日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Correction to "Targeting lncRNA DDIT4-AS1 Sensitizes Triple Negative Breast Cancer to Chemotherapy via Suppressing of Autophagy"

对“靶向lncRNA DDIT4-AS1通过抑制自噬增强三阴性乳腺癌对化疗的敏感性”一文的更正

Human DDIT4L intron retention contributes to cognitive impairment and amyloid plaque formation

人类DDIT4L内含子滞留会导致认知障碍和淀粉样斑块形成。

Li, Kai-Cheng; Shi, Hai-Xiang; Li, Zhen; You, Pu; Pan, Jing; Cai, Yi-Chuan; Li, Jin-Wen; Ma, Xue-Fei; Zhang, Shuo; Diao, Lei; Cai, Bing; Wang, Hai-Bo; Chen, Liang; Mao, Ying; Zhang, Xu

p53 promote oxidative stress, neuroinflammation and behavioral disorders via DDIT4-NF-κB signaling pathway

p53通过DDIT4-NF-κB信号通路促进氧化应激、神经炎症和行为障碍

Zhang, Kaiqi; Zhao, Yongsi; Chen, Xiao; Li, Ye; Lan, Tian; Chang, Mengni; Wang, Wenjing; Wang, Changmin; Zhuang, Xianghua; Zhang, Bin; Yu, Shuyan

Blood DDIT4 and TRIM13 Transcript Levels Mark the Early Stages of Machado-Joseph Disease.

血液中 DDIT4 和 TRIM13 转录水平标志着马查多-约瑟夫病的早期阶段

Ferreira Ana F, Raposo Mafalda, Shaw Emily D, Liu Louisa, Vasconcelos João, Kay Teresa, Bettencourt Conceição, Saraiva-Pereira Maria Luiza, Jardim Laura Bannach, Costa Maria do Carmo, Lima Manuela

Adipose-derived mesenchymal stem cell therapy modulates mitochondrial function to attenuate acetaminophen-induced liver injury by DDIT4/PGC-1α axis

脂肪来源间充质干细胞疗法通过DDIT4/PGC-1α轴调节线粒体功能,从而减轻对乙酰氨基酚引起的肝损伤。

Cen, Yelei; Xia, Caixia; Yao, Shouhan; Weng, Xinyu; Qi, Jinjin; Lou, Guohua; Zheng, Min; Liu, Yanning

The critical role of DNA damage-inducible transcript 4 (DDIT4) in stemness character of leukemia cells and leukemia initiation

DNA损伤诱导转录物4(DDIT4)在白血病细胞干性特征和白血病发生中的关键作用

Li, Yishuang; Cao, Zhijie; Xing, Haiyan; Xue, Zhenya; Liu, Wenbing; Chen, Jiayuan; Mei, Yihan; Gu, Runxia; Wei, Hui; Qiu, Shaowei; Wang, Min; Rao, Qing; Wang, Jianxiang

Correction: A review of the participation of DDIT4 in the tumor immune microenvironment through inhibiting PI3K-Akt/mTOR pathway

更正:DDIT4通过抑制PI3K-Akt/mTOR通路参与肿瘤免疫微环境的综述

Jiao, Yunshu; Xiang, Yang

A review of the participation of DDIT4 in the tumor immune microenvironment through inhibiting PI3K-Akt/mTOR pathway

本文综述了DDIT4通过抑制PI3K-Akt/mTOR通路参与肿瘤免疫微环境的机制。

Jiao, Yunshu; Xiang, Yang

METTL14-mediated m(6)A modification of DDIT4 promotes its mRNA stability in aging-related idiopathic pulmonary fibrosis

METTL14介导的DDIT4 m(6)A修饰促进其在衰老相关特发性肺纤维化中的mRNA稳定性

Li, Dan; Qian, Li; Du, Yufeng; Liu, Lifang; Sun, Ziyue; Han, Yongkang; Guo, Xiangrui; Shen, Chao; Zhang, Zheng; Liu, Xuejun

miR-199a Knockdown Alleviates Insulin Resistance and Inflammation by Targeting DDIT4 via the PI3K/AKT Pathway in vitro and in vivo.

miR-199a 敲低通过 PI3K/AKT 通路靶向 DDIT4,在体外和体内缓解胰岛素抵抗和炎症。

Cai Ya-Wei, Tang Ling-Jia, Zhu Yao, Ye Sen-Sen, Chen Tong-En