日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Complexes Formed by the K63-Specific Deubiquitinating Enzyme BRCC36: New Promising Therapeutic Targets in Human Disease

由K63特异性去泛素化酶BRCC36形成的复合物:人类疾病中新的潜在治疗靶点

Zhang, Xinyu; Pang, Xiaodong; Chen, Yili; Liu, Yue; Huang, Jian-An; Zeng, Yuanyuan

Lys-63-specific deubiquitinase BRCC36 enhances the sensitivity of multiple myeloma cells to lenalidomide by inhibiting lysosomal degradation of cereblon

Lys-63特异性去泛素化酶BRCC36通过抑制cereblon的溶酶体降解,增强多发性骨髓瘤细胞对来那度胺的敏感性。

Busong Wang # ,Min Li # ,Dan Cao ,Qing Sun ,Wenjun Yu ,Jingjing Ma ,Haigang Ren ,Guoqiang Xu ,Liang Zhou

BRCC36 associates with FLT3-ITD to regulate its protein stability and intracellular signaling in acute myeloid leukemia

BRCC36 与 FLT3-ITD 结合,调节其在急性髓系白血病中的蛋白稳定性和细胞内信号传导。

Liu, Jianwei; Isaji, Tomoya; Komatsu, Sachiko; Sun, Yuhan; Xu, Xing; Fukuda, Tomohiko; Fujimura, Tsutomu; Takahashi, Shinichiro; Gu, Jianguo

miR-369-3p Modulates Intestinal Inflammatory Response via BRCC3/NLRP3 Inflammasome Axis

miR-369-3p 通过 BRCC3/NLRP3 炎症小体轴调节肠道炎症反应

Viviana Scalavino, Emanuele Piccinno, Anna Maria Valentini, Nicolò Schena, Raffaele Armentano, Gianluigi Giannelli, Grazia Serino

Abraxas suppresses DNA end resection and limits break-induced replication by controlling SLX4/MUS81 chromatin loading in response to TOP1 inhibitor-induced DNA damage

Abraxas通过控制SLX4/MUS81染色质加载,抑制DNA末端切除并限制断裂诱导的复制,以应对TOP1抑制剂诱导的DNA损伤。

Xiao Wu ,Bin Wang

Structural Basis of BRCC36 Function in DNA Repair and Immune Regulation

BRCC36在DNA修复和免疫调节中的结构基础

Julius Rabl ,Richard D Bunker ,Andreas D Schenk ,Simone Cavadini ,Mark E Gill ,Wassim Abdulrahman ,Amparo Andrés-Pons ,Martijn S Luijsterburg ,Adel F M Ibrahim ,Emma Branigan ,Jacob D Aguirre ,Aimee H Marceau ,Claire Guérillon ,Tewis Bouwmeester ,Ulrich Hassiepen ,Antoine H F M Peters ,Martin Renatus ,Laurent Gelman ,Seth M Rubin ,Niels Mailand ,Haico van Attikum ,Ronald T Hay ,Nicolas H Thomä

Cell cycle-regulated ubiquitination of tankyrase 1 by RNF8 and ABRO1/BRCC36 controls the timing of sister telomere resolution

RNF8 和 ABRO1/BRCC36 对 tankyrase 1 进行细胞周期调控的泛素化,控制姐妹端粒分解的时间

Ekta Tripathi, Susan Smith

The Lys63-deubiquitylating Enzyme BRCC36 Limits DNA Break Processing and Repair

Lys63去泛素化酶BRCC36限制DNA断裂的处理和修复

Ng, Hoi-Man; Wei, Leizhen; Lan, Li; Huen, Michael S Y

Higher-Order Assembly of BRCC36-KIAA0157 Is Required for DUB Activity and Biological Function

BRCC36-KIAA0157 的高级组装是 DUB 活性和生物学功能所必需的

Zeqiraj, Elton; Tian, Lei; Piggott, Christopher A; Pillon, Monica C; Duffy, Nicole M; Ceccarelli, Derek F; Keszei, Alexander F A; Lorenzen, Kristina; Kurinov, Igor; Orlicky, Stephen; Gish, Gerald D; Heck, Albert J R; Guarné, Alba; Greenberg, Roger A; Sicheri, Frank

NBA1/MERIT40 and BRE interaction is required for the integrity of two distinct deubiquitinating enzyme BRCC36-containing complexes

NBA1/MERIT40 和 BRE 的相互作用对于两种不同的去泛素化酶 BRCC36 复合物的完整性是必需的。

Hu, Xin; Kim, Jin Ah; Castillo, Andy; Huang, Michael; Liu, Jianxin; Wang, Bin