文献引用 (94)
Deep-learning-based de novo discovery and design of therapeutics that reverse disease-associated transcriptional phenotypes.
基于深度学习的从头发现和设计能够逆转疾病相关转录表型的治疗方法。
Mapping the Tissue-of-Origins of Mesenchymal Stromal Cells in Injury Repair.
损伤修复中间充质基质细胞的组织起源图谱。
Functionalized magnetic hydrogel encapsulation of human dental follicle stem cells under a static magnetic field enhances multi-site bone regeneration.
在静态磁场下，功能化磁性水凝胶包裹人类牙囊干细胞可增强多部位骨再生。
H3K9 acetylation-dependent CHAC1 transcription in dihydroartemisinin-induced hepatic stellate cell ferroptosis.
在二氢青蒿素诱导的肝星状细胞铁死亡中，H3K9 乙酰化依赖的 CHAC1 转录。
Epigenetic Suppression of RASAL1 by HDAC3 and Cofactor YY1 Promotes Fibroblast-Myofibroblast Transition and Renal Fibrosis.
HDAC3 和辅因子 YY1 对 RASAL1 的表观遗传抑制促进成纤维细胞-肌成纤维细胞转化和肾纤维化。
Delivering LINE1 antisense oligonucleotides via endothelial targeting extracellular vesicles to ameliorate myocardial infarction-induced cardiac senescence.
通过内皮细胞靶向细胞外囊泡递送 LINE1 反义寡核苷酸，以改善心肌梗死引起的心脏衰老
Janus hydrogel microrobots with bioactive ions for the regeneration of tendon-bone interface
带有生物活性离子的Janus水凝胶微型机器人用于肌腱-骨界面的再生
Enhanced SIRT3 expression restores mitochondrial quality control mechanism to reverse osteogenic impairment in type 2 diabetes mellitus
增强 SIRT3 表达可恢复线粒体质量控制机制，从而逆转 2 型糖尿病中的成骨障碍
Restoration of SIRT3 Expression in Aged Mice Alleviates UUO-Induced Renal Fibrosis by Reducing GSK-3β Hyperacetylation.
恢复老年小鼠的 SIRT3 表达可通过减少 GSK-3β 过度乙酰化来缓解 UUO 诱导的肾纤维化。
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