Zhang David X相关文献与解析，生知库 | 链接生命科学的每一步

The role of NO, H(2)O(2), and non-NO/H(2)O(2) mechanisms in acetylcholine (ACh)-induced dilation of human arterioles in the absence and presence of coronary artery disease

在有无冠状动脉疾病的情况下，NO、H₂O₂以及非NO/H₂O₂机制在乙酰胆碱（ACh）诱导的人类小动脉扩张中的作用

Drachuk, Kostiantyn; Nishijima, Yoshinori; Parthasarathy, Aravind; Xie, Yangjing; Nagavally, Sneha; Dawson, Aprill; Gutterman, David D; Zhang, David X

发表日期：2025

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Phosphorylation of distal C-terminal residues promotes TRPV4 channel activation in response to arachidonic acid.

远端 C 端残基的磷酸化促进 TRPV4 通道在花生四烯酸刺激下的激活

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1016/j.jbc.2025.108260

Parthasarathy Aravind, Anishkin Andriy, Xie Yangjing, Drachuk Kostiantyn, Nishijma Yoshinori, Fang Juan, Koukouritaki Sevasti B, Wilcox David A, Zhang David X

Prolonged L-NAME exposure changes the vasodilator factor from NO to H(2)O(2) in human arterioles in response to A23187

长期暴露于 L-NAME 会使人体小动脉中血管舒张因子由 NO 转变为 H₂O₂，以响应 A23187 的刺激。

期刊:Vascular Pharmacology

影响因子:3.5

doi:10.1016/j.vph.2024.107440

Zinkevich, Natalya S; Drachuk, Kostiantyn; Zhang, David X

发表日期：2024

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Inhibiting NADPH Oxidases to Target Vascular and Other Pathologies: An Update on Recent Experimental and Clinical Studies

抑制 NADPH 氧化酶以治疗血管及其他疾病：近期实验和临床研究进展

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom12060823

Sylvester, Anthony L; Zhang, David X; Ran, Sophia; Zinkevich, Natalya S

发表日期：2022

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Endothelial Rap1 (Ras-Association Proximate 1) Restricts Inflammatory Signaling to Protect From the Progression of Atherosclerosis

内皮细胞 Rap1（Ras 关联近端蛋白 1）限制炎症信号传导，从而防止动脉粥样硬化的进展

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.120.315401

Singh, Bandana; Kosuru, Ramoji; Lakshmikanthan, Sribalaji; Sorci-Thomas, Mary G; Zhang, David X; Sparapani, Rodney; Vasquez-Vivar, Jeannette; Chrzanowska, Magdalena

Corrigendum: Distinct Signaling Functions of Rap1 Isoforms in NO Release From Endothelium

更正：Rap1亚型在内皮细胞释放NO中的不同信号传导功能

期刊:Frontiers in Cell and Developmental Biology

影响因子:4.3

doi:10.3389/fcell.2021.741935

Kosuru, Ramoji; Singh, Bandana; Lakshmikanthan, Sribalaji; Nishijima, Yoshinori; Vasquez-Vivar, Jeannette; Zhang, David X; Chrzanowska, Magdalena

Myocardin and Kv1 Channels: A Paradigm Shift in Treating Vascular Smooth Muscle Cell-Related Proliferative Disease?

心肌蛋白和Kv1通道：治疗血管平滑肌细胞相关增生性疾病的范式转变？

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.119.313531

Zhang, David X; Gutterman, David D

细胞生物学

发表日期：2019

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Endothelial Hypoxia-Inducible Factor-1α Is Required for Vascular Repair and Resolution of Inflammatory Lung Injury through Forkhead Box Protein M1

内皮缺氧诱导因子-1α 通过叉头框蛋白 M1 参与血管修复和炎症性肺损伤消退

期刊:American Journal of Pathology

影响因子:4.7

doi:10.1016/j.ajpath.2019.04.014

Xiaojia Huang, Xianming Zhang, David X Zhao, Jun Yin, Guochang Hu, Colin E Evans, You-Yang Zhao

心血管

发表日期：2019

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Endothelin receptor A and p66Shc regulate spontaneous Ca(2+) oscillations in smooth muscle cells controlling renal arterial spontaneous motion

内皮素受体A和p66Shc调节平滑肌细胞中自发的Ca(2+)振荡，从而控制肾动脉的自发运动。

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.201800776RR

Palygin, Oleg; Miller, Bradley S; Nishijima, Yoshinori; Zhang, David X; Staruschenko, Alexander; Sorokin, Andrey

An update on regulation of the polymodal TRPV4 channel by protein phosphorylation

关于蛋白质磷酸化调控多模态TRPV4通道的最新研究进展

The role of NO, H(2)O(2), and non-NO/H(2)O(2) mechanisms in acetylcholine (ACh)-induced dilation of human arterioles in the absence and presence of coronary artery disease

在有无冠状动脉疾病的情况下，NO、H₂O₂以及非NO/H₂O₂机制在乙酰胆碱（ACh）诱导的人类小动脉扩张中的作用

Phosphorylation of distal C-terminal residues promotes TRPV4 channel activation in response to arachidonic acid.

远端 C 端残基的磷酸化促进 TRPV4 通道在花生四烯酸刺激下的激活

Prolonged L-NAME exposure changes the vasodilator factor from NO to H(2)O(2) in human arterioles in response to A23187

长期暴露于 L-NAME 会使人体小动脉中血管舒张因子由 NO 转变为 H₂O₂，以响应 A23187 的刺激。

Inhibiting NADPH Oxidases to Target Vascular and Other Pathologies: An Update on Recent Experimental and Clinical Studies

抑制 NADPH 氧化酶以治疗血管及其他疾病：近期实验和临床研究进展

Endothelial Rap1 (Ras-Association Proximate 1) Restricts Inflammatory Signaling to Protect From the Progression of Atherosclerosis

内皮细胞 Rap1（Ras 关联近端蛋白 1）限制炎症信号传导，从而防止动脉粥样硬化的进展

Corrigendum: Distinct Signaling Functions of Rap1 Isoforms in NO Release From Endothelium

更正：Rap1亚型在内皮细胞释放NO中的不同信号传导功能

Myocardin and Kv1 Channels: A Paradigm Shift in Treating Vascular Smooth Muscle Cell-Related Proliferative Disease?

心肌蛋白和Kv1通道：治疗血管平滑肌细胞相关增生性疾病的范式转变？

Endothelial Hypoxia-Inducible Factor-1α Is Required for Vascular Repair and Resolution of Inflammatory Lung Injury through Forkhead Box Protein M1

内皮缺氧诱导因子-1α 通过叉头框蛋白 M1 参与血管修复和炎症性肺损伤消退

Endothelin receptor A and p66Shc regulate spontaneous Ca(2+) oscillations in smooth muscle cells controlling renal arterial spontaneous motion

内皮素受体A和p66Shc调节平滑肌细胞中自发的Ca(2+)振荡，从而控制肾动脉的自发运动。