文献引用 (232)
Allosteric activation of a cell-type-specific GPR120 inhibits amyloid pathology of Alzheimer's disease.
细胞类型特异性 GPR120 的变构激活可抑制阿尔茨海默病的淀粉样蛋白病理。
Senecavirus a VP2 protein orchestrates PRDX1 degradation through dual autophagy pathways: macroautophagy and chaperone-mediated autophagy.
塞内卡病毒 VP2 蛋白通过双重自噬途径协调 PRDX1 降解：巨自噬和分子伴侣介导的自噬。
α-Synuclein expression is required for somatodendritic dopamine release and immediate early gene induction.
α-突触核蛋白的表达是胞体树突多巴胺释放和即刻早期基因诱导所必需的。
Stroma-targeted gene delivery for efficient immunogene therapy against pancreatic cancer.
基质靶向基因递送用于有效治疗胰腺癌的免疫基因疗法。
The ISR downstream effector ATF4 promotes mGluR-dependent long-term depression and associated behavior.
ISR下游效应因子ATF4促进mGluR依赖性长期抑制及相关行为。
Oral bioavailable ITRI-148 degrades androgen receptor variants and overcomes antiandrogen resistance in advanced prostate cancer.
口服生物利用度高的 ITRI-148 可降解雄激素受体变体，克服晚期前列腺癌的抗雄激素耐药性。
Overexpression of the inwardly rectifying potassium channel Kir4.1 or Kir4.1 Tyr 9 Asp in Müller cells exerts neuroprotective effects in an experimental glaucoma model.
在实验性青光眼模型中，Müller 细胞中内向整流钾通道 Kir4.1 或 Kir4.1 Tyr 9 Asp 的过度表达可发挥神经保护作用
Activating mutations in ESR1 contribute to an immunosuppressive breast tumor microenvironment by dampening cytokine secretion.
ESR1 的激活突变会抑制细胞因子的分泌，从而导致免疫抑制性乳腺肿瘤微环境的形成。
Inhibition of Breast Cancer Bone Metastasis by LRP5-Overexpressing Osteocytes via the LIMA1/MYO5B Signaling Axis.
LRP5过表达骨细胞通过LIMA1/MYO5B信号轴抑制乳腺癌骨转移。
High Glucose-Induced Alterations in Regucalcin Expression in Podocytes and Their Potential Consequences.
高葡萄糖诱导足细胞中 Regucalcin 表达的改变及其潜在后果。
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