GABARAP相关文献与解析，生知库 | 链接生命科学的每一步

TRIM22 facilitates autophagosome-lysosome fusion by mediating the association of GABARAPs and PLEKHM1

TRIM22通过介导GABARAPs和PLEKHM1的结合来促进自噬体-溶酶体融合。

doi:10.1080/15548627.2023.2287925

Hansol Heo ,Hyungsun Park ,Myung Shin Lee ,Jongyoon Kim ,Juyeong Kim ,Soon-Young Jung ,Sun Kyeon Kim ,Seongju Lee ,Jaerak Chang

自噬

发表日期：2024

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TNIP1 inhibits selective autophagy via bipartite interaction with LC3/GABARAP and TAX1BP1

TNIP1通过与LC3/GABARAP和TAX1BP1的双向相互作用抑制选择性自噬

期刊:Molecular Cell

影响因子:14.5

doi:10.1016/j.molcel.2023.02.023

François Le Guerroué ，Eric N Bunker ，William M Rosencrans ，Jack T Nguyen ，Mohammed A Basar ，Achim Werner ，Tsui-Fen Chou ，Chunxin Wang ，Richard J Youle

Microautophagy regulated by STK38 and GABARAPs is essential to repair lysosomes and prevent aging

由STK38和GABARAPs调控的微自噬对于修复溶酶体和延缓衰老至关重要。

期刊:EMBO Reports

影响因子:6.5

doi:10.15252/embr.202357300

Monami Ogura,Tatsuya Kaminishi,Takayuki Shima,Miku Torigata,Nao Bekku,Keisuke Tabata,Satoshi Minami,Kohei Nishino,Akiko Nezu,Maho Hamasaki,Hidetaka Kosako,Tamotsu Yoshimori,Shuhei Nakamura

Effect of ATG12-ATG5-ATG16L1 autophagy E3-like complex on the ability of LC3/GABARAP proteins to induce vesicle tethering and fusion

ATG12-ATG5-ATG16L1自噬E3样复合物对LC3/GABARAP蛋白诱导囊泡系留和融合能力的影响

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-023-04704-z

Marina N Iriondo ,Asier Etxaniz ,Yaiza R Varela ,Uxue Ballesteros ,Melisa Lázaro ,Mikel Valle ,Dorotea Fracchiolla ,Sascha Martens ,L Ruth Montes ,Félix M Goñi ,Alicia Alonso

The mitophagy receptor NIX induces vIRF-1 oligomerization and interaction with GABARAPL1 for the promotion of HHV-8 reactivation-induced mitophagy

线粒体自噬受体NIX诱导vIRF-1寡聚化并与GABARAPL1相互作用，从而促进HHV-8再激活诱导的线粒体自噬。

期刊:PLoS Pathogens

影响因子:5.5

doi:10.1371/journal.ppat.1011548

Mai Tram Vo ,Chang-Yong Choi ,Young Bong Choi

The ATG8 Family Proteins GABARAP and GABARAPL1 Target Antigen to Dendritic Cells to Prime CD4+ and CD8+ T Cells

ATG8家族蛋白GABARAP和GABARAPL1靶向抗原至树突状细胞，从而启动CD4+和CD8+ T细胞

期刊:Cells

影响因子:5.1

doi:10.3390/cells11182782

Leïla Fonderflick ，Timothée Baudu ，Olivier Adotévi ，Michaël Guittaut ，Pascale Adami ，Régis Delage-Mourroux

GABARAP ameliorates IL-1β-induced inflammatory responses and osteogenic differentiation in bone marrow-derived stromal cells by activating autophagy

GABARAP通过激活自噬来改善IL-1β诱导的骨髓间充质干细胞炎症反应和成骨分化。

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-021-90586-9.

Xiaobo Guo ,Zhenyuan Wu

The GABARAP Co-Secretome Identified by APEX2-GABARAP Proximity Labelling of Extracellular Vesicles

通过APEX2-GABARAP邻近标记细胞外囊泡鉴定GABARAP共分泌组

期刊:Cells

影响因子:5.1

doi:10.3390/cells9061468

Julia L Sanwald ,Gereon Poschmann ,Kai Stühler ,Christian Behrends ,Silke Hoffmann ,Dieter Willbold

Ceramide and the membrane-fusion activity of LC3/GABARAP autophagy proteins

神经酰胺与LC3/GABARAP自噬蛋白的膜融合活性

Conserved LIR-specific interaction of Sigma-1 receptor and GABARAP

Sigma-1受体与GABARAP的保守LIR特异性相互作用

TRIM22 facilitates autophagosome-lysosome fusion by mediating the association of GABARAPs and PLEKHM1

TRIM22通过介导GABARAPs和PLEKHM1的结合来促进自噬体-溶酶体融合。

TNIP1 inhibits selective autophagy via bipartite interaction with LC3/GABARAP and TAX1BP1

TNIP1通过与LC3/GABARAP和TAX1BP1的双向相互作用抑制选择性自噬

Microautophagy regulated by STK38 and GABARAPs is essential to repair lysosomes and prevent aging

由STK38和GABARAPs调控的微自噬对于修复溶酶体和延缓衰老至关重要。

Effect of ATG12-ATG5-ATG16L1 autophagy E3-like complex on the ability of LC3/GABARAP proteins to induce vesicle tethering and fusion

ATG12-ATG5-ATG16L1自噬E3样复合物对LC3/GABARAP蛋白诱导囊泡系留和融合能力的影响

The mitophagy receptor NIX induces vIRF-1 oligomerization and interaction with GABARAPL1 for the promotion of HHV-8 reactivation-induced mitophagy

线粒体自噬受体NIX诱导vIRF-1寡聚化并与GABARAPL1相互作用，从而促进HHV-8再激活诱导的线粒体自噬。

The ATG8 Family Proteins GABARAP and GABARAPL1 Target Antigen to Dendritic Cells to Prime CD4+ and CD8+ T Cells

ATG8家族蛋白GABARAP和GABARAPL1靶向抗原至树突状细胞，从而启动CD4+和CD8+ T细胞

GABARAP ameliorates IL-1β-induced inflammatory responses and osteogenic differentiation in bone marrow-derived stromal cells by activating autophagy

GABARAP通过激活自噬来改善IL-1β诱导的骨髓间充质干细胞炎症反应和成骨分化。

The GABARAP Co-Secretome Identified by APEX2-GABARAP Proximity Labelling of Extracellular Vesicles

通过APEX2-GABARAP邻近标记细胞外囊泡鉴定GABARAP共分泌组