日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

PFOS Disrupts Oocyte Maturation and Early Embryonic Development via Ovarian FOXK1 O-GlcNAcylation in Mice.

PFOS 通过卵巢 FOXK1 O-GlcNAc 化干扰小鼠卵母细胞成熟和早期胚胎发育。

Han Shuwen, Yuan Qin, Wu Zhu, Fang Yaohui, Qian Hong, Zhu Jiale, Zhang Yuchen, Deng Ke, Su Liangliang, Xu Haibo, Shu Haotian, Gong Yiming, Xu Qiaoqiao, Du Guizhen, Wu Di, Fan Yun, Lu Chuncheng

Targeting the FOSL1/IKKα positive feedback loop attenuates glioblastoma malignancy via suppression of NF-κB signaling.

靶向 FOSL1/IKKα 正反馈回路可通过抑制 NF-α 信号传导来减弱胶质母细胞瘤的恶性程度。

Toll-like receptor signaling outcome is determined by the stoichiometry of the endogenous TRIFosome.

Toll 样受体信号传导的结果取决于内源性 TRIFosome 的化学计量比。

Moncrieffe Martin C, Suresh Prasanna, Boyle Joe, Cui Yuhao, Nawalpuri Bharti, Verstak Brett, Zhang Yu P, Zhang Ziwei, Taylor Marcus, Egelman Edward H, Gay Nicholas, Klenerman David, Bryant Clare

Corrigendum to "A viral-host redox axis: EBNA1-FOSL2-ALDH3A1 defines a targetable vulnerability in EBV-positive carcinomas" [Redox Biol. 90 (2026) 104025]

对“病毒-宿主氧化还原轴:EBNA1-FOSL2-ALDH3A1 定义了 EBV 阳性癌中的可靶向脆弱性”的更正 [Redox Biol. 90 (2026) 104025]

Liu, Qian; Liu, Binliang; Liu, Zhenbao; Gong, Lidan; Li, Min; Luo, Xiangjian

A viral-host redox axis: EBNA1-FOSL2-ALDH3A1 defines a targetable vulnerability in EBV-positive carcinomas

病毒-宿主氧化还原轴:EBNA1-FOSL2-ALDH3A1 定义了 EBV 阳性癌中一个可靶向的脆弱点

Liu, Qian; Liu, Binliang; Liu, Zhenbao; Gong, Lidan; Li, Min; Luo, Xiangjian

FOS and JUN regulate oxidative stress and steroidogenesis in human aldosterone-producing adenomas.

FOS 和 JUN 调节人类醛固酮生成腺瘤中的氧化应激和类固醇生成。

Wei Jia, Duregon Eleonora, Papotti Mauro G, Knösel Thomas, Bidlingmaier Martin, Sbiera Silviu, Reincke Martin, Williams Tracy Ann

Novel Insights into the Distribution and Effects of Perfluorooctanesulfonic acid (PFOS) in the Nervous System of a Frog Tadpole Model by Mass Spectrometry Imaging

利用质谱成像技术对蛙蝌蚪模型神经系统中全氟辛烷磺酸(PFOS)的分布和影响有了新的认识

Poulsen, Rikke; Field, Emma M; Jackson, Angela M; Kuecks-Winger, Haley; Thambirajah, Anita A; Goodlett, David R; Helbing, Caren C; Pětrošová, Helena

Perfluorooctane sulfonate (PFOS) in follicular fluid and human granulosa cell dysfunction: a physiologically based toxicokinetic model translation of long-term low-level in vitro exposure data

全氟辛烷磺酸(PFOS)在卵泡液和人颗粒细胞功能障碍中的作用:基于生理学的毒代动力学模型转化长期低剂量体外暴露数据

Tomanic, Tamara; Samardzija Nenadov, Dragana; Radovic Pletikosic, Sava; Stanic, Bojana; Obradovic, Darija; Lazovic, Sasa; Andric, Nebojsa

Gut microbial ammonia as a mediator of PFOS neurotoxicity and its remediation by the flavonoid Icaritin.

肠道微生物氨作为 PFOS 神经毒性的介质及其通过类黄酮淫羊藿苷的修复作用。

Yi Yang, Zhang Wenfang, Wei Yu, Ran Wang, Liu Dongjing, Deng Weikun, Duan Songyuan, Yao Jiyong, Wang Lianhang, Zhang Yuandong, Gao Jianmei, Gong Qihai

Inhibition of FOS-Like Antigen 1 Reduces Chemoresistance to Temozolomide Through Stemness Reprogramming via IL-6/STAT3(Tyr705) Pathway.

抑制 FOS 样抗原 1 通过 IL-6/STAT3(Tyr705) 通路进行干性重编程,从而降低对替莫唑胺的化疗耐药性。

Sim JeongMin, Park JeongMan, Heo JinHyung, Kim Yu Jin, Kim Dongkil, Jeon Yong Hyun, Lee Jae-Eon, Choi Seon Hee, Kim Hak Jin, Hwang So Jung, Kim Junhyung, Cho Kyung Gi, Yoo Jihwan, Moon Ju Hyung, Moon Jong-Seok, Sung Kyoung Su, Lim Jaejoon