日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Single-Chain Anti-IL-1β Antibody Carried by Outer Membrane Vesicles of Bacteroides fragilis Alleviates Tubular Inflammation in Chronic Kidney Disease.

由脆弱拟杆菌外膜囊泡携带的单链抗IL-1β抗体可减轻慢性肾脏病中的肾小管炎症。

Li Liang, Li Xue Qi, Zhang Jia Xiu, Wu Qin Yi, Chen Pei Pei, Ruan Xiong Zhong, Ma Kun Ling

The IL-1β/NETs/AIM2 axis participates in the formation of trauma-induced heterotopic ossification by orchestrating crosstalk between neutrophils and macrophages

IL-1β/NETs/AIM2轴通过协调中性粒细胞和巨噬细胞之间的相互作用参与创伤诱导的异位骨化的形成。

Yuan, Zhengqiang; Yao, Siyuan; Yao, Xiangyun; Zhou, Chao; Li, Juehong; Fan, Cunyi

PET-microplastics trigger endothelial glycocalyx loss via ER stress and ROS unleashing IL-1β-driven SMC switching and early aortic structural impairment

PET微塑料通过内质网应激和活性氧(ROS)诱导内皮糖萼丢失,进而引发IL-1β驱动的平滑肌细胞(SMC)细胞类型转换和早期主动脉结构损伤。

Huo, Weixue; Qu, Jin; Wang, Sen; He, Mengwei; Zeng, Zhaoxiang; Kong, Deping; Yuan, Lushun; Feng, Rui

d-amino acids restrain macrophage IL-1β release through gasdermin D acetylation

D-氨基酸通过gasdermin D乙酰化作用抑制巨噬细胞IL-1β的释放

Wu, Zebiao; Hu, Qilin; Shen, Yinhao; Fu, Jian; Liu, Bingnan; Zhang, Meimei; Hung, Ifen; Liu, Chunxue; Ren, Wenkai

Cry2 deficiency leads to cognitive impairment through the microbiota-gut-brain axis mediated S1P/NLRP3/IL-1β pathway in mice.

Cry2 缺陷通过微生物群-肠-脑轴介导的 S1P/NLRP3/IL-1β 通路导致小鼠认知障碍。

Mitochondrial DNA drives NLRP3-IL-1β axis activation in microglia by binding to NLRP3, leading to neurodegeneration in Parkinson's disease models

线粒体DNA通过与NLRP3结合,驱动小胶质细胞中NLRP3-IL-1β轴的激活,从而导致帕金森病模型中的神经退行性变。

Gan, Qinglin; Fu, Xiaolong; Zhou, Ting; Fan, Naiyu; Nan, Nan; Wang, Yi; Yang, Yonggang; Gou, Shiyi; Hu, Lizhen; Zhou, Shaoyu

An endothelial RNA splicing atlas catalogs effects of IL-1β and identifies an alternative PROCR isoform with genetic links to pleiotropic vascular disease.

内皮细胞 RNA 剪接图谱记录了 IL-1β 的作用,并确定了一种与多效性血管疾病有遗传联系的替代 PROCR 同工型。

IL-1β modulates inflammatory response of human bone marrow-derived MSCs and neutrophil recruitment in vitro via NF-kB-associated signaling

IL-1β通过NF-κB相关信号通路调节人骨髓间充质干细胞的炎症反应和中性粒细胞的体外募集

Hassaan, Nour; Schmidt, Tobias; Söderlund, Zackarias; Kalafatis, Dimitrios; Påhlman, Lisa I; Scheding, Stefan; Weiss, Daniel J; Kahn, Robin; Rolandsson Enes, Sara

Inhalation of mesenchymal stromal cell-derived extracellular vesicles activates macrophage polarization through the miR-22-3p/NLRP3/IL-1β pathway, ameliorating lung ischemia - reperfusion injury.

吸入间充质基质细胞衍生的细胞外囊泡可通过 miR-22-3p/NLRP3/IL-1β 通路激活巨噬细胞极化,从而改善肺缺血再灌注损伤。

Wang Tao, Wu Guodong, Gao Peigen, Zhuang Fenghui, Wang Zeyu, Zhou Ziheng, Li Chongwu, Wu Junqi, Zhao Deping

Targeting macrophage glycogen metabolism attenuates ulcerative colitis by suppressing IL-1β production through UDPG-P2Y(14) signaling.

靶向巨噬细胞糖原代谢可通过抑制 UDPG-P2Y(14) 信号传导产生 IL-1β 来减轻溃疡性结肠炎。