Anti-VDAC1/Porin + VDAC3 antibody [20B12AF2] - Mitochondrial Loading Control (Ab14734)

VDAC, VDAC1, Non-selective voltage-gated ion channel VDAC1, Outer mitochondrial membrane protein porin 1, Plasmalemmal porin, Porin 31HL, Porin 31HM, Voltage-dependent anion-selective channel protein 1, VDAC-1, hVDAC1, Non-selective voltage-gated ion channel VDAC3, VDAC-3, hVDAC3, Outer mitochondrial membrane protein porin 3

反应种属:Human, Mouse, Rat, Bovine
抗原名称:VDAC1
应用:WB, FCM, IF/ICC

详细信息

免疫原The exact immunogen used to generate this antibody is proprietary information.
宿主/亚型Mouse / IgG2b
抗体类型Monoclonal
产品形式Liquid
储存缓冲液pH: 7.5 Preservative: 0.02% Sodium azide Constituents: 0.88% Sodium chloride, 0.36% HEPES
储存条件运输条件: Blue Ice 推荐的短期储存条件: +4°C 推荐的长期储存条件: +4°C 储存信息: Do Not Freeze
无载体蛋白No

背景信息

Product Specifications Anti-VDAC1/Porin + VDAC3 antibody [20B12AF2] (ab14734) is a mouse monoclonal antibody and is validated for use in Flow Cyt, ICC/IF, WB in cow, human, mouse, rat samples. Anti-VDAC1/Porin + VDAC3 antibody [20B12AF2] (ab14734) specifically detects VDAC1/Porin + VDAC3 (UniProt ID: P21796; Molecular weight: 31kDa) and is sold in 100 µg selling sizes. Quality and Validation Abcam's high quality validation processes ensure Anti-VDAC1/Porin + VDAC3 antibody [20B12AF2] (ab14734) has high sensitivity and specificity. The specificity of Anti-VDAC1/Porin + VDAC3 antibody [20B12AF2] (ab14734) has been confirmed by testing in knockout samples. Anti-VDAC1/Porin + VDAC3 antibody [20B12AF2] (ab14734) has been cited over 584 times in peer reviewed journals and is trusted by the scientific community. Anti-VDAC1/Porin + VDAC3 antibody [20B12AF2] (ab14734) has 51 independent reviews from customers. Want a custom formulation? This antibody clone is manufactured by Abcam. If you require a custom buffer formulation or conjugation for your experiments, please contact orders@abcam.com

文献引用 (552)

Small molecules restore mutant mitochondrial DNA polymerase activity

小分子可恢复突变线粒体DNA聚合酶活性

Sebastian Valenzuela ,Xuefeng Zhu ,Bertil Macao ,Mattias Stamgren ,Carol Geukens ,Paul S Charifson ,Gunther Kern ,Emily Hoberg ,Louise Jenninger ,Anja V Gruszczyk ,Seoeun Lee ,Katarina A S Johansson ,Javier Miralles Fusté ,Yonghong Shi ,S Jordan Kerns ,Laleh Arabanian ,Gabriel Martinez Botella ,Sofie Ekström ,Jeremy Green ,Andrew M Griffin ,Carlos Pardo-Hernández ,Thomas A Keating ,Barbara Küppers-Munther ,Nils-Göran Larsson ,Cindy Phan ,Viktor Posse ,Juli E Jones ,Xie Xie ,Simon Giroux ,Claes M Gustafsson ,Maria Falkenberg

Engineering mtDNA deletions by reconstituting end joining in human mitochondria

通过在人类线粒体中重建末端连接来构建线粒体DNA缺失

Yi Fu ,Max Land ,Tamar Kavlashvili ,Ruobing Cui ,Minsoo Kim ,Emily DeBitetto ,Toby Lieber ,Keun Woo Ryu ,Elim Choi ,Ignas Masilionis ,Rahul Saha ,Meril Takizawa ,Daphne Baker ,Marco Tigano ,Caleb A Lareau ,Ed Reznik ,Roshan Sharma ,Ronan Chaligne ,Craig B Thompson ,Dana Pe'er ,Agnel Sfeir

LONP1 regulation of mitochondrial protein folding provides insight into beta cell failure in type 2 diabetes.

