日期:
2020 年 — 2026 年
2020
2021
2022
2023
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2025
2026
影响因子:

Urinary biomarkers for early diabetic nephropathy in type 2 diabetic patients

2型糖尿病患者早期糖尿病肾病的尿液生物标志物

Fiseha, Temesgen

Author Correction: Factor XII signaling via uPAR-integrin β1 axis promotes tubular senescence in diabetic kidney disease

作者更正:因子 XII 通过 uPAR-整合素 β1 轴发出信号,促进糖尿病肾病中的肾小管衰老

Elwakiel, Ahmed; Gupta, Dheerendra; Rana, Rajiv; Manoharan, Jayakumar; Al-Dabet, Moh'd Mohanad; Ambreen, Saira; Fatima, Sameen; Zimmermann, Silke; Mathew, Akash; Li, Zhiyang; Singh, Kunal; Gupta, Anubhuti; Pal, Surinder; Sulaj, Alba; Kopf, Stefan; Schwab, Constantin; Baber, Ronny; Geffers, Robert; Götze, Tom; Alo, Bekas; Lamers, Christina; Kluge, Paul; Kuenze, Georg; Kohli, Shrey; Renné, Thomas; Shahzad, Khurrum; Isermann, Berend

Energy-sensing molecule RORγ regulates cholesterol metabolism and immune signaling in diabetic kidney disease and aging.

能量感知分子 RORγ 调节糖尿病肾病和衰老过程中的胆固醇代谢和免疫信号传导。

Liang Zhen, Xiang Jiaqing, Yang Guangyan, Liu Xiaomai, Li Lixing, Li Yanchun, Lu Yan, Kang Lin, Chen Yuanli, Ma Chuanrui, Yang Shu

AARS1-mediated lactylation of H3K18 and STAT1 promotes ferroptosis in diabetic nephropathy.

AARS1 介导的 H3K18 和 STAT1 乳酸化促进糖尿病肾病中的铁死亡。

Hong Jia, Xu Hongjiao, Yu Lang, Yu Zhuang, Chen Xiangyuan, Meng Zhipeng, Zhu Jiali, Li Jinbao, Zhu Minmin

ESRRA-ATG5-Mediated mitophagy enhances arginine metabolism to alleviate diabetic kidney disease.

ESRRA-ATG5介导的线粒体自噬增强精氨酸代谢,从而缓解糖尿病肾病。

Hu Hongtu, Yang Qian, Hu Jijia, Fan Yanqing, Zhang Zongwei, Yang Keju, Li Weiwei, Peng Zhuan, Chen Zhaowei, Ding Guohua, Liang Wei

SETDB2 Mitigates Podocyte Dysfunction in Diabetic Kidney Disease Through Epigenetic Silencing of SMAD3

SETDB2通过表观遗传沉默SMAD3减轻糖尿病肾病中的足细胞功能障碍

Li, Lanfang; Jiang, Shimin; Jin, Qi; Qu, Peng; Guo, Yingjie; Lan, Xushan; Li, Xinyu; Sun, Cuiting; Ai, Sinan; Li, Xin; Sun, Weiliang; Guo, Jing; Li, Wenge; Peng, Liang; Liu, Lihong

Podocyte TLR4 deletion alleviates diabetic kidney disease through prohibiting PKCδ/SHP-1-dependent ER stress and relieving podocyte damage and inflammation.

足细胞 TLR4 缺失通过抑制 PKCδ/SHP-1 依赖的内质网应激,减轻足细胞损伤和炎症,从而缓解糖尿病肾病。

Li Zongze, Tan Di, Lin Jiangong, Zhang Tingwei, Liu Bing, Zhao Bing, Liang Decai, Li Li, Wei Xufeng, Lv Zhimei, Wang Rong, Hu Mengsi

An oral nanocombinatorial agent exhibits pleiotropic improvement in diabetic nephropathy via modulation of the SCAP/SREBPs pathway

一种口服纳米组合制剂通过调节SCAP/SREBPs通路,对糖尿病肾病表现出多效性改善作用。

Su, Yu; Wen, Haobo; Hu, Ke; Wen, Cuiping; Wang, Ping; Zhao, Lulu; Zou, Gang; Jiang, Wei; Chen, Yaxi; Zhao, Yunfei; Li, Qiu

Complement 5a receptor 2 attenuates diabetic kidney disease by promoting mitochondria-associated endoplasmic reticulum membrane formation mediated by PSS-MFN2 interaction.

补体 5a 受体 2 通过促进 PSS-MFN2 相互作用介导的线粒体相关内质网膜形成来减轻糖尿病肾病。

Zhao Yi-Yang, Wang Yi-Hui, Li Zi-Han, Chang Dong-Yuan, Nie Lin, Zhao Ming-Hui, Tang Sydney Chi Wai, Chen Min

PAD4 aggravates diabetic kidney disease by driving macrophage migration

PAD4 通过促进巨噬细胞迁移加剧糖尿病肾病

Hou, Dan; Yu, Tao