日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

KIN17 facilitates the initiation and progression of renal tumor progression through the PI3K-AKT-mTOR pathway.

KIN17 通过 PI3K-AKT-mTOR 通路促进肾肿瘤的发生和发展。

Wen Yibo, Lyu Lei, Zhang He, Zhang Yanping, Yang Shuai, Wang Qingwei, Song Yue, Wen Jianguo

DNA/RNA-binding protein KIN17 supports esophageal cancer progression via resolving noncanonical STING activation induced by R-loop

DNA/RNA结合蛋白KIN17通过解除R环诱导的非经典STING激活来促进食管癌的进展。

Zichao Wei # ,Ning Zhao # ,Lu Kuang ,Ji Cong ,Sujuan Zheng ,Yi Li ,Zhihua Liu

Correction: KIN17 modulates the WNT/β-catenin pathway and epithelial mesenchymal transition in non-small cell lung cancer

更正:KIN17 调节非小细胞肺癌中的 WNT/β-catenin 通路和上皮间质转化

Peng, Panli; Li, Xukai; Su, Zhanfeng; Chen, Hao; Lv, Junhong

KIN17 modulates the WNT/β-catenin pathway and epithelial mesenchymal transition in non-small cell lung cancer

KIN17 调控非小细胞肺癌中的 WNT/β-catenin 通路和上皮间质转化

Peng, Panli; Li, Xukai; Su, Zhanfeng; Chen, Hao; Lv, Junhong

Regulation of pancreatic cancer cells by suppressing KIN17 through the PI3K/AKT/mTOR signaling pathway

通过 PI3K/AKT/mTOR 信号通路抑制 KIN17 来调控胰腺癌细胞

Qiuyan Li #, Yuxia Yang #, Xiaocong Lin #, Lok Ting Chu, Helian Chen, Linsong Chen, Jinjing Tang, Tao Zeng

Onvansertib and Navitoclax Combination as a New Therapeutic Option for Mucinous Ovarian Carcinoma

Onvansertib 与 Navitoclax 联合治疗作为粘液性卵巢癌的新治疗选择

Serena Petrella, Marika Colombo, Mirko Marabese, Chiara Grasselli, Andrea Panfili, Michela Chiappa, Valentina Sancisi, Ilaria Craparotta, Maria C Barbera, Giada A Cassanmagnago, Marco Bolis, Giovanna Damia

Roles and regulatory mechanisms of KIN17 in cancers (Review)

KIN17在癌症中的作用及调控机制(综述)

Huang, Xueran; Dai, Zichang; Li, Qiuyan; Lin, Xiaocong; Huang, Qiyuan; Zeng, Tao

A genetic screen in C. elegans reveals roles for KIN17 and PRCC in maintaining 5' splice site identity

秀丽隐杆线虫的遗传筛选揭示了KIN17和PRCC在维持5'剪接位点特性中的作用

Suzuki, Jessie M N G L; Osterhoudt, Kenneth; Cartwright-Acar, Catiana H; Gomez, Destiny R; Katzman, Sol; Zahler, Alan M

Deficiency of kin17 Facilitates Apoptosis of Cervical Cancer Cells by Modulating Caspase 3, PARP, and Bcl-2 Family Proteins

kin17 缺乏通过调节 Caspase 3、PARP 和 Bcl-2 家族蛋白促进宫颈癌细胞凋亡

Bingsen Su, Meifeng Zhong, Yuzhao Zhang, Kunhe Wu, Qiyuan Huang, Chuiyu Zhu, Tao Zeng

Interactome Analysis of KIN (Kin17) Shows New Functions of This Protein

KIN(Kin17)的相互作用组分析揭示了该蛋白质的新功能

Vanessa Pinatto Gaspar, Anelise Cardoso Ramos, Philippe Cloutier, José Renato Pattaro Junior, Francisco Ferreira Duarte Junior, Annie Bouchard, Flavio Augusto Vicente Seixas, Benoit Coulombe, Maria Aparecida Fernandez