Yu Jungeun相关文献与解析，生知库 | 链接生命科学的每一步

A NOTCH3 pathogenic variant influences osteogenesis and can be targeted by antisense oligonucleotides in induced pluripotent stem cells

NOTCH3致病变异会影响成骨作用，并且可以通过诱导多能干细胞中的反义寡核苷酸进行靶向治疗。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0316644

Canalis, Ernesto; Yu, Jungeun; Schilling, Lauren; Jafar-Nejad, Paymaan; Carrer, Michele

A NOTCH2 pathogenic variant and HES1 regulate osteoclastogenesis in induced pluripotent stem cells

NOTCH2致病变异体和HES1调控诱导多能干细胞中的破骨细胞生成

期刊:

影响因子:

doi:10.1016/j.bone.2024.117334

Canalis, Ernesto; Schilling, Lauren; Denker, Emily; Stoddard, Christopher; Yu, Jungeun

1,3-Dibenzyl-5-Fluorouracil Prevents Ovariectomy-Induced Bone Loss by Suppressing Osteoclast Differentiation

1,3-二苄基-5-氟尿嘧啶通过抑制破骨细胞分化来预防卵巢切除术引起的骨质流失

期刊:Immune Network

影响因子:4.1

doi:10.4110/in.2022.22.e43

Jeon, Hyoeun; Yu, Jungeun; Hwang, Jung Me; Park, Hye-Won; Yu, Jiyeon; Lee, Zee-Won; Kim, Taesoo; Rho, Jaerang

细胞生物学

发表日期：2022

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Use of antisense oligonucleotides to target Notch3 in skeletal cells

利用反义寡核苷酸靶向骨骼细胞中的Notch3

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0268225

Canalis, Ernesto; Carrer, Michele; Eller, Tabitha; Schilling, Lauren; Yu, Jungeun

T-Cell Death Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγ-RXRα Heterodimer Formation in Adipogenesis

T细胞死亡相关基因51是PPARγ的一种新型负调控因子，可抑制脂肪生成过程中PPARγ-RXRα异二聚体的形成

期刊:Molecules and Cells

影响因子:6.5

doi:10.14348/molcells.2020.0143

Kim, Sumi; Lee, Nari; Park, Eui-Soon; Yun, Hyeongseok; Ha, Tae-Uk; Jeon, Hyoeun; Yu, Jiyeon; Choi, Seunga; Shin, Bongjin; Yu, Jungeun; Rhee, Sang Dal; Choi, Yongwon; Rho, Jaerang

An antibody to Notch3 reverses the skeletal phenotype of lateral meningocele syndrome in male mice

针对 Notch3 的抗体可逆转雄性小鼠侧脑膜膨出综合征的骨骼表型

期刊:Journal of Cellular Physiology

影响因子:4

doi:10.1002/jcp.28960

Yu, Jungeun; Siebel, Christian W; Schilling, Lauren; Canalis, Ernesto

Nuclear factor of activated T cells 1 and 2 are required for vertebral homeostasis

活化T细胞核因子1和2是维持椎骨稳态所必需的

期刊:Journal of Cellular Physiology

影响因子:4

doi:10.1002/jcp.29696

Canalis, Ernesto; Schilling, Lauren; Eller, Tabitha; Yu, Jungeun

Notch and the regulation of osteoclast differentiation and function

Notch信号通路及其对破骨细胞分化和功能的调控

期刊:

影响因子:

doi:10.1016/j.bone.2020.115474

Yu, Jungeun; Canalis, Ernesto

TDAG51 is a crucial regulator of maternal care and depressive-like behavior after parturition.

TDAG51 是产后母性照护和抑郁样行为的关键调节因子

期刊:PLoS Genetics

影响因子:3.7

doi:10.1371/journal.pgen.1008214

Yun Hyeongseok, Park Eui-Soon, Choi Seunga, Shin Bongjin, Yu Jungeun, Yu Jiyeon, Amarasekara Dulshara Sachini, Kim Sumi, Lee Nari, Choi Jong-Soon, Choi Yongwon, Rho Jaerang

A novel variant of NOTCH2 causes skeletal fragility

NOTCH2 的一种新型变体导致骨骼脆弱

A NOTCH3 pathogenic variant influences osteogenesis and can be targeted by antisense oligonucleotides in induced pluripotent stem cells

NOTCH3致病变异会影响成骨作用，并且可以通过诱导多能干细胞中的反义寡核苷酸进行靶向治疗。

A NOTCH2 pathogenic variant and HES1 regulate osteoclastogenesis in induced pluripotent stem cells

NOTCH2致病变异体和HES1调控诱导多能干细胞中的破骨细胞生成

1,3-Dibenzyl-5-Fluorouracil Prevents Ovariectomy-Induced Bone Loss by Suppressing Osteoclast Differentiation

1,3-二苄基-5-氟尿嘧啶通过抑制破骨细胞分化来预防卵巢切除术引起的骨质流失

Use of antisense oligonucleotides to target Notch3 in skeletal cells

利用反义寡核苷酸靶向骨骼细胞中的Notch3

T-Cell Death Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγ-RXRα Heterodimer Formation in Adipogenesis

T细胞死亡相关基因51是PPARγ的一种新型负调控因子，可抑制脂肪生成过程中PPARγ-RXRα异二聚体的形成

An antibody to Notch3 reverses the skeletal phenotype of lateral meningocele syndrome in male mice

针对 Notch3 的抗体可逆转雄性小鼠侧脑膜膨出综合征的骨骼表型

Nuclear factor of activated T cells 1 and 2 are required for vertebral homeostasis

活化T细胞核因子1和2是维持椎骨稳态所必需的

Notch and the regulation of osteoclast differentiation and function

Notch信号通路及其对破骨细胞分化和功能的调控

TDAG51 is a crucial regulator of maternal care and depressive-like behavior after parturition.

TDAG51 是产后母性照护和抑郁样行为的关键调节因子