用户验证的应用
推荐稀释比例
- IHC-P1/1000
- WB1/1000
- Flow Cyt1/500
- IP1/30
详细信息
背景信息
Product Specifications Anti-CD9 antibody [EPR23105-125] (ab263019) was developed by Abcam using patented rabbit monoclonal antibody technology and is validated for use in Flow Cyt, IHC-P, IP, WB in human samples. Anti-CD9 antibody [EPR23105-125] (ab263019) specifically detects CD9 (UniProt ID: P21926; Molecular weight: 25kDa) and is sold in 100 µL and 1 mL selling sizes. Quality and Validation Abcam's high quality manufacturing and validation processes ensure Anti-CD9 antibody [EPR23105-125] (ab263019) has high sensitivity and specificity alongside high lot-to-lot consistency and reproducibility. The specificity of Anti-CD9 antibody [EPR23105-125] (ab263019) has been confirmed by testing in knockout samples. Anti-CD9 antibody [EPR23105-125] (ab263019) has been cited over 22 times in peer reviewed journals and is trusted by the scientific community. Related Products Conjugation-ready, carrier free format available for antibody clone EPR23105-125 - ab263024. Antibody clone EPR23105-125 is also available pre-conjugated to a variety of labels for your convenience - Alexa Fluor® 488 ( ab316875 ). Target Information CD9 also known as p24 is a member of the tetraspanin family, which includes proteins involved in various cellular processes. CD9 (p24) plays a significant role in cancer progression and metastasis by influencing cell adhesion, migration, and signaling. CD9 (p24) is found on the surface of cancer cells and within exosomes, impacting cancer progression and therapy resistance. CD9's role in cancer is complex, as it can both suppress and promote tumor growth depending on the context. The protein's interactions within tetraspanin-enriched microdomains (TEMs) are crucial for its function. Patented technology Our RabMAb ® technology is a patented hybridoma-based technology for making rabbit monoclonal antibodies. For details on our patents, please refer to RabMAb ® patents. What are the advantages of a recombinant monoclonal antibody? This product is a recombinant monoclonal antibody, which offers several advantages including - High batch-to-batch consistency and reproducibility - Improved sensitivity and specificity - Long-term security of supply - Animal-free batch production For more information, read more on recombinant antibodies.
文献引用 (89)
MicroRNA-493-5p engineered exosomes delivered via piezoelectric microneedles for epigenetic modulation of macrophages in diabetic wound healing.
利用压电微针递送经 MicroRNA-493-5p 工程改造的外泌体,对糖尿病伤口愈合中的巨噬细胞进行表观遗传调控。
Extracellular vesicles from TNF-α and TGF-β1-treated ADSCs promote tissue-engineered cartilage regeneration by modulating macrophages via the miR-378a-3p/SIRPα axis.
TNF-α 和 TGF-β1 处理的 ADSC 的细胞外囊泡通过 miR-378a-3p/SIRPα 轴调节巨噬细胞,从而促进组织工程软骨再生。
MSC-Derived Exosomal lnc-AGT-3: A Novel Anti-Angiogenic Target in Age-Related Macular Degeneration Through p53 Signaling Pathway.
MSC衍生的外泌体lnc-AGT-3:通过p53信号通路在年龄相关性黄斑变性中发挥新型抗血管生成作用。
MYO1C is a urinary extracellular vesicle biomarker and mediator of podocyte injury in diabetic nephropathy.
MYO1C 是尿液细胞外囊泡生物标志物,也是糖尿病肾病中足细胞损伤的介质。
Wilforine attenuates inflammatory osteolysis by suppressing osteoclast fusion through JAK-STAT-stomatin immunoregulatory signaling.
Wilforine 通过 JAK-STAT-stomatin 免疫调节信号抑制破骨细胞融合,从而减轻炎症性骨溶解。
Proteomic Analysis of Small Extracellular Vesicles From Lymphatic Affluents in Developing Premetastatic Niche in Melanoma.
黑色素瘤转移前微环境淋巴渗出液中小细胞外囊泡的蛋白质组学分析。
TSG-6 Activated MSC-derived Extracellular Vesicles Present Altered micro-RNA Contents and Ameliorate the Inflammatory Phenotype of Macrophages in Vitro.
TSG-6 激活的 MSC 衍生的细胞外囊泡表现出改变的 microRNA 含量,并改善了体外巨噬细胞的炎症表型。
Adipose extracellular vesicles carrying miR-210-3p drive macrophage inflammation and nicotine-induced atherosclerosis.
携带 miR-210-3p 的脂肪细胞外囊泡驱动巨噬细胞炎症和尼古丁诱导的动脉粥样硬化。
Tongue squamous cell carcinoma exosomal miR-122-5p promotes the conversion from fibroblasts to inflammatory cancer-associated fibroblasts.
舌鳞状细胞癌外泌体 miR-122-5p 促进成纤维细胞向炎症性癌症相关成纤维细胞的转化。
hUMSCs-exo@Cyasterone protects the cell model of steroid-induced femur head necrosis by regulating N-glycosylation modification of CTSD-N258A.
hUMSCs-exo@Cyasterone 通过调节 CTSD-N258A 的 N-糖基化修饰来保护类固醇诱导的股骨头坏死的细胞模型。
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