日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Structural basis for the recruitment and selective phosphorylation of Akt by mTORC2.

mTORC2 募集和选择性磷酸化 Akt 的结构基础。

Taylor Martin S, Chen Maggie, Hancock Matthew, Wranik Maximilian, Miller Bryant D, O'Meara Timothy R, Palanski Brad A, Ficarro Scott B, Groendyke Brian J, Xiang Yufei, Kondo Kazuma T, Linde-Garelli Karen Y, Lee Michelle J, Mondal Dibyendu, Freund Daniel, Congreve Samantha, Matas Kaay, Hennink Maximiliaan, Xibinaku Kera, Valenstein Max L, van Eeuwen Trevor, Marto Jarrod A, Sali Andrej, Shi Yi, Gray Nathanael S, Sabatini David M, Chu Nam, Rogala Kacper B, Cole Philip A

EIF4A3/circPTGR1/miR-4725-5p positive- feedback loop promotes colorectal cancer progression via FAK/AKT signaling pathway

EIF4A3/circPTGR1/miR-4725-5p正反馈环路通过FAK/AKT信号通路促进结直肠癌进展

Dong, Yi; Ding, Yi-Han; Yang, Xiao; Song, Xiao-Hang; Lei, Xue-Tao; Tang, Ya-Nan; Zhang, Xin-Er; Huang, Wei-Cai; Zhang, Kai; Chen, Jing-Song

3D-printed magnetic scaffolds promote bone and vessel regeneration through CRYAB/PI3K-AKT and NF-κB pathways identified by proteomics

蛋白质组学研究表明,3D打印磁性支架可通过CRYAB/PI3K-AKT和NF-κB通路促进骨骼和血管再生。

Liu, Jieying; Liu, Fuze; Li, Cairong; Li, Zhengyao; Li, Tianle; Wu, Yuanhao; Wu, Di; Huang, Yue; Chen, Hui; Wang, Hai; Lai, Yuxiao; Wu, Zhihong

TLR2-PI3K/Akt mediated microbe-mimetic priming boosts the therapeutic paracrine function of GelMA-Encapsulated MSCs for diabetic wound regeneration.

TLR2-PI3K/Akt 介导的微生物模拟启动增强了 GelMA 封装的 MSC 的治疗性旁分泌功能,用于糖尿病伤口再生。

Tian Feng, Kong Yue, Liu Qinghua, Zhu Shuoji, Guo Xu, Su YanLin, Yu Bingyang, Zhang Chao, Zhu Dongzhen, Li Zhao, Song Wei, Kong Yi, Yin Xiangye, Huang Yuyan, Tan Yaxin, Zhang Mengde, Du Jinpeng, Liang Liting, Li Jianjun, Zhu Ping, Fu Xiaobing, Huang Sha

Harnessing piezoelectric stimulation to modulate PI3K-AKT signaling for intervertebral disc regeneration.

利用压电刺激调节PI3K-AKT信号通路促进椎间盘再生。

The UFL1-AKT positive feedback loop promotes breast cancer progression by enhancing lipid synthesis.

UFL1-AKT正反馈回路通过增强脂质合成促进乳腺癌进展。

Targeting the UFL1-AKT cascade suppresses triple-negative breast cancer progression.

靶向 UFL1-AKT 级联可抑制三阴性乳腺癌的进展。

Bufalin Inhibits the PI3K/AKT Pathway by Targeting GTF3C4 to Impede Breast Cancer Progression

布法林通过靶向GTF3C4抑制PI3K/AKT通路,从而阻碍乳腺癌进展

Guo, Siyu; Chen, Xiaodong; Wang, Haojia; Zhou, Jiying; Lu, Peiying; Liu, Jiangying; Chai, Keyan; Zhang, Jingyuan; Yang, Siyun; Lu, Shan; Gao, Yifei; Jin, Zhengsen; Tao, Xiaoyu; Hu, Zhongdong; Li, Qinglin; Wu, Jiarui

B4GALNT4-Mediated Glycosylation of PDK1 Activates the PI3K-AKT Signaling Pathway to Promote Prostate Cancer Progression

B4GALNT4介导的PDK1糖基化激活PI3K-AKT信号通路促进前列腺癌进展

Jiang, Shaoqin; Li, Mengqiang; Su, Qingfu; Dou, Ruiling; Lin, Shaoshan; Zhang, Jili; Sun, Xiaochen; Yu, Haolan; Bao, Wei; Qu, Min; Wang, Yan; Yang, Chenghua; Gao, Xu

PEAKS activation by PROTAC EPIC-0726 potentiates temozolomide in glioblastoma via K63/K48 ubiquitination-dependent ERK/AKT suppression and p21 stabilization

PROTAC EPIC-0726 通过 K63/K48 泛素化依赖的 ERK/AKT 抑制和 p21 稳定增强 PEAKS 激活,从而增强替莫唑胺在胶质母细胞瘤中的疗效。

Hong, Biao; Su, Dongyuan; Cui, Xiaoteng; Zhao, Jixing; Wang, Qixue; Zhan, Qi; Yang, Eryan; Yang, Shixue; Ju, Jiasheng; Huang, Yanping; Cheng, Chunchao; Ding, Yaqing; Xu, Hanyi; Cui, Longtao; Zhao, Yilin; Zhao, Xun; Liang, Siwen; Liu, Yuhao; Kang, Chunsheng