日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

mTORC1 activation drives astrocyte reactivity in cortical tubers and brain organoid models of TSC.

mTORC1 激活驱动 TSC 皮质结节和脑类器官模型中的星形胶质细胞反应性

Li Thomas L, Blair John D, Yoo Taesun, Grant Gerald A, Hockemeyer Dirk, Porter Brenda E, Bateup Helen S

Author Correction: SLC6A20 transporter: a novel regulator of brain glycine homeostasis and NMDAR function

作者更正:SLC6A20 转运蛋白:一种新型的脑甘氨酸稳态和 NMDAR 功能调节因子

Bae, Mihyun; Roh, Junyeop Daniel; Kim, Youjoung; Kim, Seong Soon; Han, Hye Min; Yang, Esther; Kang, Hyojin; Lee, Suho; Kim, Jin Yong; Kang, Ryeonghwa; Jung, Hwajin; Yoo, Taesun; Kim, Hyosang; Kim, Doyoun; Oh, Heejeong; Han, Sungwook; Kim, Dayeon; Han, Jinju; Bae, Yong Chul; Kim, Hyun; Ahn, Sunjoo; Chan, Andrew M; Lee, Daeyoup; Kim, Jin Woo; Kim, Eunjoon

Correction: Adnp-mutant mice with cognitive inflexibility, CaMKIIα hyperactivity, and synaptic plasticity deficits

更正:Adnp突变小鼠表现出认知僵化、CaMKIIα过度活跃和突触可塑性缺陷

Cho, Heejin; Yoo, Taesun; Moon, Heera; Kang, Hyojin; Yang, Yeji; Kang, MinSoung; Yang, Esther; Lee, Dowoon; Hwang, Daehee; Kim, Hyun; Kim, Doyoun; Kim, Jin Young; Kim, Eunjoon

Age, brain region, and gene dosage-differential transcriptomic changes in Shank3-mutant mice

年龄、脑区和基因剂量——Shank3突变小鼠的差异性转录组变化

Yoo, Taesun; Yoo, Ye-Eun; Kang, Hyojin; Kim, Eunjoon

Brain region and gene dosage-differential transcriptomic changes in Shank2-mutant mice

Shank2突变小鼠脑区和基因剂量差异转录组变化

Yoo, Ye-Eun; Yoo, Taesun; Kang, Hyojin; Kim, Eunjoon

A DLG2 deficiency in mice leads to reduced sociability and increased repetitive behavior accompanied by aberrant synaptic transmission in the dorsal striatum.

小鼠的 DLG2 缺陷会导致社交能力下降和重复行为增加,并伴有背侧纹状体突触传递异常

Yoo Taesun, Kim Sun-Gyun, Yang Soo Hyun, Kim Hyun, Kim Eunjoon, Kim Soo Young

Early Correction of N-Methyl-D-Aspartate Receptor Function Improves Autistic-like Social Behaviors in Adult Shank2(-/-) Mice

早期纠正N-甲基-D-天冬氨酸受体功能可改善成年Shank2(-/-)小鼠的自闭症样社交行为

Chung, Changuk; Ha, Seungmin; Kang, Hyojin; Lee, Jiseok; Um, Seung Min; Yan, Haidun; Yoo, Ye-Eun; Yoo, Taesun; Jung, Hwajin; Lee, Dongwon; Lee, Eunee; Lee, Seungjoon; Kim, Jihye; Kim, Ryunhee; Kwon, Yonghan; Kim, Woohyun; Kim, Hyosang; Duffney, Lara; Kim, Doyoun; Mah, Won; Won, Hyejung; Mo, Seojung; Kim, Jin Yong; Lim, Chae-Seok; Kaang, Bong-Kiun; Boeckers, Tobias M; Chung, Yeonseung; Kim, Hyun; Jiang, Yong-Hui; Kim, Eunjoon

Shank3 Exons 14-16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability.

谷氨酸能神经元中 Shank3 外显子 14-16 的缺失导致社交和重复行为缺陷,并伴有皮质第 2/3 层神经元兴奋性增加

Yoo Taesun, Cho Heejin, Park Haram, Lee Jiseok, Kim Eunjoon

Shank3 Mice Carrying the Human Q321R Mutation Display Enhanced Self-Grooming, Abnormal Electroencephalogram Patterns, and Suppressed Neuronal Excitability and Seizure Susceptibility.

携带人类 Q321R 突变的 Shank3 小鼠表现出增强的自我梳理行为、异常的脑电图模式以及抑制的神经元兴奋性和癫痫易感性

Yoo Ye-Eun, Yoo Taesun, Lee Seungjoon, Lee Jiseok, Kim Doyoun, Han Hye-Min, Bae Yong-Chul, Kim Eunjoon