Anti-OPA1 antibody (Ab42364)
Optic atrophy protein 1 homolog
反应种属：Human, Mouse, Rat
抗原名称：OPA1
应用：WB, IP
详细信息
免疫原The exact immunogen used to generate this antibody is proprietary information.
宿主/亚型Rabbit / IgG
抗体类型Polyclonal
产品形式Liquid
纯化方式Affinity purification Immunogen
储存缓冲液pH: 7.4 Preservative: 0.02% Sodium azide Constituents: PBS, 1% BSA
储存条件运输条件: Blue Ice
推荐的短期储存时间: 1-2 weeks
推荐的短期储存条件: +4°C
推荐的长期储存条件: -20°C
储存信息: Avoid freeze / thaw cycle
文献引用 (153)
Trans-Omics Integration Reveals That the Kidney Contributes to Systemic Aging via Sexually Dimorphic Accumulation of Glycosphingolipids.
跨组学整合揭示肾脏通过糖鞘脂的性别二态性积累促进全身衰老。
Volitional exercise elicits physiological and molecular improvements in the severe D2.mdx mouse model of Duchenne muscular dystrophy.
自主运动可引起杜氏肌营养不良症严重 D2.mdx 小鼠模型的生理和分子水平改善。
Cadmium-Induced Mitochondrial and MAMs Dysregulation in Rat Testis: The Protective Role of D-Aspartate.
镉诱导大鼠睾丸线粒体和 MAMs 失调：D-天冬氨酸的保护作用。
Pla2g7 regulates bone homeostasis via Alox12/12-HETE/Gpr31 signaling axis.
Pla2g7 通过 Alox12/12-HETE/Gpr31 信号轴调节骨稳态。
The crucial function of IDO1 in pulmonary fibrosis: From the perspective of mitochondrial fusion in lung fibroblasts and targeted molecular inhibition.
IDO1 在肺纤维化中的关键作用：从肺成纤维细胞线粒体融合和靶向分子抑制的角度
A long-lived pool of PINK1 imparts a molecular memory of depolarization-induced activity
PINK1 的长寿命池赋予去极化诱导活性的分子记忆
Peroxiredoxin Ⅲ mitigates mitochondrial H2O2-mediated damage and supports quality control in cardiomyocytes under hypoxia-reoxygenation stress
过氧化物酶Ⅲ可减轻线粒体H2O2介导的损伤，并在缺氧-复氧应激下支持心肌细胞的质量控制。
Macrophage-derived mitochondria-rich extracellular vesicles aggravate bone loss in periodontitis by disrupting the mitochondrial dynamics of BMSCs
巨噬细胞来源的富含线粒体的细胞外囊泡通过破坏骨髓间充质干细胞的线粒体动力学，加剧牙周炎中的骨丢失。
mTOR inhibition triggers mitochondrial fragmentation in cardiomyocytes through proteosome-dependent prohibitin degradation and OPA-1 cleavage.
mTOR 抑制通过蛋白酶体依赖的prohibitin降解和 OPA-1 裂解引发心肌细胞线粒体断裂
Cerium dioxide nanoparticles coated with benzo[a]pyrene modify aryl hydrocarbon receptor activity, trophoblast differentiation and mitochondrial network phenotype in human placenta.
苯并[a]芘包覆的二氧化铈纳米颗粒可改变人类胎盘中的芳烃受体活性、滋养层分化和线粒体网络表型。
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