日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

NEDD 4 Pyre-fighters to extinguish inflammation: NEDD4L ubiquitinates Gasdermin D and Gasdermin E to dampen pyroptosis

NEDD4 是对抗炎症的“火药杀手”:NEDD4L 通过泛素化 Gasdermin D 和 Gasdermin E 来抑制细胞焦亡。

Gulshan, Kailash

NEDD4L-mediated Gasdermin D and E ubiquitination regulates cell death and tissue injury.

NEDD4L 介导的 Gasdermin D 和 E 泛素化调节细胞死亡和组织损伤。

Shah Sonia S, Manning Jantina A, Lim Yoon, Sinha Diva, Murthy Ambika Mosale Venkatesh, Ganesan Raja, Robinson Nirmal, Alnemri Emad S, Masters Seth L, Vince James E, Kumar Sharad

PHB2 ameliorates ferroptosis and aortic aneurysm/dissection through NEDD4L-dependent ubiquitination of NCOA4.

PHB2 通过 NEDD4L 依赖的 NCOA4 泛素化来改善铁死亡和主动脉瘤/夹层。

Xiong Shengjun, Lin Jie, An Ying, Du Yuxin, Li Yiran E, Du Zunhui, Zou Rongjun, Abudureyimu Miyesaier, Ge Junbo, Zhang Yingmei, Ren Jun

Macrophage NEDD4L restrains liver fibrosis by preventing scar-associated macrophage expansion via ubiquitination of phospho-SMAD3.

巨噬细胞 NEDD4L 通过泛素化磷酸化 SMAD3 来抑制瘢痕相关巨噬细胞的扩张,从而抑制肝纤维化。

He Yanghuan, Ge Shujun, Ling Shijia, Yang Siting, Yang Feiran, Liu Xinyi, Dong Siyue, Chen Yingfen, Zhang Ziling, Zhou Yue, Hwang Seonghwan, Kim Seung-Jin, Wang Peng, He Yong, Chen Yuanwen

Palmitic acid aggravates atopic dermatitis by regulating SGK1/NEDD4L-involved cutaneous neuroimmune inflammation through driving TRPV1 and MRGPRB2 S-palmitoylation.

棕榈酸通过驱动 TRPV1 和 MRGPRB2 S-棕榈酰化来调节 SGK1/NEDD4L 参与的皮肤神经免疫炎症,从而加剧特应性皮炎。

Chen Bangtao, Yang Jing, Song Tingting, Wu Weiyu, Hao Fei, Yang Zhi

Increased Urinary Extracellular Vesicles and Reduced Expression of NEDD4L in Patients With Type 2 Diabetes and Diabetic Nephropathy

2型糖尿病合并糖尿病肾病患者尿液细胞外囊泡增多,NEDD4L表达降低

Thyagarajan, Nikhil; Le Leu, Richard; Kumar, Sharad; Jesudason, Shilpanjali; Manning, Jantina A

Activating NEDD4L suppresses EGFR-driven lung adenocarcinoma growth via facilitating EGFR proteasomal degradation

激活NEDD4L可通过促进EGFR蛋白酶体降解来抑制EGFR驱动的肺腺癌生长。

Maojian Chen #,Wei Jiang #,Jianhua Zhan #,Shaoping Zhang #,Jiani Zheng #,Yihua Huang,Junyi He,Yunpeng Yang,Shen Zhao,Yaxiong Zhang,Jiaqing Liu,Lanlan Pang,Li Zhang,Wenfeng Fang,Jing Li

SLC7A11 upregulation via AR and NEDD4L ubiquitination contributes to ferroptosis inhibition and enzalutamide resistance in castration-resistant prostate cancer

SLC7A11通过AR和NEDD4L泛素化上调,导致去势抵抗性前列腺癌中铁死亡抑制和恩扎卢胺耐药。

Tang, Min; Xue, Luotong; Wang, Bao; Jiang, Zhixin; Li, Pu; Bu, Hengtao; Zhao, Chesong; Zhang, Yurong; Liu, Shuaimei; Chen, Xiyuan; Liu, Bianjiang; Meng, Xiaoxin; Li, Jie

Correction: SLC7A11 upregulation via AR and NEDD4L ubiquitination contributes to ferroptosis inhibition and enzalutamide resistance in castration-resistant prostate cancer

更正:SLC7A11 通过 AR 和 NEDD4L 泛素化上调,导致去势抵抗性前列腺癌中铁死亡抑制和恩扎卢胺耐药。

Tang, Min; Xue, Luotong; Wang, Bao; Jiang, Zhixin; Li, Pu; Bu, Hengtao; Zhao, Chesong; Zhang, Yurong; Liu, Shuaimei; Chen, Xiyuan; Liu, Bianjiang; Meng, Xiaoxin; Li, Jie

NEDD4L induces mitochondrial dysfunction and neurodegeneration by promoting LIPT2 degradation in Huntington's disease

NEDD4L通过促进LIPT2降解诱导亨廷顿病中的线粒体功能障碍和神经退行性变

Fan, Pan; Liu, Yaqing; Liu, Chunyue; Yao, Hao; Xu, Shibo; Jiang, Yueqing; Wu, Yang; Liu, Yan; Guo, Xing