Anti-Collagen I + Collagen III antibody (Ab34710)
Collagen alpha-1(I) chain, Alpha-1 type I collagen, COL1A1, Collagen alpha-1(III) chain, COL3A1, Collagen alpha-2(I) chain, Alpha-2 type I collagen, COL1A2
详细信息
特异性
This product is not recommended for use under denaturing conditions in WB. We would suggest testing it under native conditions. Denaturing and reducing conditions will greatly diminish reactivity and selectivity of this antibody. Abcam does not test ab34710 with endogenous samples in WB. We do recommend to look at the guidelines for blotting large proteins. Customers have been successful using ab34710 in this application, please see reference Tillgren V et al. J Biol Chem 290:918-25; 2015. Positive Control: Human stomach, skin and adrenal gland tissue lysates.
背景信息
Anti-Collagen I + Collagen III antibody (ab34710) is a rabbit polyclonal antibody and is validated for use in ICC/IF, IHC-P and WB. Anti-Collagen I + Collagen III antibody (ab34710) was first used in a scientific publication in 1977 and has been cited over 1859 times in peer reviewed journals. It's performance in Western blot and IHC in human samples is trusted by the scientific community. Abcam's high quality validation processes ensure Anti-Collagen I + Collagen III antibody (ab34710) has high sensitivity and specificity. Anti-Collagen I + Collagen III antibody (ab34710) has 88 independent reviews from customers. Anti-Collagen I + Collagen III antibody (ab34710) specifically detects Collagen I + Collagen III (UniProt ID: P02452; P08123; Molecular weight: 95kDa; 94kDa) and is sold in 100 µg selling sizes. Top cited antibody to Collagen I and Collagen II on the market with >2200 citations. This reagent has >58 5 star reviews. Collagen I and Collagen III are essential for studying tissue structure, repair and fibrosis, aiding in research on skin, muscle and cardiovascular health.
文献引用 (1920)
IGSF11-VISTA is a critical and targetable immune checkpoint axis in diffuse midline glioma.
IGSF11-VISTA 是弥漫性中线胶质瘤中一个关键且可靶向的免疫检查点轴。
Biodegradable Mg(2+)-releasing piezoelectric scaffold for segmental bone defect repair.
用于节段性骨缺损修复的可生物降解的 Mg(2+) 释放压电支架。
Adaptable sliding hydrogels enable pericellular pocket formation while enhancing MSC chondrogenesis and survival in 3D.
适应性滑动水凝胶能够形成细胞周围囊袋，同时增强 MSC 在 3D 中的软骨形成和存活率。
CircAASS alleviates renal injury and fibrosis by regulating mitochondrial homeostasis in tubular epithelial cells.
CircAASS 通过调节肾小管上皮细胞中的线粒体稳态来减轻肾损伤和纤维化。
Tubule-Derived IFN-α Promotes GSDMD-Mediated Macrophage Pyroptosis to Drive Renal Inflammation and Fibrosis Through JAK2/STAT2 Activation.
肾小管衍生的 IFN-α 促进 GSDMD 介导的巨噬细胞焦亡，通过 JAK2/STAT2 激活驱动肾脏炎症和纤维化。
IL-4/STAT6-signaling Influences Local Inflammation and Regeneration Processes During Acute Pancreatitis and Promotes Fibrosis by a Direct Activation of Pancreatic Fibroblasts During Chronic Pancreatitis.
IL-4/STAT6 信号传导影响急性胰腺炎期间的局部炎症和再生过程，并通过直接激活慢性胰腺炎期间的胰腺成纤维细胞促进纤维化。
Lineage tracing reveals the origins and dynamics of macrophages in lung injury and repair.
谱系追踪揭示了巨噬细胞在肺损伤和修复中的起源和动态。
Modification of the dermal matrix by senescence associated lipids and its functional consequence.
衰老相关脂质对真皮基质的修饰及其功能后果。
Hyodeoxycholic acid attenuates atherosclerosis by antagonizing FXR and modulating the PD-1/mTORC1 signaling axis.
脱氧胆酸通过拮抗 FXR 和调节 PD-1/mTORC1 信号轴来减轻动脉粥样硬化。
Single-cell and spatial profiling highlights TB-induced myofibroblasts as drivers of lung pathology.
单细胞和空间分析表明，结核病诱导的肌成纤维细胞是肺部病理的驱动因素。
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