日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

TLR2-PI3K/Akt mediated microbe-mimetic priming boosts the therapeutic paracrine function of GelMA-Encapsulated MSCs for diabetic wound regeneration.

TLR2-PI3K/Akt 介导的微生物模拟启动增强了 GelMA 封装的 MSC 的治疗性旁分泌功能,用于糖尿病伤口再生。

Tian Feng, Kong Yue, Liu Qinghua, Zhu Shuoji, Guo Xu, Su YanLin, Yu Bingyang, Zhang Chao, Zhu Dongzhen, Li Zhao, Song Wei, Kong Yi, Yin Xiangye, Huang Yuyan, Tan Yaxin, Zhang Mengde, Du Jinpeng, Liang Liting, Li Jianjun, Zhu Ping, Fu Xiaobing, Huang Sha

Anti-TLR2 immunotherapy modulates neuron-to-oligodendrocyte propagation of α-synuclein in mouse and human models.

抗 TLR2 免疫疗法可调节小鼠和人类模型中 α-突触核蛋白从神经元到少突胶质细胞的传播。

Bae Eun-Jin, Ham Sangwoo, Jeong Yeonwoo William, Yang Woo Seung, Shin Jimin, Lee Won-Jae, Ahn Woo Jung, Yoon Ye-Seul, Lee He-Jin, Lee Sang Hwan, Park Sung-Hye, Lee Seung-Jae

Synovial Fluid-derived Micrococcus Luteus G18 Exacerbates Osteoarthritis Progression by Promoting Chondrocyte Degradation via TLR2/JNK/AP-1 Signaling Pathway

滑液来源的黄微球菌G18通过TLR2/JNK/AP-1信号通路促进软骨细胞降解,从而加剧骨关节炎的进展。

Chen, Tingtao; Zeng, Qingwei; Xu, Tangchang; Qi, Xinyue; Li, Kaiyi; Wei, Jing; Hao, Liang

Wedelolactone, a Novel TLR2 Agonist, Promotes Neutrophil Differentiation and Ameliorates Neutropenia: A Multi-Omics Approach to Unravel the Mechanism

新型TLR2激动剂Wedelolactone促进中性粒细胞分化并改善中性粒细胞减少症:多组学方法揭示其机制

Wang, Long; Li, Zhichao; Hu, Tianci; Li, Qinyao; Zhang, Linwei; Mei, Xinyue; Qi, Xiao; Liu, Sheng; Kong, Weijie; Luo, Jiesi; Wu, Anguo; Huang, Feihong; Li, Sirui; Dai, Shuang; Zhang, Chunxiang; Li, Rong; Wu, Jianming

Apolipoprotein C3 exacerbates vascular calcification by promoting ferroptosis via the TLR2/AMPK pathway in vascular smooth muscle cells.

载脂蛋白 C3 通过 TLR2/AMPK 通路促进血管平滑肌细胞发生铁死亡,从而加剧血管钙化。

Zhou Zihao, Chen Jinxuan, Wang Huan, Han Yufei, Liu Changyun, Shen Qiang, Dong Zhao, Lu Kaikai, Zhai Chao, Xu Yitong, Zhang Lianxin, He Lei, Fang Yuan, Zhang Ling, Wang Yuhui, Xian Xunde, Yao Lin, Dong Erdan, Huang Wei

Dexmedetomidine protects against myocardial ischemia reperfusion injury by inhibiting the TLR2-NF-κB pathway in diabetic mice

右美托咪定通过抑制糖尿病小鼠体内的TLR2-NF-κB通路,发挥保护作用,防止心肌缺血再灌注损伤。

Jackson, David G; Chen, Zhengjie; Zheng, Liangyu; Chen, Gang

Computational-experimental study reveals direct target and bioactives of Ajania fruticulosa against NAFLD via TLR2/NF-κB/PPAR-γ signaling.

计算实验研究揭示了 Ajania fruticulosa 通过 TLR2/NF-κB/PPAR-κ 信号通路对 NAFLD 的直接靶点和生物活性物质。

Cathelicidin-Ka, the first frog-derived TLR2 and TLR4 agonist, induces macrophage activation and promotes inflammation.

Cathelicidin-Ka 是第一个源自青蛙的 TLR2 和 TLR4 激动剂,可诱导巨噬细胞活化并促进炎症。

TLR2 activation potentiates P-glycoprotein-mediated methotrexate efflux and enhances cytotoxicity of human NK cells against acute lymphoblastic leukemia

TLR2激活可增强P-糖蛋白介导的甲氨蝶呤外排,并增强人NK细胞对急性淋巴细胞白血病的细胞毒性。

Álvarez-Carrasco, Pablo; Basurto-Olvera, Paola; Jiménez-Hernández, Elva; Medina-Sanson, Aurora; Maldonado-Bernal, Carmen

An underlying mechanism of bovine mastitis: PGE(2) regulates Staphylococcus aureus-induced inflammatory response through TLR2, TLR4, and NLRP3 in macrophages.

牛乳腺炎的潜在机制:PGE(2)通过巨噬细胞中的TLR2、TLR4和NLRP3调节金黄色葡萄球菌诱导的炎症反应。

Gong Zhiguo, Yu Zhuoya, Ren Peipei, Zhang Shuangyi, Gao Ruifeng, Zhao Jiamin, Wang Yixin, Qin Shaojie, Bao Wenhui, Shuang Feng