Unusually persistent Gα(i)-signaling of the neuropeptide Y(2) receptor depletes cellular G(i/o) pools and leads to a G(i)-refractory state
神经肽Y(2)受体的Gα(i)信号异常持续激活会耗竭细胞内的G(i/o)蛋白池，导致细胞进入G(i)蛋白难治状态。
期刊:Cell Communication and Signaling
影响因子:8.9
doi:10.1186/s12964-020-00537-6
Ziffert, Isabelle; Kaiser, Anette; Babilon, Stefanie; Mörl, Karin; Beck-Sickinger, Annette G
