日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

NUP98 regulates orthoflavivirus replication through interaction with vRNA and can be targeted for antiviral purposes.

NUP98 通过与病毒 RNA 相互作用来调节正黄病毒的复制,因此可以作为抗病毒治疗的靶点。

Peters Marie B A, Lindqvist Richard, Madhu Priyanka, Lundmark Richard, Ivarsson Ylva, Överby Anna K

Strategies for identifying NUP98 rearrangements in adult myeloid neoplasms

成人髓系肿瘤中NUP98重排的鉴定策略

Yuen, Lisa D; Hasserjian, Robert P; Fathi, Amir T; Luskin, Marlise R; Winer, Eric S; Dal Cin, Paola; Kim, Annette S; Lindsley, R Coleman; Tsai, Harrison K; Nardi, Valentina

Rare NUP98::SETBP1 fusion transcript in a refractory adult T-cell lymphoblastic lymphoma

难治性成人T细胞淋巴母细胞淋巴瘤中罕见的NUP98::SETBP1融合转录本

Chen, Xiaoyong; Wang, Linlin; Zou, Junyan; Zhou, Jihao; Zhou, Yaying; Zuo, Min; Hu, Lina; Dong, Xiaoying; Ke, Peng; Hou, Junjie

Preferential formation of NUP98-KDM5A condensates at specific H3K4me3-rich loci drives leukemogenic gene expression

NUP98-KDM5A 凝聚体在特定富含 H3K4me3 的位点优先形成,驱动白血病基因表达。

Berrocal, Augusto; Sandoval, Jonathan E; Khetan, Neha; Ma, Alexa; Wang, Tianhong; Moore, Camille; Narlikar, Geeta J; Li, Hao; Fujimori, Danica Galonić; Huang, Bo

KAT6A and KAT7 Histone Acetyltransferase Complexes Are Molecular Dependencies and Therapeutic Targets in NUP98-Rearranged Acute Myeloid Leukemia

KAT6A 和 KAT7 组蛋白乙酰转移酶复合物是 NUP98 重排急性髓系白血病的分子依赖性和治疗靶点

Michmerhuizen, Nicole L; Heikamp, Emily B; Iacobucci, Ilaria; Umeda, Masayuki; Arthur, Bright; Mishra, Vibhor; Park, Chun Shik; Di Giacomo, Danika; Hiltenbrand, Ryan; Gao, Qingsong; Radko-Juettner, Sandi; Lott, Josi; Martucci, Cynthia; Subramanyam, Varsha; Hatton, Charlie; Wenge, Daniela V; Baviskar, Pradyuamna; Portola, Pablo; Claquin, Aurelie; Chandra, Bappaditya; Baggett, David W; Khalighifar, Ali; Huang, Hongling; Zhou, Peipei; Long, Lingyun; Shi, Hao; Sun, Yu; Papachristou, Evangelia K; Sekhar Reddy Chilamakuri, Chandra; de Luna Vitorino, Francisca N; Gongora, Joanna M; Wu, Huiyun; Pounds, Stanley B; Janke, Laura J; Kentsis, Alex; D'Santos, Clive S; Garcia, Benjamin A; Kriwacki, Richard W; Chi, Hongbo; Klco, Jeffery M; Armstrong, Scott A; Mullighan, Charles G

Fusion oncoproteins and cooperating mutations define disease phenotypes in NUP98-rearranged leukemia

融合癌蛋白和协同突变决定了NUP98重排白血病的疾病表型。

Umeda, Masayuki; Hiltenbrand, Ryan; Michmerhuizen, Nicole L; Barajas, Juan M; Thomas Iii, Melvin E; Arthur, Bright; Walsh, Michael P; Song, Guangchun; Ma, Jing; Westover, Tamara; Kumar, Amit; Pölönen, Petri; Mecucci, Cristina; Di Giacomo, Danika; Locatelli, Franco; Masetti, Riccardo; Bertuccio, Salvatore N; Pigazzi, Martina; Pruett-Miller, Shondra M; Pounds, Stanley; Rubnitz, Jeffrey; Inaba, Hiroto; Papadopoulos, Kyriakos P; Wick, Michael J; Iacobucci, Ilaria; Mullighan, Charles G; Klco, Jeffery M

Precision medicine for high-risk gene fusions in pediatric AML: a focus on KMT2A, NUP98, and GLIS2 rearrangements

针对儿童急性髓系白血病高危基因融合的精准医疗:聚焦KMT2A、NUP98和GLIS2重排

Egan, Grace; Tasian, Sarah K

Differential conformational expansion of NUP98-HOXA9 oncoprotein from nanosized assemblies to macrophases.

NUP98-HOXA9癌蛋白从纳米级聚集体到宏观相的差异性构象扩展。

Ruan Hao, Dillenburg Rodrigo F, Hosseini Elnaz, Wittmann Sina, Girard Martin, Lemke Edward A

The phenylalanine-and-glycine repeats of NUP98 oncofusions form condensates that selectively partition transcriptional coactivators

NUP98 癌基因融合蛋白中的苯丙氨酸和甘氨酸重复序列形成凝聚体,选择性地分隔转录共激活因子。

Jeong Hyun Ahn,Yiran Guo,Heankel Lyons,Samuel G Mackintosh,Benjamin K Lau,Ricky D Edmondson,Stephanie D Byrum,Aaron J Storey,Alan J Tackett,Ling Cai,Benjamin R Sabari,Gang Greg Wang

HoxBlinc: a key driver of chromatin dynamics in NUP98 fusion-driven leukemia

HoxBlinc:NUP98融合驱动白血病中染色质动力学的关键驱动因素

Xu, Jian; Du, Wei