日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

A tumor-promoting inflammatory SPP1+ macrophage-IL-6-CRP axis drives immune dysfunction in bladder cancer

肿瘤促进性炎症SPP1+巨噬细胞-IL-6-CRP轴驱动膀胱癌免疫功能障碍

Tran, Michelle A; Cho, Byuri Angela; Izadmehr, Sudeh; Yoo, Seong-Keun; Youssef, Dina; Anker, Jonathan F; Farkas, Adam M; Figueiredo, Igor; Lee, Karen; Ananthanarayanan, Aparna; Balan, Sreekumar; Onkar, Sayali; Banchereau, Romain; Gupta, Saurabh; Chhibber, Aparna; Wang, Li; Beaumont, Kristin G; Li, Ziao; Garcia-Barros, Monica; Bicak, Mesude; Cordon-Cardo, Carlos; Brody, Rachel; Kim-Schulze, Seunghee; Yuen, Kobe; Mariathasan, Sanjeev; Atiq, Saad O; Adeyelu, Tolulope T; Elliott, Andrew; Barata, Pedro; McKay, Rana R; Gnjatic, Sacha; Gonzalez-Kozlova, Edgar; Mehrazin, Reza; Horowitz, Amir; Sfakianos, John P; Chowell, Diego; Galsky, Matthew D; Bhardwaj, Nina

P-1555. Plasma proteomics demonstrate association between IL-6 and expansion of an immunosuppressive monocyte substate during sepsis

P-1555. 血浆蛋白质组学研究表明,脓毒症期间IL-6与免疫抑制性单核细胞亚群的扩增相关。

Ishii, Takefumi; Wang, Jiapeng; Zhang, Wei; Mascarenhas, John; Hoffman, Ronald; Dai, Ying; Wisch, Nathaniel; Xu, Mingjiang; Ankomah, Pierre; DuBois, Alyssa; Bhattacharyya, Roby P

Multi-omics-informed hydrogel design: modulating IL-6 to reduce endoplasmic reticulum stress in bone regeneration.

基于多组学信息的凝胶设计:调节IL-6以减少骨再生中的内质网应激。

Zhou Jiannan, Dai Jingtao, Hu Shixian, Qi Cancan, Chen Jiahao, Zhang Wentai, Alexander Dorothea, Li An, Xiao Yin, Li Ping

TRIM32 promotes anoikis resistance and metastasis in NSCLC by degrading CHEK2 to enhance IL-6 secretion.

TRIM32 通过降解 CHEK2 来增强 IL-6 的分泌,从而促进 NSCLC 的抗凋亡和转移。

Xu Jia, Liu Wenjun, Chen Li, Zhang Juan, Zhang Dongze, Huang Xue, Zhang Guangbo

FomA-Containing Outer Membrane Vesicles of Fusobacterium Nucleatum Facilitate Bladder Cancer Lymphatic Metastasis via IL-6-Dependent M2b Macrophage Polarization

具核梭杆菌含Foma的外膜囊泡通过IL-6依赖性M2b巨噬细胞极化促进膀胱癌淋巴转移

Shangguan, Wentai; Li, Weijia; Huang, Wenxue; Wu, Jilin; Yu, Yao; Huang, Yiyao; Yang, Lin; Xie, Ming; Yang, Qishen; Zheng, Jun; Zhu, Yuexuan; Sun, Qi; Li, Biao; Li, Leqian; Wang, Zongwei; Zhao, Jie; Wu, Peng; Cheng, Bisheng

Corrigendum to "E3 ubiquitin ligase RNF128 attenuates colitis and colorectal tumorigenesis by triggering the degradation of IL-6 receptors" [J. Adv. Res. 72 (2025) 107-120]

对“E3泛素连接酶RNF128通过触发IL-6受体的降解来减弱结肠炎和结直肠肿瘤发生”的更正[J. Adv. Res. 72 (2025) 107-120]

He, Tian Sheng; Cai, Kuntai; Lai, Weiling; Yu, Jingge; Qing, Furong; Shen, Ao; Sui, Lina; He, Wenji; Wang, Weihua; Xiao, Qiuxiang; Lei, Xiong; Guo, Tianfu; Liu, Zhiping

Endothelial-targeted modification of ginseng-derived exosomes for IL-6 SiRNA delivery ameliorates hepatic ischemia-reperfusion injury.

内皮细胞靶向修饰人参来源的外泌体以递送IL-6 siRNA可改善肝脏缺血再灌注损伤。

Huang Kaijun, Zhang Xuanlin, Yan Xufeng, Qin Meiting, Lv Linpei, Yang Shuangqin, Zheng Yang, Yin Guohao, Tian Xinyu, Wang Xueyun, Deng Wenjing, Yan Yaning, Wu Wenjing, Yuan Haoxiang, Fan Zhijin, Wang Chunming, Ma Jian

NFκB/IL-6/JAK2/STAT axis in myelofibrosis is a key vulnerability that is targetable and relevant to JAK2 inhibitor treatment resistance

骨髓纤维化中的NFκB/IL-6/JAK2/STAT通路是一个关键的脆弱点,可作为靶点,并与JAK2抑制剂治疗耐药性相关。

Bian, Huiqin; Gangat, Naseema; Shen, Na; Pang, Jiuxia; Wald, David; Tse, William; Tefferi, Ayalew; Yan, Fei; Liu, Shujun

Effect of IL-6 receptor inhibition on infarct volume after endovascular treatment for ischaemic stroke: a phase 2, randomised, placebo-controlled trial

IL-6受体抑制剂对缺血性卒中血管内治疗后梗死体积的影响:一项II期随机、安慰剂对照试验

Chu, Xuehong; Lan, Jing; Ma, Zhengfei; Yin, Yunjian; Gu, Hongqiu; Baron, Jean Claude; Liesz, Arthur; Wen, Changming; Liu, Yifeng; Sun, Jun; Wang, Ning; Li, Chaoqun; Feng, Xiangyang; Li, Jianqiao; Wang, Benxiao; Feng, Yan; Wu, Chunxue; Dong, Xiao; Zhou, Chen; Li, Chuanhui; Zhao, Wenbo; Liu, Guiyou; Fisher, Marc; Liebeskind, David S; Fang, Mingde; Jia, Xiaole; Ma, Hongrui; Ji, Xunming; Wu, Chuanjie

Inhibition of FOS-Like Antigen 1 Reduces Chemoresistance to Temozolomide Through Stemness Reprogramming via IL-6/STAT3(Tyr705) Pathway.

抑制 FOS 样抗原 1 通过 IL-6/STAT3(Tyr705) 通路进行干性重编程,从而降低对替莫唑胺的化疗耐药性。

Sim JeongMin, Park JeongMan, Heo JinHyung, Kim Yu Jin, Kim Dongkil, Jeon Yong Hyun, Lee Jae-Eon, Choi Seon Hee, Kim Hak Jin, Hwang So Jung, Kim Junhyung, Cho Kyung Gi, Yoo Jihwan, Moon Ju Hyung, Moon Jong-Seok, Sung Kyoung Su, Lim Jaejoon