Anti-NeuN antibody [1B7] - Neuronal Marker (Ab104224)
RNA binding protein fox-1 homolog 3, Fox-1 homolog C, Neuronal nuclei antigen, NeuN antigen, RBFOX3
用户验证的应用
推荐稀释比例
- IHC-P5 µg/mL
- WB1/1000 - 1/2000
- ICC/IF1 µg/mL
详细信息
背景信息
Anti-NeuN antibody [1B7] - Neuronal Marker (ab104224) is a mouse monoclonal antibody and is validated for use in ICC/IF, IHC-P and WB. Anti-NeuN antibody [1B7] - Neuronal Marker (ab104224) was first used in a scientific publication in 2013 and has been cited over 621 times in peer reviewed journals. It's performance in Western blot, immunofluorescence and IHC in human, mouse and rat samples is trusted by the scientific community. Abcam's high quality validation processes ensure Anti-NeuN antibody [1B7] - Neuronal Marker (ab104224) has high sensitivity and specificity. Anti-NeuN antibody [1B7] - Neuronal Marker (ab104224) has 26 independent reviews from customers. Anti-NeuN antibody [1B7] - Neuronal Marker (ab104224) specifically detects NeuN (UniProt ID: A6NFN3; Molecular weight: 34kDa) and is sold in 100 ug selling sizes. NeuN is a widely used marker in neuro research for identifying mature neurons. It is expressed in the nuclei and, to a lesser extent, the cytoplasm of post-mitotic neurons. Researchers use NeuN to assess neuronal density and integrity, making it essential for studying neuronal development, neurodegenerative diseases and brain injuries. Its reliable expression in mature neurons helps in evaluating neuronal loss and therapeutic effects in various neurological conditions.
文献引用 (986)
Melatonin-incorporated brain extracellular matrix hydrogel enhances NSCs mitochondrial metabolism to promote neuroregeneration via the AMPK-PGC-1α-NRF1/TFAM axis after spinal cord injury.
褪黑素掺入的脑细胞外基质水凝胶增强神经干细胞线粒体代谢，通过 AMPK-PGC-1α-NRF1/TFAM 轴促进脊髓损伤后的神经再生。
3D-MSCs apoptotic bodies-integrated conductive hydrogel mitigates spinal cord injury via immunoregulation and alleviating neuronal pyroptosis.
3D-MSCs凋亡小体整合导电水凝胶通过免疫调节和减轻神经元焦亡来减轻脊髓损伤。
Metabolites released from apoptotic cells in central nervous system orchestrates the pathological process of Alzheimer disease through improving autophagy.
中枢神经系统凋亡细胞释放的代谢物通过改善自噬作用来调控阿尔茨海默病的病理过程。
Cerebral FURIN deficiency impairs astrocytic lipophagy through ITGAV maturation.
脑内 FURIN 缺乏会通过 ITGAV 成熟损害星形胶质细胞的脂质自噬。
Autophagy Activators Normalize Aberrant Tau Proteostasis and Rescue Synapses in Human Familial Alzheimer's Disease iPSC-Derived Cortical Organoids.
自噬激活剂可使异常的 Tau 蛋白稳态正常化，并挽救人类家族性阿尔茨海默病 iPSC 衍生皮质类器官中的突触。
Spinal α2δ-1 induces GluA3 degradation to regulate assembly of calcium-permeable AMPA receptors and pain hypersensitivity.
脊髓α2α-1诱导GluA3降解，从而调节钙离子通透性AMPA受体的组装和疼痛过敏。
Subtype-Specific Roles of Anterior Cingulate Cortex Neurons in Pain-Induced Social Deficits in Mice.
前扣带皮层神经元亚型特异性作用在小鼠疼痛诱发的社交缺陷中的作用。
Near-infrared light-activated lidocaine microneedle patch for rapid local anesthesia.
近红外光激活利多卡因微针贴片，用于快速局部麻醉。
Hyodeoxycholic acid relieves neuropathic pain by activating farnesoid X receptor signaling.
脱氧胆酸通过激活法尼醇X受体信号传导来缓解神经性疼痛。
HMGCS2-dependent β-OHB/H3K9bhb ameliorates synaptic plasticity and cognition in Alzheimer's disease.
HMGCS2 依赖的 β-OHB/H3K9bhb 可改善阿尔茨海默病患者的突触可塑性和认知能力。
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