LONP1 对线粒体蛋白折叠的调控为 2 型糖尿病中 β 细胞功能衰竭提供了见解

Li Jin, Deng Yamei, Gasser Marie, Zhu Jie, Walker Emily M, Sidarala Vaibhav, Reck Emma C, Hubers Dre L, Pasmooij Mabelle B, Shin Chun-Shik, Bandesh Khushdeep, Motakis Eftyhmios, Nargund Siddhi, Kursawe Romy, Basrur Venkatesha, Nesvizhskii Alexey I, Stitzel Michael L, Chan David C, Rutter Guy A, Soleimanpour Scott A

TMEM65 regulates and is required for NCLX-dependent mitochondrial calcium efflux.

TMEM65 调节 NCLX 依赖的线粒体钙外流,并且是其必需的

Garbincius Joanne F, Salik Oniel, Cohen Henry M, Choya-Foces Carmen, Mangold Adam S, Makhoul Angelina D, Schmidt Anna E, Khalil Dima Y, Doolittle Joshua J, Wilkinson Anya S, Murray Emma K, Lazaropoulos Michael P, Hildebrand Alycia N, Tomar Dhanendra, Elrod John W

TANGO2 binds crystallin alpha B and its loss causes desminopathy.

TANGO2 与晶状体蛋白 α B 结合,其缺失会导致结蛋白病

Stentenbach Maike, Hughes Laetitia A, Fagan Samuel V, Payne Blake, Rudler Danielle L, Siira Stefan J, McCubbin Tim, Chopin Anaëlle, Perks Kara L, Ermer Judith A, Hendry James, Er Teagan S, Balasubramaniam Shanti, Eliades Joel A, Hool Livia C, Packer Nicolle H, Moh Edward S X, Padman Benjamin S, Rackham Oliver, Filipovska Aleksandra

Noncanonical function of Pannexin1 promotes cellular senescence and renal fibrosis post-acute kidney injury.

Pannexin1 的非经典功能促进急性肾损伤后的细胞衰老和肾纤维化

Huang Liuwei, Shen Yanting, Pan Xiaoling, Li Jiaqi, Li Caizhen, Ruan Lixin, He Sitan, Huang Lanlan, Liu Kangyi, Zhao Xin, Geng Jian, Guo Jie, Hou Fan Fan, Wang Jun

Single cell suppression profiling of human regulatory T cells

人类调节性T细胞的单细胞抑制谱分析

Jonas Nørskov Søndergaard ,Janyerkye Tulyeu ,David Priest ,Shimon Sakaguchi ,James B Wing

Mitochondrial bioenergetics stimulates autophagy for pathological MAPT/Tau clearance in tauopathy neurons

线粒体生物能量学刺激自噬,清除 tau 病神经元中的病理性 MAPT/Tau

Nuo Jia, Dhasarathan Ganesan, Hongyuan Guan, Yu Young Jeong, Sinsuk Han, Gavesh Rajapaksha, Marialaina Nissenbaum, Alexander W Kusnecov, Qian Cai

Endogenous interactomes of MFN1 and MFN2 provide novel insights into interorganelle communication and autophagy.

MFN1 和 MFN2 的内源性相互作用组为细胞器间通讯和自噬提供了新的见解

Gordaliza-Alaguero Isabel, Sànchez-Fernàndez-de-Landa Paula, Radivojevikj Dragana, Villarreal Laura, Arauz-Garofalo Gianluca, Gay Marina, Martinez-Vicente Marta, Seco Jorge, Martín-Malpartida Pau, Vilaseca Marta, Macías María J, Palacin Manuel, Ivanova Saška, Zorzano Antonio

Poor glycaemic control in type 2 diabetes compromises leukocyte oxygen consumption rate, OXPHOS complex content and neutrophil-endothelial interactions.

2 型糖尿病血糖控制不佳会损害白细胞耗氧率、氧化磷酸化复合物含量和中性粒细胞-内皮细胞相互作用

Cacace Julia, Luna-Marco Clara, Hermo-Argibay Alberto, Pesantes-Somogyi Catherine, Hernández-López Omar A, Pelechá-Salvador María, Bañuls Celia, Apostolova Nadezda, de Miguel-Rodríguez Luis, Morillas Carlos, Rocha Milagros, Rovira-Llopis Susana, Víctor Víctor M
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