Yu Cheng-Rong相关文献与解析，生知库 | 链接生命科学的每一步

Nagarajan, Vijayaraj; Shi, Guangpu; Horai, Reiko; Yu, Cheng-Rong; Gopalakrishnan, Jaanam; Yadav, Manoj; Liew, Michael H; Gentilucci, Calla; Caspi, Rachel R

发表日期：2025

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Optimal Secretory Expression of Acetaldehyde Dehydrogenase from Issatchenkia terricola in Bacillus subtilis through a Combined Strategy

通过组合策略优化Issatchenkia terricola乙醛脱氢酶在枯草芽孢杆菌中的分泌表达

期刊:Molecules

影响因子:4.2

doi:10.3390/molecules27030747

Jing Lu, Yu Zhao, Yu Cheng, Rong Hu, Yaowei Fang, MingSheng Lyu, Shujun Wang, Zhaoxin Lu

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发表日期：2022

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IL-27-producing B-1a cells suppress neuroinflammation and CNS autoimmune diseases

产生IL-27的B-1a细胞可抑制神经炎症和中枢神经系统自身免疫性疾病。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.2109548118

Choi, Jin Kyeong; Yu, Cheng-Rong; Bing, So Jin; Jittayasothorn, Yingyos; Mattapallil, Mary J; Kang, Minkyung; Park, Seung Bum; Lee, Hyun-Su; Dong, Lijin; Shi, Guangpu; Caspi, Rachel R; Egwuagu, Charles E

STAT3-Specific Single Domain Nanobody Inhibits Expansion of Pathogenic Th17 Responses and Suppresses Uveitis in Mice

STAT3特异性单域纳米抗体抑制致病性Th17反应的扩增并抑制小鼠葡萄膜炎

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2021.724609

Mbanefo, Evaristus C; Yan, Ming; Kang, Minkyung; Alhakeem, Sahar A; Jittayasothorn, Yingyos; Yu, Cheng-Rong; Parihar, Ashutosh; Singh, Sunanda; Egwuagu, Charles E

Deletion of Irf4 in T Cells Suppressed Autoimmune Uveitis and Dysregulated Transcriptional Programs Linked to CD4(+) T Cell Differentiation and Metabolism

T细胞中Irf4的缺失抑制了自身免疫性葡萄膜炎，并导致与CD4(+) T细胞分化和代谢相关的转录程序失调。

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms22052775

Kang, Minkyung; Lee, Hyun-Su; Choi, Jin Kyeong; Yu, Cheng-Rong; Egwuagu, Charles E

STAT3 deficiency in B cells exacerbates uveitis by promoting expansion of pathogenic lymphocytes and suppressing regulatory B cells (Bregs) and Tregs

B细胞中STAT3的缺乏会促进致病性淋巴细胞的扩增，并抑制调节性B细胞（Bregs）和Tregs，从而加剧葡萄膜炎。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-020-73093-1

Oladipupo, Favour O; Yu, Cheng-Rong; Olumuyide, Ezekiel; Jittaysothorn, Yingyos; Choi, Jin Kyeong; Egwuagu, Charles E

Persistent Activation of STAT3 Pathway in the Retina Induced Vision Impairment and Retinal Degenerative Changes in Ageing Mice

视网膜中STAT3通路持续激活导致衰老小鼠出现视力障碍和视网膜退行性改变

期刊:Advances in Experimental Medicine and Biology

影响因子:

doi:10.1007/978-3-030-27378-1_58

Marrero, Bernadette; He, Chang; Oh, Hyun-Mee; Ukwu, Umegbewe T; Yu, Cheng-Rong; Dambuza, Ivy M; Sun, Lin; Egwuagu, Charles E

Production of IL-35 by Bregs is mediated through binding of BATF-IRF-4-IRF-8 complex to il12a and ebi3 promoter elements

Breg细胞通过BATF-IRF-4-IRF-8复合物与il12a和ebi3启动子元件结合来介导IL-35的产生。

期刊:Journal of Leukocyte Biology

影响因子:3.1

doi:10.1002/JLB.3A0218-071RRR

Yu, Cheng-Rong; Choi, Jin Kyeong; Uche, Anita N; Egwuagu, Charles E

IL-12p35 induces expansion of IL-10 and IL-35-expressing regulatory B cells and ameliorates autoimmune disease.

IL-12p35 诱导表达 IL-10 和 IL-35 的调节性 B 细胞扩增，并改善自身免疫性疾病

期刊:Nature Communications

影响因子:15.7

doi:10.1038/s41467-017-00838-4

Dambuza Ivy M, He Chang, Choi Jin Kyeong, Yu Cheng-Rong, Wang Renxi, Mattapallil Mary J, Wingfield Paul T, Caspi Rachel R, Egwuagu Charles E

IL-12p35 Inhibits Neuroinflammation and Ameliorates Autoimmune Encephalomyelitis

IL-12p35抑制神经炎症并改善自身免疫性脑脊髓炎

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2017.01258

Choi, Jin Kyeong; Dambuza, Ivy M; He, Chang; Yu, Cheng-Rong; Uche, Anita N; Mattapallil, Mary J; Caspi, Rachel R; Egwuagu, Charles E

IAN: An Intelligent System for Omics Data Analysis and Discovery

IAN：用于组学数据分析和发现的智能系统

Optimal Secretory Expression of Acetaldehyde Dehydrogenase from Issatchenkia terricola in Bacillus subtilis through a Combined Strategy

通过组合策略优化Issatchenkia terricola乙醛脱氢酶在枯草芽孢杆菌中的分泌表达

IL-27-producing B-1a cells suppress neuroinflammation and CNS autoimmune diseases

产生IL-27的B-1a细胞可抑制神经炎症和中枢神经系统自身免疫性疾病。

STAT3-Specific Single Domain Nanobody Inhibits Expansion of Pathogenic Th17 Responses and Suppresses Uveitis in Mice

STAT3特异性单域纳米抗体抑制致病性Th17反应的扩增并抑制小鼠葡萄膜炎

Deletion of Irf4 in T Cells Suppressed Autoimmune Uveitis and Dysregulated Transcriptional Programs Linked to CD4(+) T Cell Differentiation and Metabolism

T细胞中Irf4的缺失抑制了自身免疫性葡萄膜炎，并导致与CD4(+) T细胞分化和代谢相关的转录程序失调。

STAT3 deficiency in B cells exacerbates uveitis by promoting expansion of pathogenic lymphocytes and suppressing regulatory B cells (Bregs) and Tregs

B细胞中STAT3的缺乏会促进致病性淋巴细胞的扩增，并抑制调节性B细胞（Bregs）和Tregs，从而加剧葡萄膜炎。

Persistent Activation of STAT3 Pathway in the Retina Induced Vision Impairment and Retinal Degenerative Changes in Ageing Mice

视网膜中STAT3通路持续激活导致衰老小鼠出现视力障碍和视网膜退行性改变

Production of IL-35 by Bregs is mediated through binding of BATF-IRF-4-IRF-8 complex to il12a and ebi3 promoter elements

Breg细胞通过BATF-IRF-4-IRF-8复合物与il12a和ebi3启动子元件结合来介导IL-35的产生。

IL-12p35 induces expansion of IL-10 and IL-35-expressing regulatory B cells and ameliorates autoimmune disease.

IL-12p35 诱导表达 IL-10 和 IL-35 的调节性 B 细胞扩增，并改善自身免疫性疾病

IL-12p35 Inhibits Neuroinflammation and Ameliorates Autoimmune Encephalomyelitis

IL-12p35抑制神经炎症并改善自身免疫性脑脊髓炎