日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Preliminary Anti-Melanoma Activity of a Chlorogenic Acid-Based PROTAC Targeting MDM4, a Candidate Protein Identified by Proteomics

基于绿原酸的PROTAC靶向MDM4(一种通过蛋白质组学鉴定的候选蛋白)的初步抗黑色素瘤活性

Mei, Zhiting; Sun, Jiali; Zhao, Pengfei; Luo, Yiming; Niu, Jine; Huang, Danfei

MDM4 at the Crossroads: Beyond p53 and MDM2

MDM4 的十字路口:超越 p53 和 MDM2

Thapa, Dipesh; St John, Allison; Parrales, Alejandro; Ranjan, Atul; Iwakuma, Tomoo

MDM4 overexpression alleviates developmental and hematopoietic defects in Fancg deficient mice

MDM4 过表达可缓解 Fancg 缺陷小鼠的发育和造血缺陷。

Loock, Maeva; Fédronie, Carèle; Torset, Carine; Hernandez, Lucie; Latour, Emmanuelle; Quentin, Samuel; Gachard, Nathalie; Lefrançois, Julie; Maillard, Loïc; Langa Vives, Francina; Parietti, Véronique; Arcangeli, Marie-Laure; Lobry, Camille; Soulier, Jean; Bluteau, Olivier; Bluteau, Dominique

Correction for AbuHammad et al., Regulation of PRMT5-MDM4 axis is critical in the response to CDK4/6 inhibitors in melanoma

对 AbuHammad 等人的论文“PRMT5-MDM4 轴的调控在黑色素瘤对 CDK4/6 抑制剂的反应中至关重要”的更正

Targeting the MDM2-MDM4 interaction interface reveals an otherwise therapeutically active wild-type p53 in colorectal cancer

靶向MDM2-MDM4相互作用界面揭示了结直肠癌中一种具有治疗活性的野生型p53

Sonia Valentini,Giada Mele,Marika Attili,Maria Rita Assenza,Fulvio Saccoccia,Francesca Sardina,Cinzia Rinaldo,Roberto Massari,Nicola Tirelli,Alfredo Pontecorvi,Fabiola Moretti

RETRACTION: Mutual Regulation of MDM4 and TOP2A in Cancer Cell Proliferation

撤稿:MDM4和TOP2A在癌细胞增殖中的相互调控

Structure and function of MDM2 and MDM4 in health and disease

MDM2和MDM4在健康和疾病中的结构和功能

Zhu, Ivy Yiyi; Lloyd, Alec; Critchley, William R; Saikia, Queen; Jade, Dhananjay; Divan, Aysha; Zeqiraj, Elton; Harrison, Michael A; Brown, Christopher J; Ponnambalam, Sreenivasan

MDM4 enables efficient human iPS cell generation from PBMCs using synthetic RNAs

MDM4能够利用合成RNA从外周血单核细胞高效生成人类诱导多能干细胞。

Masato Nakagawa # ,Mizuho Nogi # ,Hatsuki Doi ,Ryuhei Hayashi ,Tomohiko Katayama ,Hirohisa Ohno ,Megumi Mochizuki ,Karin Hayashi ,Hirohide Saito

[Retracted] Tan IIA inhibits H1299 cell viability through the MDM4‑IAP3 signaling pathway

【已撤稿】Tan IIA 通过 MDM4-IAP3 信号通路抑制 H1299 细胞活力

Zu, Yukun; Wang, Jianning; Ping, Wei; Sun, Wei

Erratum: MiR-1205 functions as a tumor suppressor by disconnecting the synergy between KRAS and MDM4/E2F1 in non-small cell lung cancer

更正:miR-1205通过阻断KRAS和MDM4/E2F1在非小细胞肺癌中的协同作用发挥抑癌作用。

Yan, Hong; Chen, Xiaoying; Li, Yu; Fan, Lei; Tai, Yusi; Zhou, Yang; Chen, Yuxiang; Qi, Xinming; Huang, Ruimin; Ren, Jin