日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

CCL24 recruits CCR3(+) TAMs to promote immunosuppression via YAP1 activation and serves as a therapeutic target for Gracillin in colorectal cancer

CCL24通过激活YAP1募集CCR3(+)肿瘤相关巨噬细胞(TAM)促进免疫抑制,并可作为结直肠癌中格拉西林治疗的靶点。

Huang, Shiyu; Lin, Wanqiong; Ding, Xu; Shi, Jinjun; Liu, Fei; Wang, Guoqing; Gao, Chanchan; Su, Xiangyu

EBF3 suppresses lung adenocarcinoma progression and immune evasion via transcriptional repression of CCL24

EBF3通过转录抑制CCL24来抑制肺腺癌的进展和免疫逃逸。

Li, Lan; Yang, YiRan; Li, Dan; Chen, ChuMao; Mu, XinMei; Wu, JiaXin; Yuan, Jin

Peritoneal MSCs-derived exosomes suppress CCL24 synthesis through miR-320d delivery contributing to the improvement of peritoneal dialysis-associated fibrosis.

腹膜间充质干细胞衍生的外泌体通过 miR-320d 递送抑制 CCL24 合成,从而有助于改善腹膜透析相关纤维化。

Zhao Xinhui, Xi Minhui, Qi Hualin, Xu Jia, Yuan Rui, Shi Danye, Lu Jingyuan

Macrophage-Derived CCL24 Promotes Cardiac Fibrosis Via Fibroblast CCR3

巨噬细胞衍生的CCL24通过成纤维细胞CCR3促进心脏纤维化

Parthiban, Preethy; Barrow, Fanta; Wang, Haiguang; Chalise, Upendra; Araujo, Natalia; Souza-Neto, Fernando; Nguyen, Huy; Draxler, Micah; Pallais, Jean Pierre; Yucel, Dogacan; Liu, Hong; Ciske, Erin; Fehrenbach, Patrick; Hakeem, Andrew; Lee, Sang Hun; Herman, Adam; Rothenberg, Marc E; Dudley, Samuel; van Berlo, Jop H; Revelo, Xavier S

When Resident Macrophages Call, Fibroblasts Answer: CCL24-CCR3 in Fibrosis

当驻留巨噬细胞发出信号时,成纤维细胞响应:CCL24-CCR3 在纤维化中的作用

Bayer, Abraham L; Alcaide, Pilar

CCL24 and Fibrosis: A Narrative Review of Existing Evidence and Mechanisms

CCL24与纤维化:现有证据和机制的叙述性综述

Greenman, Raanan; Weston, Chris J

The Role of CCL24 in Primary Sclerosing Cholangitis: Bridging Patient Serum Proteomics to Preclinical Data

CCL24在原发性硬化性胆管炎中的作用:连接患者血清蛋白质组学与临床前数据

Raanan Greenman ,Tom Snir ,Avi Katav ,Revital Aricha ,Inbal Mishalian ,Ophir Hay ,Matthew Frankel ,John Lawler ,Francesca Saffioti ,Massimo Pinzani ,Douglas Thorburn ,Amnon Peled ,Adi Mor ,Ilan Vaknin

Machine Learning Identifies Key Proteins in Primary Sclerosing Cholangitis Progression and Links High CCL24 to Cirrhosis

机器学习识别原发性硬化性胆管炎进展中的关键蛋白,并将高水平CCL24与肝硬化联系起来

Snir, Tom; Greenman, Raanan; Aricha, Revital; Frankel, Matthew; Lawler, John; Saffioti, Francesca; Pinzani, Massimo; Thorburn, Douglas; Mor, Adi; Vaknin, Ilan

CCL24 regulates biliary inflammation and fibrosis in primary sclerosing cholangitis

CCL24 调节原发性硬化性胆管炎中的胆道炎症和纤维化

Raanan Greenman ,Michal Segal-Salto ,Neta Barashi ,Ophir Hay ,Avi Katav ,Omer Levi ,Ilan Vaknin ,Revital Aricha ,Sarit Aharoni ,Tom Snir ,Inbal Mishalian ,Devorah Olam ,Johnny Amer ,Ahmad Salhab ,Rifaat Safadi ,Yaakov Maor ,Palak Trivedi ,Christopher J Weston ,Francesca Saffioti ,Andrew Hall ,Massimo Pinzani ,Douglas Thorburn ,Amnon Peled ,Adi Mor

CCL24/CCR3 axis plays a central role in angiotensin II-induced heart failure by stimulating M2 macrophage polarization and fibroblast activation

CCL24/CCR3轴通过刺激M2巨噬细胞极化和成纤维细胞活化,在血管紧张素II诱导的心力衰竭中发挥核心作用。

Wang, Zhen; Xu, Hongfei; Chen, Miao; Lu, Yunlong; Zheng, Liangrong; Ma, Liang