Sato

Fibrosis, vascular activation, and immune abnormalities resembling systemic sclerosis in bleomycin-treated Fli-1-haploinsufficient mice

接受博来霉素治疗的 Fli-1 单倍体不足小鼠出现纤维化、血管活化和类似系统性硬化症的免疫异常

期刊:Arthritis & Rheumatology

影响因子:11.4

doi:10.1002/art.38948

Takashi Taniguchi, Yoshihide Asano, Kaname Akamata, Shinji Noda, Takehiro Takahashi, Yohei Ichimura, Tetsuo Toyama, Maria Trojanowska, Shinichi Sato

Multifaceted contribution of the TLR4-activated IRF5 transcription factor in systemic sclerosis

TLR4 激活的 IRF5 转录因子在系统性硬化症中的多方面作用

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.1520997112

Ryosuke Saigusa, Yoshihide Asano, Takashi Taniguchi, Takashi Yamashita, Yohei Ichimura, Takehiro Takahashi, Tetsuo Toyama, Ayumi Yoshizaki, Koji Sugawara, Daisuke Tsuruta, Tadatsugu Taniguchi, Shinichi Sato

Simultaneous downregulation of KLF5 and Fli1 is a key feature underlying systemic sclerosis

KLF5 和 Fli1 的同时下调是系统性硬化症的关键特征。

期刊:Nature Communications

影响因子:14.7

doi:10.1038/ncomms6797

Shinji Noda ，Yoshihide Asano ，Satoshi Nishimura ，Takashi Taniguchi ，Katsuhito Fujiu ，Ichiro Manabe ，Kouki Nakamura ，Takashi Yamashita ，Ryosuke Saigusa ，Kaname Akamata ，Takehiro Takahashi ，Yohei Ichimura ，Tetsuo Toyama ，Daisuke Tsuruta ，Maria Trojanowska ，Ryozo Nagai ，Shinichi Sato

A possible contribution of altered cathepsin B expression to the development of skin sclerosis and vasculopathy in systemic sclerosis

蛋白酶 B 表达改变可能导致系统性硬化症的皮肤硬化和血管病变的发展

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0032272

Shinji Noda, Yoshihide Asano, Kaname Akamata, Naohiko Aozasa, Takashi Taniguchi, Takehiro Takahashi, Yohei Ichimura, Tetsuo Toyama, Hayakazu Sumida, Koichi Yanaba, Yayoi Tada, Makoto Sugaya, Takafumi Kadono, Shinichi Sato

Primary subdural tumor mimicking subdural hematoma: illustrative case

原发性硬膜下肿瘤酷似硬膜下血肿：病例分析

Increased expression of aquaporin-1 in dermal fibroblasts and dermal microvascular endothelial cells possibly contributes to skin fibrosis and edema in patients with systemic sclerosis

真皮成纤维细胞和真皮微血管内皮细胞中水通道蛋白-1表达增加可能导致系统性硬化症患者的皮肤纤维化和水肿

Fli1-haploinsufficient dermal fibroblasts promote skin-localized transdifferentiation of Th2-like regulatory T cells

Fli1单倍体不足的真皮成纤维细胞促进皮肤局部Th2样调节性T细胞的转分化

Epithelial Fli1 deficiency drives systemic autoimmunity and fibrosis: Possible roles in scleroderma

上皮细胞Fli1缺陷驱动系统性自身免疫和纤维化：可能在硬皮病中发挥作用

Fibrosis, vascular activation, and immune abnormalities resembling systemic sclerosis in bleomycin-treated Fli-1-haploinsufficient mice

接受博来霉素治疗的 Fli-1 单倍体不足小鼠出现纤维化、血管活化和类似系统性硬化症的免疫异常

Multifaceted contribution of the TLR4-activated IRF5 transcription factor in systemic sclerosis

TLR4 激活的 IRF5 转录因子在系统性硬化症中的多方面作用

Simultaneous downregulation of KLF5 and Fli1 is a key feature underlying systemic sclerosis

KLF5 和 Fli1 的同时下调是系统性硬化症的关键特征。

A possible contribution of altered cathepsin B expression to the development of skin sclerosis and vasculopathy in systemic sclerosis

蛋白酶 B 表达改变可能导致系统性硬化症的皮肤硬化和血管病变的发展