Anti-VE Cadherin antibody - Intercellular Junction Marker (Ab33168)
CD144, Cadherin-5, 7B4 antigen, Vascular endothelial cadherin, VE-cadherin, CDH5
详细信息
特异性
From May 2024, QC testing of replenishment batches of this polyclonal changed. All tested and expected application and reactive species combinations are still covered by our Abcam product promise. However, we no longer test all applications. For more information on a specific batch, please contact our Scientific Support who will be happy to help.
背景信息
Anti-VE Cadherin antibody - Intercellular Junction Marker (ab33168) is a rabbit polyclonal antibody and is validated for use in ICC/IF, WB in mouse, rat samples. Anti-VE Cadherin antibody - Intercellular Junction Marker (ab33168) has been cited over 361 times in peer reviewed journals and is trusted by the scientific community. Abcam's high quality validation processes ensure Anti-VE Cadherin antibody - Intercellular Junction Marker (ab33168) has high sensitivity and specificity. Anti-VE Cadherin antibody - Intercellular Junction Marker (ab33168) has 39 independent reviews from customers. Anti-VE Cadherin antibody - Intercellular Junction Marker (ab33168) specifically detects VE Cadherin (UniProt ID: P33151; Molecular weight: 83kDa) and is sold in 100 µg selling sizes.
文献引用 (450)
Deployable 3D-Printed Vascular Stent with Surface-Catalysed Endogenous Nitric Oxide Generation.
可部署的 3D 打印血管支架，具有表面催化内源性一氧化氮生成功能。
Synergistic fusion of CD47, VE-cadherin and mussel adhesion protein promotes endothelialization and suppresses inflammation in vascular stents.
CD47、VE-钙黏蛋白和贻贝黏附蛋白的协同融合可促进血管支架内皮化并抑制炎症。
Vascular Endothelial Growth Factor-D Improves Lung Vascular Integrity During Acute Lung Injury.
血管内皮生长因子-D可改善急性肺损伤期间的肺血管完整性。
FGF 13 functions as a regulator of the ERK/aerobic glycolysis axis in the inflammatory state during septic lung injury.
FGF 13 在脓毒症肺损伤的炎症状态下，作为 ERK/有氧糖酵解轴的调节因子发挥作用。
Activator protein-1 (AP-1) inhibition prevents endothelial to mesenchymal transition in diabetes-associated atherosclerosis: a translational study.
激活蛋白-1 (AP-1) 抑制可预防糖尿病相关动脉粥样硬化中的内皮细胞向间质细胞转化：一项转化研究。
Lysolecithin reprogramming via LPCAT1 modulation restores endothelial function and prevents diabetes-associated dysmetabolism.
通过 LPCAT1 调节溶血卵磷脂重编程可恢复内皮功能并预防糖尿病相关的代谢紊乱。
Automating Vascular Biology: An End-to-End Automated Workflow for High-Throughput Blood Vessel-on-a-Chip Production and Multi-Site Validation.
血管生物学自动化：用于高通量芯片血管生产和多站点验证的端到端自动化工作流程。
Bioengineered Lymphatic Vessels in Synthetic Matrices to Study Breast Cancer Cell Functions.
利用合成基质中的生物工程淋巴管研究乳腺癌细胞功能。
Macrophage pyroptosis inhibition alleviates postinjury neointimal formation and vascular restenosis.
抑制巨噬细胞焦亡可减轻损伤后新生内膜形成和血管再狭窄。
RAB5C Increases Endothelial Release of VWF by Regulating Vesicle Trafficking.
RAB5C 通过调节囊泡运输增加内皮细胞释放 VWF。
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