Zhang Pumin相关文献与解析，生知库 | 链接生命科学的每一步

The deubiquitinase USP28 maintains the expression of PPARγ and its inactivation protects mice from diet-induced MASH and hepatocarcinoma

去泛素化酶USP28维持PPARγ的表达，其失活可保护小鼠免受饮食诱导的MASH和肝癌的侵害。

doi:10.1016/j.ymthe.2025.01.046

Cai, Changzhou; Luo, Hangqi; Peng, Jin; Zhen, Xinghua; Shen, Xiang; Xi, Xiaomei; Zhu, Jianrong; Fang, Yanfei; Chen, Xiaoli; Wang, Jiewei; Yu, Chaohui; Zhang, Pumin; Xu, Chengfu

USP25 maintains KRAS expression and inhibiting the deubiquitinase suppresses KRAS signaling in human cancer

USP25 维持 KRAS 表达，抑制去泛素化酶可抑制人类癌症中的 KRAS 信号传导。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1016/j.jbc.2025.110337

Ma, Huailu; Guan, Huiyuan; Sun, Xiao; Wu, Lingzhi; Cai, Mengjiao; Zhen, Xinghua; Shen, Xiang; Han, Suxia; Liu, Guangxue; Peng, Jin; Zhang, Pumin

Advances in preclinical and clinical studies of oncolytic virus combination therapy

溶瘤病毒联合疗法的临床前和临床研究进展

期刊:Frontiers in Oncology

影响因子:3.3

doi:10.3389/fonc.2025.1545542

Du, Wenlong; Na, Jintong; Zhong, Liping; Zhang, Pumin

微生物学

发表日期：2025

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METTL14 suppresses the expression of YAP1 and the stemness of triple-negative breast cancer.

METTL14 抑制 YAP1 的表达和三阴性乳腺癌的干细胞特性

期刊:Journal of Experimental & Clinical Cancer Research

影响因子:12.8

doi:10.1186/s13046-024-03225-2

Bai Xupeng, Liu Jiarui, Zhou Shujie, Wu Lingzhi, Feng Xiaojie, Zhang Pumin

RAD51 plays critical roles in DNMT1-mediated maintenance methylation of genomic DNA by dually regulating the ubiquitin ligase UHRF1

RAD51通过双重调控泛素连接酶UHRF1，在DNMT1介导的基因组DNA维持性甲基化中发挥关键作用。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.2410119121

Liu, Guangxue; Huang, Kaiyan; Liu, Shiyao; Xie, Yali; Huang, Jinyan; Liang, Tingbo; Zhang, Pumin

Cdh1 Deficiency Sensitizes TNBC Cells to PARP Inhibitors

Cdh1 缺乏使 TNBC 细胞对 PARP 抑制剂敏感

期刊:Genes

影响因子:

doi:10.3390/genes13050803

Junjun Li, Mengjiao Lan, Jin Peng, Qunli Xiong, Yongfeng Xu, Yang Yang, Ying Zhou, Jinlu Liu, Zhu Zeng, Xiaojuan Yang, Zhiwei Zhang, Pumin Zhang, Qing Zhu, Wei Wu

Transcription-Replication Collisions and Chromosome Fragility

转录-复制碰撞和染色体脆性

期刊:Frontiers in Genetics

影响因子:2.8

doi:10.3389/fgene.2021.804547

Wu, Wei; He, Jing Na; Lan, Mengjiao; Zhang, Pumin; Chu, Wai Kit

发表日期：2021

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Acetylation of TIP60 at K104 is essential for metabolic stress-induced apoptosis in cells of hepatocellular cancer

TIP60 K104 位点的乙酰化对于肝细胞癌细胞代谢应激诱导的细胞凋亡至关重要。

期刊:Experimental Cell Research

影响因子:3.5

doi:10.1016/j.yexcr.2017.11.028

Fang, Xiao; Lu, Guojun; Ha, Kyungsoo; Lin, Han; Du, Ye; Zuo, Qiuhong; Fu, Yi; Zou, Chaoxia; Zhang, Pumin

Targeted p16(Ink4a) epimutation causes tumorigenesis and reduces survival in mice

靶向p16(Ink4a)表观突变会导致小鼠肿瘤发生并降低其生存率。

期刊:Journal of Clinical Investigation

影响因子:13.6

doi:10.1172/JCI76507

Yu, Da-Hai; Waterland, Robert A; Zhang, Pumin; Schady, Deborah; Chen, Miao-Hsueh; Guan, Yongtao; Gadkari, Manasi; Shen, Lanlan

UHRF1 Controls the Timing of RAD51 Removal During DNA Damage Repair Through Suppressing RFWD3

UHRF1通过抑制RFWD3来控制DNA损伤修复过程中RAD51移除的时机。

The deubiquitinase USP28 maintains the expression of PPARγ and its inactivation protects mice from diet-induced MASH and hepatocarcinoma

去泛素化酶USP28维持PPARγ的表达，其失活可保护小鼠免受饮食诱导的MASH和肝癌的侵害。

USP25 maintains KRAS expression and inhibiting the deubiquitinase suppresses KRAS signaling in human cancer

USP25 维持 KRAS 表达，抑制去泛素化酶可抑制人类癌症中的 KRAS 信号传导。

Advances in preclinical and clinical studies of oncolytic virus combination therapy

溶瘤病毒联合疗法的临床前和临床研究进展

METTL14 suppresses the expression of YAP1 and the stemness of triple-negative breast cancer.

METTL14 抑制 YAP1 的表达和三阴性乳腺癌的干细胞特性

RAD51 plays critical roles in DNMT1-mediated maintenance methylation of genomic DNA by dually regulating the ubiquitin ligase UHRF1

RAD51通过双重调控泛素连接酶UHRF1，在DNMT1介导的基因组DNA维持性甲基化中发挥关键作用。

Cdh1 Deficiency Sensitizes TNBC Cells to PARP Inhibitors

Cdh1 缺乏使 TNBC 细胞对 PARP 抑制剂敏感

Transcription-Replication Collisions and Chromosome Fragility

转录-复制碰撞和染色体脆性

Acetylation of TIP60 at K104 is essential for metabolic stress-induced apoptosis in cells of hepatocellular cancer

TIP60 K104 位点的乙酰化对于肝细胞癌细胞代谢应激诱导的细胞凋亡至关重要。

Targeted p16(Ink4a) epimutation causes tumorigenesis and reduces survival in mice

靶向p16(Ink4a)表观突变会导致小鼠肿瘤发生并降低其生存率。