文献引用 (72)
Polystyrene Microplastics Exposure Aggravates Clear Cell Renal Cell Carcinoma Progression via the NF-κB and TGF-β Signaling Pathways.
聚苯乙烯微塑料暴露通过 NF-κB 和 TGF-β 信号通路加剧透明细胞肾细胞癌的进展。
Arginase 2 Promotes Colorectal Cancer Metastasis via PI3K/AKT Pathway Activation and Regulates Tumor Immune Infiltration.
精氨酸酶 2 通过 PI3K/AKT 通路激活促进结直肠癌转移并调节肿瘤免疫浸润。
Epidermal Stem Cell-Derived Extracellular Vesicles Induce Fibroblasts Mesenchymal-Epidermal Transition to Alleviate Hypertrophic Scar Formation via miR-200s Inhibition of ZEB1 and 2
表皮干细胞来源的细胞外囊泡通过miR-200抑制ZEB1和ZEB2诱导成纤维细胞间质-表皮转化，从而减轻肥厚性瘢痕形成。
Restoration of SIRT3 Expression in Aged Mice Alleviates UUO-Induced Renal Fibrosis by Reducing GSK-3β Hyperacetylation.
恢复老年小鼠的 SIRT3 表达可通过减少 GSK-3β 过度乙酰化来缓解 UUO 诱导的肾纤维化。
PTEN restoration and CXCR2 depletion synergistically enhance the effect of enzalutamide and inhibit bone metastatic CRPC.
PTEN 恢复和 CXCR2 耗竭协同增强恩扎卢胺的作用，并抑制骨转移性 CRPC
LncRNA small nucleolar RNA host gene 1 (SNHG1) mediates acidic bile salt-induced EMT via the ULK1-Notch1 axis in Barrett's esophagus.
LncRNA 小核仁 RNA 宿主基因 1 (SNHG1) 通过 ULK1-Notch1 轴介导 Barrett 食管中酸性胆汁盐诱导的 EMT
Distinct functional heterogeneity of TP53 R175 mutations in platinum-resistant ovarian cancer: unveiling molecular mechanisms and therapeutic targets.
TP53 R175 突变在铂耐药卵巢癌中具有独特的功能异质性：揭示分子机制和治疗靶点。
ESPN activates ZEB1-mediated EMT through the PI3K/AKT/mTOR axis to promote osteosarcoma metastasis.
ESPN 通过 PI3K/AKT/mTOR 轴激活 ZEB1 介导的 EMT，从而促进骨肉瘤转移
Nordihydroguaiaretic acid inhibits bladder cancer metastasis through suppression of α1,3-mannosyltransferase expression and LRFN4 N-glycosylation.
去甲二氢愈创木酸通过抑制α1,3-甘露糖基转移酶的表达和LRFN4 N-糖基化来抑制膀胱癌转移
Osteopontin derived from hypoxia-induced M2 macrophages promotes osteosarcoma progression through modulation of EGR3/ISG15 signaling and RIG-I expression
缺氧诱导的M2巨噬细胞来源的骨桥蛋白通过调节EGR3/ISG15信号通路和RIG-I表达促进骨肉瘤进展。
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