文献引用 (78)
Engineering NIR-II carbon dots through aniline extension with graphene and nitrogen enrichment for hepatobiliary theranostics.
利用苯胺延伸法，通过石墨烯和氮富集法制备近红外二区碳点，用于肝胆诊疗。
Picornavirus VP2 protein suppresses innate immunity through selective autophagic degradation of IKBKE/IKKε.
小核糖核酸病毒 VP2 蛋白通过选择性自噬降解 IKBKE/IKKε 来抑制先天免疫。
Female-biased astrocytic priming shapes early locus coeruleus vulnerability in an Aβ oligomer milieu.
女性偏向的星形胶质细胞启动在 Aβ 寡聚体环境中塑造了早期蓝斑核的脆弱性。
Fluorination augments tumor engagement and antitumor immunity of a D-peptide-based radiotheranostic agent for combinatorial immune checkpoint blockade therapy.
氟化作用增强了基于 D 肽的放射性治疗诊断剂的肿瘤结合和抗肿瘤免疫力，可用于联合免疫检查点阻断疗法。
Parasitic castration by a viral protein tyrosine phosphatase targeting the host cell cycle checkpoint protein Rad9A.
病毒蛋白酪氨酸磷酸酶靶向宿主细胞周期检查点蛋白Rad9A，导致寄生性去势。
The metabolite α-ketoglutarate induces AIM2-dependent PANoptosis through demethylase TET2.
代谢物α-酮戊二酸通过去甲基酶TET2诱导AIM2依赖性PANoptosis。
Itaconate and its derivatives ameliorate autoimmunity by suppressing Th17 cells via regulating mitophagy.
衣康酸及其衍生物通过调节线粒体自噬抑制 Th17 细胞，从而改善自身免疫性疾病。
Palmitic acid aggravates atopic dermatitis by regulating SGK1/NEDD4L-involved cutaneous neuroimmune inflammation through driving TRPV1 and MRGPRB2 S-palmitoylation.
棕榈酸通过驱动 TRPV1 和 MRGPRB2 S-棕榈酰化来调节 SGK1/NEDD4L 参与的皮肤神经免疫炎症，从而加剧特应性皮炎。
Proline-Mediated Inhibition of ATPIF1-mTOR Signaling Alleviates Radiation-Induced Macrophage Polarization and Colon Inflammation.
脯氨酸介导的 ATPIF1-mTOR 信号抑制可减轻辐射诱导的巨噬细胞极化和结肠炎症。
Extracellular Vesicles Derived from M2 Microglia and Enriched in miRâ27bâ3p Attenuate MitochondriaâDependent Endothelial Apoptosis via the MKK4/JNK Pathway and Alleviate BBB Disruption After Intracerebral Hemorrhage.
源自 M2 小胶质细胞且富含 miR'27b'3p 的细胞外囊泡通过 MKK4/JNK 通路减弱线粒体依赖性内皮细胞凋亡，并减轻脑出血后的血脑屏障破坏。
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