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Anti-PSD95 antibody [EPR23124-118] - Synaptic Marker (ab238135) was developed by Abcam using patented rabbit monoclonal antibody technology and is validated for use in IHC-Fr, IHC-P, IP, WB in human, mouse, rat samples. Anti-PSD95 antibody [EPR23124-118] - Synaptic Marker (ab238135) has been cited over 24 times in peer reviewed journals and is trusted by the scientific community. Abcams high quality manufacturing and validation processes ensure Anti-PSD95 antibody [EPR23124-118] - Synaptic Marker (ab238135) has high sensitivity and specificity alongside high lot-to-lot consistency and reproducibility. The specificity of Anti-PSD95 antibody [EPR23124-118] - Synaptic Marker (ab238135) has been confirmed by testing in knockout samples. Anti-PSD95 antibody [EPR23124-118] - Synaptic Marker (ab238135) has 4 independent reviews from customers. Anti-PSD95 antibody [EPR23124-118] - Synaptic Marker (ab238135) specifically detects PSD95 (UniProt ID: Q62108; Molecular weight: 80kDa) and is sold in 100 uL and 1 mL selling sizes. Conjugation-ready, carrier free format available for antibody clone EPR23124-118 - ab269863. Antibody clone EPR23124-118 is also available pre-conjugated to a variety of labels for your convenience - Alexa Fluor® 647, Alexa Fluor® 555, Alexa Fluor® 568 ( ab32566, ab32571, ab3286 ). Post Synaptic Density 95 kDa (PSD-95), also known as synapse-associated protein 90 kDa (SAP90), is a vital protein located in the postsynaptic density of neurons. It plays a key role in synaptic signaling and plasticity by organizing and anchoring various synaptic proteins, such as NMDA receptors and potassium channels. Changes in PSD-95 expression are associated with several neurological and psychiatric disorders, including schizophrenia, autism, and Alzheimer's disease (AD). Research on PSD-95 is crucial for understanding synaptic function and dysfunction, emphasizing its importance in the study of neurodegenerative and neuropsychiatric diseases. Patented technology Our RabMAb ® technology is a patented hybridoma-based technology for making rabbit monoclonal antibodies. For details on our patents, please refer to RabMAb ® patents.
文献引用 (90)
Piezo1-Targeted Magnetoacoustic Nanobubbles Rescue Alzheimer's Pathology by Electromechanical Neuromodulation and Amyloid-β Clearance.
靶向压电1的磁声纳米气泡通过电机械神经调节和淀粉样蛋白β清除来挽救阿尔茨海默病病理。
A bioinspired anisotropic anti-inflammatory scaffold enhances spinal nerve regeneration and neural circuit reconstruction via FGF13/Ca(2+)/CaMK2A/CREB pathway.
仿生各向异性抗炎支架通过 FGF13/Ca(2+)/CaMK2A/CREB 通路增强脊神经再生和神经回路重建。
The novel IDO-1 inhibitor 3-047 combined with icaritin ameliorates neuroinflammation and diabetes-associated cognitive dysfunction with suppression of TLR4/MyD88/NF-κB signaling.
新型 IDO-1 抑制剂 3-047 与淫羊藿苷联合使用，可通过抑制 TLR4/MyD88/NF-κB 信号传导来改善神经炎症和糖尿病相关的认知功能障碍。
Repeated Oral Methylphenidate Administration Evokes Changes in Brain Plasticity Proteins in Juvenile Wistar Kyoto Rats: Evidence for Sex-Related Differences.
重复口服哌甲酯引起幼年 Wistar Kyoto 大鼠脑可塑性蛋白的变化：性别差异的证据。
Ectoparasites enhance survival by suppressing host exploration and limiting dispersal.
体外寄生虫通过抑制宿主的探索行为和限制其扩散来提高自身的生存率
MS4A6A/Ms4a6d deficiency disrupts neuroprotective microglia functions and promotes inflammation in Alzheimer's disease model
MS4A6A/Ms4a6d 缺陷会破坏神经保护性小胶质细胞功能，并促进阿尔茨海默病模型中的炎症反应。
Repetitive unidirectional spinal tactile stimulation engages microglial Bmal1 pathways to promote synaptic remodeling in the mPFC of adolescent VPA-exposed mice.
重复的单向脊髓触觉刺激可激活小胶质细胞 Bmal1 通路，从而促进暴露于 VPA 的青少年小鼠 mPFC 中的突触重塑。
Microglial double stranded DNA accumulation induced by DNase II deficiency drives neuroinflammation and neurodegeneration
DNase II 缺乏诱导的小胶质细胞双链 DNA 积累会驱动神经炎症和神经退行性变。
The scaffold protein DLG4 facilitates RNF63-mediated ubiquitination and degradation of STAT3 in non-small cell lung cancer.
支架蛋白 DLG4 促进非小细胞肺癌中 RNF63 介导的 STAT3 泛素化和降解
TREM2 deficiency exacerbates cognitive impairment by aggravating α-Synuclein-induced lysosomal dysfunction in Parkinson's disease.
TREM2 缺乏会加剧帕金森病中 α-突触核蛋白诱导的溶酶体功能障碍，从而加重认知障碍
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