Anti-NF-kB p65 (phospho S536) antibody [EP2294Y] (Ab76302)
NFKB3, RELA, Transcription factor p65, Nuclear factor NF-kappa-B p65 subunit, Nuclear factor of kappa light polypeptide gene enhancer in B-cells 3
详细信息
特异性
Stimulation may be required to allow detection of the phosphorylated protein. We recommend using NIH/3T3 (Mouse embryonic fibroblast) treated with 100nM Calyculin A for 30 minutes as a positive control.
背景信息
Product Specifications Anti-NF-kB p65 (phospho S536) antibody [EP2294Y] (ab76302) was developed by Abcam using patented rabbit monoclonal antibody technology and is validated for use in Dot blot, IP, WB in human, mouse, rat samples. Anti-NF-kB p65 (phospho S536) antibody [EP2294Y] (ab76302) specifically detects NF-kB p65 Phospho-S536 (UniProt ID: Q04206; Molecular weight: 60kDa) and is sold in a convenient trial size to enable initial testing (20 µL) and larger sizes for subsequent scaling up experiments (100 µL and 1 mL). Quality and Validation Abcam's high quality manufacturing and validation processes ensure Anti-NF-kB p65 (phospho S536) antibody [EP2294Y] (ab76302) has high sensitivity and specificity alongside high lot-to-lot consistency and reproducibility. Anti-NF-kB p65 (phospho S536) antibody [EP2294Y] (ab76302) has been cited over 247 times in peer reviewed journals and is trusted by the scientific community. Anti-NF-kB p65 (phospho S536) antibody [EP2294Y] (ab76302) has 7 independent reviews from customers. Patented technology Our RabMAb ® technology is a patented hybridoma-based technology for making rabbit monoclonal antibodies. For details on our patents, please refer to RabMAb ® patents.
文献引用 (503)
SphK1/S1P signaling-mediated crosstalk between pancreatic acinar cell and macrophage M1 polarization aggravates acute pancreatitis progression.
SphK1/S1P信号介导的胰腺腺泡细胞与巨噬细胞M1极化之间的串扰加剧了急性胰腺炎的进展。
DHCR24 Deficiency Drives Age-Related Meibomian Gland Dysfunction by Regulating Lipid Metabolic Imbalance and Cytosolic mtDNA-Induced cGAS-STING Activation.
DHCR24 缺乏通过调节脂质代谢失衡和胞质 mtDNA 诱导的 cGAS-STING 激活驱动与年龄相关的睑板腺功能障碍。
Mitochondrial DNA release via VDAC1 in keratinocytes: a key driver of innate immunity and vitiligo pathogenesis.
角质形成细胞中通过 VDAC1 释放线粒体 DNA：先天免疫和白癜风发病机制的关键驱动因素。
Exploring possible mechanisms of antiviral effect of Shufeng Jiedu Capsule on viral pneumonia: A comprehensive analysis.
探讨舒风解毒胶囊对病毒性肺炎的抗病毒作用机制：一项综合分析。
Icariin alleviates corneal neovascularization by regulating inflammation through inhibition of macrophage polarization and the NF-κB pathway.
淫羊藿苷通过抑制巨噬细胞极化和 NF-κB 通路来调节炎症，从而减轻角膜新生血管形成。
Inflammation-induced LncRNA SNHG1 orchestrates spermatogonium development in non-obstructive azoospermia via IL-17Â A signaling pathway.
炎症诱导的 LncRNA SNHG1 通过 IL-17A 信号通路调控非梗阻性无精子症中的精原细胞发育。
An Acebuche Oil-Enriched Diet Prevents Early-Stage Cerebrovascular Alterations in the 5xFAD Mouse Model of Alzheimer's Disease.
富含醋栗油的饮食可预防 5xFAD 阿尔茨海默病小鼠模型早期脑血管改变。
Integrated Transcriptomics and Experimental Validation Reveal That Ellagic Acid Alleviates Fuchs Endothelial Corneal Dystrophy via PLAU/NF-κB Signaling
整合转录组学和实验验证表明，鞣花酸通过PLAU/NF-κB信号通路缓解Fuchs内皮角膜营养不良
Morpho-Functional Characterization and miRNA Profiling of the Retina in the 5xFAD Murine Model of Alzheimer's Disease.
5xFAD 阿尔茨海默病小鼠模型视网膜的形态功能特征及 miRNA 分析。
Targeting CRBN With Thalidomide Suppresses Fibrosis After Glaucoma Drainage Surgery Through NF-κB Pathway Inhibition.
通过沙利度胺靶向 CRBN，抑制 NF-κB 通路，从而抑制青光眼引流手术后的纤维化。
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