日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

SLC25A11 Is Associated with KDM2A-Dependent Reduction in rRNA Transcription Induced by Aminooxyacetic Acid

SLC25A11 与氨氧乙酸诱导的 KDM2A 依赖性 rRNA 转录减少有关

Tanaka, Yuji; Miyazawa, Nagisa; Toba, Yuuki

A novel KDM2A::YAP1 fusion in a pediatric supratentorial CNS neoplasm resembling a tumor with BCOR internal tandem duplication

在儿童幕上中枢神经系统肿瘤中发现一种新型的KDM2A::YAP1融合基因,该肿瘤类似于具有BCOR内部串联重复的肿瘤。

Tauziède-Espariat, Arnault; Métais, Alice; Bochaton, Dorian; Servant, Euphrasie; Chotard, Guillaume; Le Quang, Mégane; Bonhomme, Benjamin; Truffaux, Nathalène; Dangouloff-Ros, Volodia; Boddaert, Nathalie; Hasty, Lauren; Gimbert, Edouard; Varlet, Pascale

Histone demethylase KDM2A recruits HCFC1 and E2F1 to orchestrate male germ cell meiotic entry and progression

组蛋白去甲基化酶KDM2A募集HCFC1和E2F1,从而调控雄性生殖细胞减数分裂的进入和进程。

Shenglei Feng #,Yiqian Gui #,Shi Yin #,Xinxin Xiong,Kuan Liu,Jinmei Li,Juan Dong,Xixiang Ma,Shunchang Zhou,Bingqian Zhang,Shiyu Yang,Fengli Wang,Xiaoli Wang,Xiaohua Jiang,Shuiqiao Yuan

DUX4-induced HSATII transcription causes KDM2A/B-PRC1 nuclear foci and impairs DNA damage response

DUX4诱导的HSATII转录导致KDM2A/B-PRC1核内聚集,并损害DNA损伤反应。

Tessa Arends ,Hiroshi Tsuchida ,Richard O Adeyemi ,Stephen J Tapscott

The Role of KDM2A and H3K36me2 Demethylation in Modulating MAPK Signaling During Neurodevelopment

KDM2A和H3K36me2去甲基化在神经发育过程中调节MAPK信号通路中的作用

Zongyao Ren ,Haiyan Tang ,Wendiao Zhang ,Minghui Guo ,Jingjie Cui ,Hua Wang ,Bin Xie ,Jing Yu ,Yonghao Chen ,Ming Zhang ,Cong Han ,Tianyao Chu ,Qiuman Liang ,Shunan Zhao ,Yingjie Huang ,Xuelian He ,Kefu Liu ,Chunyu Liu ,Chao Chen

The Unique Role of the Second Coordination Sphere to Unlock and Control Catalysis in Nonheme Fe(II)/2-Oxoglutarate Histone Demethylase KDM2A

第二配位层在非血红素Fe(II)/2-酮戊二酸组蛋白去甲基酶KDM2A催化中的独特作用

Thomas, Midhun George; Jaber Sathik Rifayee, Simahudeen Bathir; Chaturvedi, Shobhit S; Gorantla, Koteswara Rao; White, Walter; Wildey, Jon; Schofield, Christopher J; Christov, Christo Z

Histone demethylase KDM2A is a selective vulnerability of cancers relying on alternative telomere maintenance

组蛋白去甲基化酶KDM2A是依赖替代性端粒维持机制的癌症的一种选择性弱点。

Li, Fei; Wang, Yizhe; Hwang, Inah; Jang, Ja-Young; Xu, Libo; Deng, Zhong; Yu, Eun Young; Cai, Yiming; Wu, Caizhi; Han, Zhenbo; Huang, Yu-Han; Huang, Xiangao; Zhang, Ling; Yao, Jun; Lue, Neal F; Lieberman, Paul M; Ying, Haoqiang; Paik, Jihye; Zheng, Hongwu

Lysine Demethylase KDM2A Promotes Proteasomal Degradation of TCF/LEF Transcription Factors in a Neddylation-Dependent Manner

赖氨酸脱甲基酶 KDM2A 以 Neddylation 依赖的方式促进 TCF/LEF 转录因子的蛋白酶体降解

Tijana Šopin 34wwq3,František Liška, Tomáš Kučera, Dušan Cmarko, Tomáš Vacík

KDM2A Targets PFKFB3 for Ubiquitylation to Inhibit the Proliferation and Angiogenesis of Multiple Myeloma Cells

KDM2A通过泛素化靶向PFKFB3来抑制多发性骨髓瘤细胞的增殖和血管生成

Liu, Xinling; Li, Jiaqiu; Wang, Zhanju; Meng, Jie; Wang, Aihong; Zhao, Xiaofei; Xu, Qilu; Cai, Zhen; Hu, Zhenbo

Histone demethylase KDM2A: Biological functions and clinical values (Review)

组蛋白去甲基化酶KDM2A:生物学功能和临床价值(综述)

Liu, Lisheng; Liu, Jiangnan; Lin, Qinghai