日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

3D-printed magnetic scaffolds promote bone and vessel regeneration through CRYAB/PI3K-AKT and NF-κB pathways identified by proteomics

蛋白质组学研究表明,3D打印磁性支架可通过CRYAB/PI3K-AKT和NF-κB通路促进骨骼和血管再生。

Liu, Jieying; Liu, Fuze; Li, Cairong; Li, Zhengyao; Li, Tianle; Wu, Yuanhao; Wu, Di; Huang, Yue; Chen, Hui; Wang, Hai; Lai, Yuxiao; Wu, Zhihong

Single chiral center epimerization-modulated multi-carrier-free oral hydrogel for remolding inflammatory microenvironment via PI3K/Akt/NF-κB and MAPK signaling pathways

单手性中心异构化调控的多载体无口腔水凝胶,可通过PI3K/Akt/NF-κB和MAPK信号通路重塑炎症微环境

Pi, Wenmin; Li, Gen; Qiu, Hailing; Zhao, Yihang; Zhang, Yaozhi; Tan, Xinru; Lu, Jihui; Wang, Zhijia; Huang, Xuemei; Wang, Penglong

SPT6 maintains epidermal homeostasis by inhibiting an NF-κB-positive feedback loop to prevent excessive inflammation

SPT6通过抑制NF-κB正反馈环路来维持表皮稳态,从而防止过度炎症反应。

Sun, Yushuang; Xu, Shuqia; Wang, Dongyu; Gao, Shuiying; Qi, Xiaowen; Jiang, Miao; Jian, Dan; Li, Yaqiong; Jiang, Zhaoyan; Zhen, Miao; Wang, Peng; Shu, Bin; Xie, Julin; Chen, Demeng; Tang, Qing; Li, Jingting

MDA5 regulates BCR signaling and B-cell function via NF-κB-mediated DNM1

MDA5 通过 NF-κB 介导的 DNM1 调节 BCR 信号传导和 B 细胞功能

Luo, Li; Wang, Yi; Fang, Guofeng; Chang, Jiang; Dai, Xin; Gu, Heng; Huang, Yanmei; Miller, Heather; Li, Yue; Chen, Ran; Liu, Ju; Jing, Yukai; Jiang, Panpan; Yang, Lu; Chen, Qianglin; Yang, Jingzhi; Luo, Xi; Kang, Danqing; Liu, Qi; Lai, Juan; Xu, Pengwei; Mao, Huawei; Hu, Xiufen; Du, Xingrong; Lei, Jiahui; Tang, Xiuran; Kuang, Weibing; Wu, Cheng; Li, Zhanguo; Huang, Zhenli; Liu, Wanli; Liu, Chaohong

Adipocyte-Derived Exosomal miR-5099 Mitigates M1 Macrophage Polarization and Adipose Inflammation via c-Met/NF-κB Axis to Improve Metabolic Health

脂肪细胞来源的外泌体 miR-5099 通过 c-Met/NF-κB 轴减轻 M1 巨噬细胞极化和脂肪组织炎症,从而改善代谢健康

Tang, Ping; Tai, Lixin; Xu, Hongxia; Zhu, Dongliang; Li, Jiajia; Feng, Hao; Cui, Jinghui; Lin, Shuhai; Di, Li-Jun; Wang, Li

SV2B Promotes the Progression of TFE3-Rearranged Renal Cell Carcinoma by Interacting with HERC2 to Impede the Degradation of NF-κB Subunits

SV2B通过与HERC2相互作用抑制NF-κB亚基的降解,从而促进TFE3重排肾细胞癌的进展

Feng, Huayi; Xiong, Zhuang; Yu, Shaoqing; Liu, Junxiao; Cao, Shouqing; Liu, Kan; Huang, Xing; Wen, Lequan; Wang, Baojun; Huang, Qingbo; Pan, Tiejun; Zhang, Xu; Ma, Xin; Li, Xiubin

Mechanical Stimuli-Induced Manipulation of Malignant Behavior in Bioprinted Cancer Microtissues via PI3K/NF-κB Activation

通过PI3K/NF-κB激活诱导机械刺激调控生物打印癌微组织中的恶性行为

Lee, Seok-Hyeon; Lee, Jeongho; Choi, Min-Seo; Ahn, Minjun; Yoon, Sik; Lee, Dongjun; Oh, Sae-Ock; Cho, Won-Woo; Kim, Byoung Soo

Lysophosphatidic acid-mediated NF-κB activation promotes FOXC2 expression essential for lymphatic valve development

溶血磷脂酸介导的 NF-κB 激活促进 FOXC2 表达,而 FOXC2 表达对于淋巴瓣膜发育至关重要。

Yasuda, Daisuke; Sato, Nana; Yanagida, Keisuke; Hashidate-Yoshida, Tomomi; Shiiya, Tomohiro; Shindou, Hideo; Taira, Atsuki; Ebihara, Takashi; Shimizu, Takao; Hirashima, Masanori; Mizuno, Seiya; Takahashi, Satoru; Ishii, Satoshi

Correction: Strong constitutive NF-κB signaling in B cells drives SLL/CLL-like lymphomagenesis and overcomes microenvironmental dependencies

更正:B细胞中强烈的组成型NF-κB信号通路驱动SLL/CLL样淋巴瘤的发生,并克服了对微环境的依赖性。

Soberón, Valeria; Osswald, Lena; Moore, Andrew; Sosnowska, Dominika; Swinerd, Gene; Chen, Jingyu; Baygün, Seren; Diehl, Carina; Seyhan, Gönül; Kraus, Laura; Gölling, Vanessa; Trapp, Ricarda; O'Neill, Thomas J; Bortoluzzi, Sabrina; Kovacs, Daniel; Ammon, Tim; Singroul, Pankaj; Hubarzhevska, Yuliia; Öllinger, Rupert; Mueller, Sebastian; Baranov, Olga; Giansanti, Piero; Gillhuber, Felix; Grath, Sonja; Weigert, Oliver; Rosenwald, Andreas; Sasaki, Yoshiteru; Rajewsky, Klaus; Steiger, Katja; Bassermann, Florian; Rad, Roland; Krappmann, Daniel; Ringshausen, Ingo; Schmidt-Supprian, Marc

Coptis chinensis extracellular vesicles loaded with CA1-siRNA promote endothelial repair and stent restenosis therapy by regulating the PADI2 and NF-κB pathway

黄连胞外囊泡负载CA1-siRNA可通过调节PADI2和NF-κB通路促进内皮修复和支架内再狭窄治疗。

Wang, Xinxing; Liu, Yan; Li, Wei; Hao, Juan; Zhao, Zhongjie; Fan, Hua; Wu, Xiumin; Liu, Xiangyu; Xu, Haowen; Yu, Tao; Liu, Mingyuan; Guo, Mingjin; Li, Yongxin