CTCFL相关文献与解析，生知库 | 链接生命科学的每一步

Interaction of CTCF and CTCFL in genome regulation through chromatin architecture during the spermatogenesis and carcinogenesis

CTCF和CTCFL在精子发生和癌变过程中通过染色质结构参与基因组调控的相互作用

期刊:PeerJ

影响因子:2.4

doi:10.7717/peerj.18240

Tong, Xin; Gao, Yang; Su, Zhongjing

Boris knockout eliminates AOM/DSS-induced in situ colorectal cancer by suppressing DNA damage repair and inflammation

Boris 基因敲除可通过抑制 DNA 损伤修复和炎症消除 AOM/DSS 诱发的原位结直肠癌

期刊:Cancer Science

影响因子:4.5

doi:10.1111/cas.15732

Bo-Wen Zuo, Wan-Xin Yao, Meng-Die Fang, Juan Ren, Ling-Lan Tu, Run-Jie Fan, Yan-Mei Zhang

BTApep-TAT peptide inhibits ADP-ribosylation of BORIS to induce DNA damage in cancer

BTApep-TAT肽抑制BORIS的ADP核糖基化从而诱导癌症DNA损伤

期刊:Molecular Cancer

影响因子:27.7

doi:10.1186/s12943-022-01621-w

Yanmei Zhang #, Mengdie Fang #, Shouye Li, Hao Xu, Juan Ren, Linglan Tu, Bowen Zuo, Wanxin Yao, Guang Liang

Author Correction: Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation

作者更正：定义 CTCF 和 CTCFL 对基因组调控的相对和联合贡献

期刊:Genome Biology

影响因子:9.4

doi:10.1186/s13059-020-02056-6

Nishana, Mayilaadumveettil; Ha, Caryn; Rodriguez-Hernaez, Javier; Ranjbaran, Ali; Chio, Erica; Nora, Elphege P; Badri, Sana B; Kloetgen, Andreas; Bruneau, Benoit G; Tsirigos, Aristotelis; Skok, Jane A

BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

BORIS/CTCFL促进黑色素瘤细胞从增殖表型向侵袭表型转变

期刊:Cell Death Discovery

影响因子:6.1

doi:10.1038/s41420-019-0235-x

Sanne Marlijn Janssen ,Roy Moscona ,Mounib Elchebly ,Andreas Ioannis Papadakis ,Margaret Redpath ,Hangjun Wang ,Eitan Rubin ,Léon Cornelis van Kempen ,Alan Spatz

Genome-wide bioinformatics analysis reveals CTCFL is upregulated in high-grade epithelial ovarian cancer

全基因组生物信息学分析显示，CTCFL在高级别上皮性卵巢癌中表达上调。

期刊:Oncology Letters

影响因子:2.5

doi:10.3892/ol.2019.10736

Mi Gong,Changsheng Yan,Yi Jiang,Huangyang Meng,Mingming Feng,Wenjun Cheng

Choice of binding sites for CTCFL compared to CTCF is driven by chromatin and by sequence preference

与 CTCF 相比，CTCFL 结合位点的选择受染色质和序列偏好性的驱动。

期刊:Nucleic Acids Research

影响因子:13.1

doi:10.1093/nar/gky483

Bergmaier, Philipp; Weth, Oliver; Dienstbach, Sven; Boettger, Thomas; Galjart, Niels; Mernberger, Marco; Bartkuhn, Marek; Renkawitz, Rainer

Targeting CTCFL/BORIS for the immunotherapy of cancer

以CTCFL/BORIS为靶点的癌症免疫疗法

期刊:Cancer Immunology Immunotherapy

影响因子:5.1

doi:10.1007/s00262-018-2251-8

Brother of Regulator of Imprinted Sites (BORIS) suppresses apoptosis in colorectal cancer

印迹位点调节因子兄弟 (BORIS) 抑制结直肠癌细胞凋亡

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/srep40786

Yanmei Zhang, Mengdie Fang, Yongfei Song, Juan Ren, Jianfei Fang, Xiaoju Wang

Atractyloside inhibits gefitinib‑resistant non‑small‑cell lung cancer cell proliferation

苍术苷抑制吉非替尼耐药非小细胞肺癌细胞增殖

Interaction of CTCF and CTCFL in genome regulation through chromatin architecture during the spermatogenesis and carcinogenesis

CTCF和CTCFL在精子发生和癌变过程中通过染色质结构参与基因组调控的相互作用

Boris knockout eliminates AOM/DSS-induced in situ colorectal cancer by suppressing DNA damage repair and inflammation

Boris 基因敲除可通过抑制 DNA 损伤修复和炎症消除 AOM/DSS 诱发的原位结直肠癌

BTApep-TAT peptide inhibits ADP-ribosylation of BORIS to induce DNA damage in cancer

BTApep-TAT肽抑制BORIS的ADP核糖基化从而诱导癌症DNA损伤

Author Correction: Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation

作者更正：定义 CTCF 和 CTCFL 对基因组调控的相对和联合贡献

BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

BORIS/CTCFL促进黑色素瘤细胞从增殖表型向侵袭表型转变

Genome-wide bioinformatics analysis reveals CTCFL is upregulated in high-grade epithelial ovarian cancer

全基因组生物信息学分析显示，CTCFL在高级别上皮性卵巢癌中表达上调。

Choice of binding sites for CTCFL compared to CTCF is driven by chromatin and by sequence preference

与 CTCF 相比，CTCFL 结合位点的选择受染色质和序列偏好性的驱动。

Targeting CTCFL/BORIS for the immunotherapy of cancer

以CTCFL/BORIS为靶点的癌症免疫疗法

Brother of Regulator of Imprinted Sites (BORIS) suppresses apoptosis in colorectal cancer

印迹位点调节因子兄弟 (BORIS) 抑制结直肠癌细胞凋亡