TEAD3 + high-risk melanoma cells crosstalk with GAS6 + macrophages via the GAS6-TYRO3 ligand-receptor axis to modulate propionate metabolism and drive melanoma progression

TEAD3+高危黑色素瘤细胞通过GAS6-TYRO3配体-受体轴与GAS6+巨噬细胞相互作用，从而调节丙酸代谢并驱动黑色素瘤进展。