日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

JAM-C prevents ocular fibrosis by suppressing the TAZ/KLF6 pathway.

JAM-C 通过抑制 TAZ/KLF6 通路来预防眼纤维化。

Chen Min, Li Wanhong, Du Yuxiang, Zhao Yuanlong, Guo Ying, Li Ying, Wang Xiaolu, Huang Lijuan, Zeng Xiaoling, Zhang Yihan, Huang Guanqun, Wang Shasha, Kuang Haiqing, Sun Guangli, Jiang Qin, Li Xuri, Lu Weisi

Network medicine modeling of the m⁶A regulatory landscape identifies a KLF6-WTAP axis as a therapeutic target in pulmonary fibrosis.

网络医学模型揭示了调控格局,并将 KLF6-WTAP 轴确定为肺纤维化的治疗靶点。

Xu Chengyuan, Zhou Ziheng, Lin Yani, Zhang Siqi, Cai Shanshan, Li Bing, Wang Zhifang

Perturbation of BRD4 and p300 activity suppresses super enhancer‑driven KLF6 expression in renal carcinoma

BRD4和p300活性的扰动抑制肾癌中超级增强子驱动的KLF6表达

Zamberi, Nurul Nadia Mohamad; Abuhamad, Asmaa Y; Yusuf, Siti Nur Hasanah Mohd; Rahman, Nur Qurratu Athirah A; Mohtar, Mohamad Aimanuddin; Syafruddin, Saiful Effendi

The possible renoprotective effect of denatonium benzoate in a rat model of type 2 diabetes: role of Krüppel-like factor 6 (KLF6)

苯甲酸地那铵在 2 型糖尿病大鼠模型中可能具有肾脏保护作用:Krüppel 样因子 6 (KLF6) 的作用

Zakaria, Hend S; El-Fetiany, Ola S; Omar, Walaa; Roushdy, Walaa N

Pivotal contribution of super-enhancer-driven KLF6 expression to the adipogenesis of human adipose-derived stem cells

超级增强子驱动的KLF6表达对人脂肪干细胞脂肪生成的关键作用

Nguyen, Mai-Phuong; Yamagata, Kaoru; Nguyen, Anh Phuong; Zhang, Tong; Sakai, Hidenori; Ngo, Uyen Thi; Shan, Yu; Otsuka, Takashi; Huong, Ngo Thi Dieu; Aritomi, Takafumi; Yuan, Meng; Kato, Shigeaki; Tanaka, Yoshiya; Nakayamada, Shingo

The KLF6-RPB1 Pathway: Setting the Transcriptional Stage for Failed Tubular Recovery

KLF6-RPB1通路:为肾小管恢复失败奠定转录基础

Vallon, Volker

Circ-AARS plays an important role during the odontogenic differentiation of dental pulp stem cells by modulating miR-24-3p/KLF6 expression

Circ-AARS通过调节miR-24-3p/KLF6的表达,在牙髓干细胞的成牙分化过程中发挥重要作用。

Sui, Meizhi; Lyu, Jiaxuan; Zhou, Jiaxin; Liao, Qian; Xiao, Zexu; Jin, Mingming; Tao, Jiang

TET1-Mediated DNA hydroxymethylation of KLF6 promotes LPS-induced pulmonary fibrosis by activating the S1PR3/RhoA/ROCK signaling pathway.

TET1 介导的 KLF6 DNA 羟甲基化通过激活 S1PR3/RhoA/ROCK 信号通路促进 LPS 诱导的肺纤维化。

Chen Jiangchuan, Chen Lingjia, Wang Zeqiang, Cao Jun, Yang Huan, Li Jianmin

Correction: KLF6 activates Sp1-mediated prolidase transcription during TGF-β1 signaling

更正:KLF6 在 TGF-β1 信号传导过程中激活 Sp1 介导的脯氨酸酶转录

Eni-Aganga, Ireti; Lanaghan, Zeljka Miletic; Ismail, Farah; Korolkova, Olga; Goodwin, Jeffery Shawn; Balasubramaniam, Muthukumar; Dash, Chandravanu; Pandhare, Jui

Cholangiocyte-derived exosomal miR-381-3p promotes hepatic stellate cell activation and cholestatic liver fibrosis via targeting Klf6.

胆管细胞来源的外泌体 miR-381-3p 通过靶向 Klf6 促进肝星状细胞活化和胆汁淤积性肝纤维化。

Gao Yang, Chen Yonglin, Mang Yuanyi, Zhang Xibing, Li Xiaoshan, Zhang Shengning