日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Influences of probiotics plus enteral nutrition on intestinal flora and inflammatory response in patients with severe acute pancreatitis.

益生菌联合肠内营养对重症急性胰腺炎患者肠道菌群和炎症反应的影响。

Cheng Liping, Jiang Chunying, Jiang Wei, Zhu Changhong

Alleviating Overgrazing Stress and Promoting Grassland Plant Regeneration via Root Exudate-Mediated Recruitment of Beneficial Bacteria

通过根系分泌物介导的有益细菌招募来缓解过度放牧胁迫并促进草地植物再生

Yuan, Ting; Zhang, Jiatao; Zhang, Shaohong; Liang, Shuang; Zhu, Changhong; Ren, Weibo; Liang, Jialu

Efficacy of Endoscopic Tissue Adhesive in Patients with Gastrointestinal Tumor Bleeding

内镜组织粘合剂治疗胃肠道肿瘤出血患者的疗效

Shen, Jun; Ni, Lingna; Zhu, Changhong; Jiang, Chunying; Zhu, Wenyu; Bi, Yanzhi

Bioinformatics analysis of the common targets of miR-223-3p, miR-122-5p, and miR-93-5p in polycystic ovarian syndrome

对多囊卵巢综合征中 miR-223-3p、miR-122-5p 和 miR-93-5p 的共同靶点进行生物信息学分析

Zou, Liping; Feng, Qiwen; Xia, Wei; Zhu, Changhong

SETD8 cooperates with MZF1 to participate in hyperglycemia-induced endothelial inflammation via elevation of WNT5A levels in diabetic nephropathy

SETD8 与 MZF1 协同通过提高糖尿病肾病中的 WNT5A 水平参与高血糖诱导的内皮炎症

Fei Wang #, Wenting Hou #, Xue Li, Lihong Lu, Ting Huang, Minmin Zhu, Changhong Miao

Correction to: EndMT: New fndings on the origin of myofbroblasts in endometrial fbrosis of intrauterine adhesions

更正:EndMT:关于子宫内膜纤维化和宫腔粘连中成肌细胞起源的新发现

Xu, Chengcheng; Bao, Meng; Fan, Xiaorong; Huang, Jin; Zhu, Changhong; Xia, Wei

c-Myc participates in high glucose-mediated endothelial inflammation via upregulation of IRAK1 expression in diabetic nephropathy

c-Myc 通过上调糖尿病肾病中的 IRAK1 表达参与高糖介导的内皮炎症

Wenting Hou, Lihong Lu, Xue Li, Minli Sun, Minmin Zhu, Changhong Miao

KMT5A downregulation participated in High Glucose-mediated EndMT via Upregulation of ENO1 Expression in Diabetic Nephropathy

KMT5A 下调通过上调糖尿病肾病中的 ENO1 表达参与高糖介导的 EndMT

Lihong Lu, Xue Li, Ziwen Zhong, Wenchang Zhou, Di Zhou, Minmin Zhu, Changhong Miao

The CREB/KMT5A complex regulates PTP1B to modulate high glucose-induced endothelial inflammatory factor levels in diabetic nephropathy

CREB/KMT5A复合物通过调节PTP1B来调控糖尿病肾病中高糖诱导的内皮炎症因子水平。

Ting Huang #,Xue Li #,Fei Wang,Lihong Lu,Wenting Hou,Minmin Zhu,Changhong Miao

ets1 associates with KMT5A to participate in high glucose-mediated EndMT via upregulation of PFN2 expression in diabetic nephropathy

ets1 与 KMT5A 关联,通过上调糖尿病肾病中的 PFN2 表达参与高糖介导的 EndMT

Lihong Lu #, Ziwen Zhong #, Jiahui Gu #, Ke Nan, Minmin Zhu, Changhong Miao