Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis
烟酰胺单核苷酸通过抑制CD36-TLR4轴,激活v-ATPase介导的糖酵解,从而改善脂质诱导的心肌病。
期刊:Circulation Research
影响因子:16.2
doi:10.1161/CIRCRESAHA.123.322910
Wang, Shujin; Han, Yinying; Liu, Ruimin; Hou, Mengqian; Neumann, Dietbert; Zhang, Jun; Wang, Fang; Li, Yumeng; Zhao, Xueya; Schianchi, Francesco; Dai, Chao; Liu, Lizhong; Nabben, Miranda; Glatz, Jan F C; Wu, Xin; Lu, Xifeng; Li, Xi; Luiken, Joost J F P