Anti-SQSTM1 / p62 antibody [2C11] - BSA and Azide free (Ab56416)
ORCA, OSIL, SQSTM1, Sequestosome-1, EBI3-associated protein of 60 kDa, Phosphotyrosine-independent ligand for the Lck SH2 domain of 62 kDa, Ubiquitin-binding protein p62, EBIAP, p60, p62
用户验证的应用
推荐稀释比例
- IHC-PPerform heat mediated antigen retrieval with citrate buffer pH 6 before commencing with IHC staining protocol.
- ICC/IFFix cells for 15 minutes with 2 mL of 4% paraformaldehyde solution (pH 7.4 with NaOH in PBS). Permeabilize cells by incubating for 15 minutes on ice with 2 mL of 0.1% Triton X-100 in PBS.
- Flow Cytab91361 or ab18415 - Mouse monoclonal IgG2a, is suitable for use as an isotype control with this antibody. We recommend Goat Anti-Mouse IgG H&L (DyLight ® 488) preadsorbed ( ab96879 ) secondary antibody
详细信息
背景信息
Product Specifications Anti-SQSTM1 / p62 antibody [2C11] - BSA and Azide free (ab56416) is a mouse monoclonal antibody provided in a carrier-free, conjugation ready format. ab56416 is validated in Flow Cyt, ICC/IF, IHC-P, IP, WB in human samples. Carrier-free antibodies are compatible with our best-in-class range of Lightning-Link® conjugation kits. Anti-SQSTM1 / p62 antibody [2C11] - BSA and Azide free (ab56416) specifically detects SQSTM1 / p62 (UniProt ID: Q13501; Molecular weight: 48kDa) and is sold in 100 ug selling sizes. Quality and Validation Antibody clone 2C11 was first used in a scientific publication in 1976 and has been cited over 1046 times in peer reviewed journals and is trusted by the scientific community. Anti-SQSTM1 / p62 antibody [2C11] - BSA and Azide free (ab56416) has 36 independent reviews from customers. The specificity of Anti-SQSTM1 / p62 antibody [2C11] - BSA and Azide free (ab56416) has been confirmed by testing in knockout samples.
文献引用 (1166)
GGC repeat expansions within new open reading frames are translated into toxic polyglycine proteins in oculopharyngodistal myopathy.
在眼咽远端肌病中，新的开放阅读框内的 GGC 重复扩增被翻译成有毒的聚甘氨酸蛋白。
VAMP7-dependent late endosomal secretion of ER and mitochondrial proteins impacts the tumor microenvironment and macrophage engagement.
VAMP7 依赖的内质网和线粒体蛋白的晚期内体分泌会影响肿瘤微环境和巨噬细胞的参与。
Internalized SNCA/α-synuclein fibrils become truncated and resist degradation in neurons while glial cells rapidly degrade SNCA fibrils.
神经元内化的 SNCA/α-突触核蛋白原纤维会发生截断并抵抗降解，而神经胶质细胞则会迅速降解 SNCA 原纤维。
CircAASS alleviates renal injury and fibrosis by regulating mitochondrial homeostasis in tubular epithelial cells.
CircAASS 通过调节肾小管上皮细胞中的线粒体稳态来减轻肾损伤和纤维化。
REEP1 Accumulation Disrupts ER Integrity and Drives Spinal Motoneuron Degeneration in Distal Hereditary Motor Neuropathy.
REEP1 积累破坏内质网完整性，导致远端遗传性运动神经病脊髓运动神经元退化。
CCDC41 Drives Oocyte Meiotic Progression by Promoting Rab11a/Rab7-Positive Vesicle Fusion with Target Membranes.
CCDC41 通过促进 Rab11a/Rab7 阳性囊泡与靶膜融合来驱动卵母细胞减数分裂进程。
Comparative neurotoxic effects of PFOA and GenX in hiPSC-derived cortical neurons.
PFOA 和 GenX 对 hiPSC 衍生皮层神经元的神经毒性作用比较。
Bimetallic Copper-Manganese Zeolitic Imidazolate Framework Nanozyme Scavenges Reactive Oxygen Species to Alleviate Osteoarthritis via Phosphoinositide 3-Kinase/Mammalian Target of Rapamycin Axis and Autophagic Flux Restoration.
双金属铜锰沸石咪唑骨架纳米酶通过磷脂酰肌醇3-激酶/哺乳动物雷帕霉素靶蛋白轴和自噬流恢复清除活性氧以缓解骨关节炎。
Calcium overload induced mitochondrial and lysosomal dysfunction is regulated by Tousled-like kinase in a-synucleinopathy.
α-突触核蛋白病中，钙超载引起的线粒体和溶酶体功能障碍受Tousled样激酶的调节。
MYO5A-mediated stabilization promotes the acquisition of fusion competence in sealed autophagosomes.
MYO5A介导的稳定作用促进了封闭自噬体获得融合能力。
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