日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

DUSP6 ablation restores CAR T-cell fitness impaired by tumor CD58 loss through invigoration of AP-1 signaling

DUSP6 消融可通过增强 AP-1 信号传导来恢复因肿瘤 CD58 丢失而受损的 CAR T 细胞活性。

Ma, Xinran; Zhang, Yang; Wang, Yao; Han, Fuxin; Lu, Yuting; Tong, Chuan; Guo, Yelei; Wei, Jianshu; Zhu, Qi; Dong, Liang; Cao, Zhi; Meng, Zhenzhen; Shi, Jinhong; Wu, Zhiqiang; Han, Weidong

CAP-CD56(+)CD271(+) BMSCs exos-loaded PVA/SA sustained-release hydrogel attenuates chondrocyte senescence and ameliorates lumbar facet joint osteoarthritis

载有CAP-CD56(+)CD271(+) BMSCs外泌体的PVA/SA缓释水凝胶可减轻软骨细胞衰老并改善腰椎小关节骨关节炎

Xie, Yong; Liu, Quanbo; Tan, Jinghua; Lu, Yingjie; Li, Yunlong; Ye, Hongyi; Xu, Zhun; Wang, Ming; Wang, Cheng; Li, Xuelin; Xue, Jingbo; Yan, Yiguo

ROS Activated NETosis of Bone Marrow CD55(+) Intermediate Mature Neutrophils Through HIF1α-PADI4 Pathway to Initiate Bone Aging

ROS通过HIF1α-PADI4通路激活骨髓CD55(+)中间成熟中性粒细胞的NETosis,从而启动骨骼老化

Guo, Yutong; Cui, Shengjie; Wen, Xi; Wang, Yidi; Wu, Ben; Wang, Yixiang; Gu, Yan

PM(10) Impairs CD56(dim) NK Cell Cytotoxicity via FNBP1 Suppression to Exacerbate Rheumatoid Arthritis: Insights from Multimodal Multi-Omics

PM(10)通过抑制FNBP1损害CD56(dim) NK细胞的细胞毒性,从而加剧类风湿性关节炎:来自多模式多组学的见解

Zhao, Runhan; Zhang, Qinyang; Jiang, Yu; Tang, Dagang; Qu, Xiao; Zhang, Jun; Chen, Yi; Duan, Weixia; Li, Tao; Cai, Zhengwei; Huang, Yanran; Luo, Xiaoji

Single-Cell Dissection Reveals Immune Dysregulation After CD5 or CD7-Directed Chimeric Antigen Receptor T-Cell Therapy.

单细胞解剖揭示 CD5 或 CD7 靶向嵌合抗原受体 T 细胞疗法后的免疫失调。

A CD57+CD8+ T cell subset links T cell cytotoxicity to fibrotic lung disease in systemic sclerosis

CD57+CD8+ T 细胞亚群将 T 细胞细胞毒性与系统性硬化症中的纤维化肺病联系起来

Sasaki, Takanori; Cao, Ye; Sowerby, John M; Higashioka, Kazuhiko; Marks, Kathryne E; Elahee, Mehreen; Kamiya, Mari; Dellaripa, Paul F; Ainsworth, Richard I; Taylor, Kimberly E; Bottini, Nunzio; Wolters, Paul; Kim, Edy Y; Boin, Francesco; Rao, Deepak A

Cell-free secretome of CD56(bright)CD16(bright) directly reprogrammed NK cells enhances wound healing via CCL3/4/5-CCR5 signaling.

CD56(bright)CD16(bright)直接重编程NK细胞的无细胞分泌组通过CCL3/4/5-CCR5信号通路增强伤口愈合。

Correction: NK-92 cells labeled with Fe(3)O(4)-PEG-CD56/Avastin@Ce6 nanoprobes for the targeted treatment and noninvasive therapeutic evaluation of breast cancer

更正:用 Fe(3)O(4)-PEG-CD56/Avastin@Ce6 纳米探针标记的 NK-92 细胞用于乳腺癌的靶向治疗和非侵入性治疗评估

Lian, Jingge; Li, Meng; Duan, Meng; Sun, Xiaoqian; Wang, Zilin; Guo, Xinyu; Li, Jingchao; Gao, Guo; Li, Kangan

Role of CD5 signalling for pro-inflammatory Th17 response in multiple sclerosis

CD5信号通路在多发性硬化症中促炎性Th17反应中的作用

Pape, Katrin; Hanuscheck, Nicholas; Schmaul, Samantha; Kneilmann, Flores; Bündgen, Georg; Wasser, Beatrice; Steffen, Falk; Engel, Sinah; Luessi, Felix; Klein, Matthias; Bohn, Toszka; Bopp, Tobias; Bittner, Stefan; Zipp, Frauke

Efficacy and safety of autologous CD5-KO anti-CD5 CAR-T cells in relapsed/refractory CD5+ hematological malignancies

自体CD5-KO抗CD5 CAR-T细胞治疗复发/难治性CD5+血液系统恶性肿瘤的疗效和安全性

Jiali Cheng,Li Zhu,Jia Wu,Yuhao Zeng,Xia Mao,Shengnan Ding,Jue Wang,Yi Xiao,Xiaoxi Zhou,Wei Mu,Xiaojian Zhu