日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Thyroid Hormone Receptor β1 and PGC1α Coordinately Regulate OPA1/MFN2-Mediated Mitochondrial Fusion and UCP1-Mediated Lipid Browning in ccRCC.

甲状腺激素受体β1和PGC1α协同调节ccRCC中OPA1/MFN2介导的线粒体融合和UCP1介导的脂质褐变。

Meng Xiangui, Yu Tiexi, Lv Fang, Li Weiquan, Yang Hongmei, Zhang Xiaoping, Xiao Wen

Complement 5a receptor 2 attenuates diabetic kidney disease by promoting mitochondria-associated endoplasmic reticulum membrane formation mediated by PSS-MFN2 interaction.

补体 5a 受体 2 通过促进 PSS-MFN2 相互作用介导的线粒体相关内质网膜形成来减轻糖尿病肾病。

Zhao Yi-Yang, Wang Yi-Hui, Li Zi-Han, Chang Dong-Yuan, Nie Lin, Zhao Ming-Hui, Tang Sydney Chi Wai, Chen Min

RIPK3-driven phosphorylation of MFN2 orchestrates endoplasmic reticulum-mitochondria interaction and cardiomyocyte stress responses.

RIPK3 驱动的 MFN2 磷酸化协调内质网-线粒体相互作用和心肌细胞应激反应。

Wang Yu, Xu Tao, Li Qi, Ye Lin, Wang Peiyan, Wang Puhan, Song Yihan, Ao Xiang, Wang Jianxun, Ding Wei

Arginine ameliorates motor and survival deficits in MFN2-Deficient Drosophila models

精氨酸可改善MFN2缺陷果蝇模型中的运动和生存缺陷

Ando, Masahiro; Okamoto, Yuji; Higuchi, Yujiro; Yuan, Jun-Hui; Yoshimura, Akiko; Yano, Chikashi; Nagatomo, Risa; Hobara, Takahiro; Kojima, Fumikazu; Hiramatsu, Yu; Nozuma, Satoshi; Sakiyama, Yusuke; Takashima, Hiroshi

Correction: Bavachin ameliorates cisplatin-induced nephrotoxicity by enhancing mitochondrial β-oxidation and lipid metabolism through MFN2

更正:Bavachin 通过 MFN2 增强线粒体 β-氧化和脂质代谢,从而减轻顺铂引起的肾毒性。

Luo, Shilu; Yang, Ming; Jiang, Na; Li, Chenrui; Liu, Yan; Sun, Lin

ZEB1 maintains mitochondrial fission and macrophage efferocytosis by restraining MFN2, thereby limiting inflammation and improving tendon‑bone healing.

ZEB1 通过抑制 MFN2 来维持线粒体分裂和巨噬细胞吞噬作用,从而限制炎症并改善肌腱骨愈合。

Zhao Yan, Zhang Yuankai, Lei Tian, Zhang Shangqing, Nan Kai, Zhang Xin, Fan Li-Hong

Correction to: Inhibition of USP30 Promotes Mitophagy by Regulating Ubiquitination of MFN2 by Parkin to Attenuate Early Brain Injury after SAH

更正:USP30抑制通过Parkin调控MFN2的泛素化促进线粒体自噬,从而减轻蛛网膜下腔出血后的早期脑损伤。

Liu, Yang; Yao, Chenbei; Sheng, Bin; Zhi, Simin; Chen, Xiangxin; Ding, Pengfei; Zhang, Jiatong; Tao, Zhennan; Li, Wei; Zhuang, Zong; Mao, Jiannan; Peng, Zheng; Yan, Huiying; Jin, Wei

Nationwide Characterization of MFN2-Related CMT in 176 Japanese Patients: Clinical and Genetic Insights

对176例日本MFN2相关CMT患者的全国性特征分析:临床和遗传学见解

Ando, Masahiro; Higuchi, Yujiro; Yuan, Jun-Hui; Yoshimura, Akiko; Yano, Chikashi; Hobara, Takahiro; Kojima, Fumikazu; Hiramatsu, Yu; Nozuma, Satoshi; Nakamura, Tomonori; Sakiyama, Yusuke; Mitsui, Jun; Tsuji, Shoji; Takashima, Hiroshi

High‑intensity exercise training inhibits excessive autophagy in the hyperlipidemic myocardium of ApoE(‑/‑) mice via the NAD(+)‑mediated SIRT1/MFN2 pathway.

高强度运动训练通过 NAD(+)介导的 SIRT1/MFN2 通路抑制 ApoE(â€'/â€') 小鼠高脂血症心肌中的过度自噬。

Gao Shan, Yao Wei, Yang Jin, Liu Yujie, Pei Zuowei

Mitofusin MFN2 acts as a molecular sensor preventing protein aggregation and mitophagy, with a protective effect against apoptosis in Charcot-Marie-Tooth type 2A disease

线粒体融合蛋白MFN2作为一种分子传感器,可防止蛋白质聚集和线粒体自噬,从而对夏科-马里-图斯病2A型中的细胞凋亡具有保护作用。

Joaquim, Mariana; Dohrn, Maike F; Chevrollier, Arnaud; Escobar-Henriques, Mafalda