Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (Ab14748)

ATP5A, ATP5A1, ATP5AL2, ATPM, ATP5F1A, ATP synthase F1 subunit alpha

反应种属:Human, Mouse, Rat, Bovine, Drosophila
抗原名称:ATP5F1A
应用:WB, IHC, FCM, IF/ICC

详细信息

宿主/亚型Mouse / IgG2b
抗体类型Monoclonal
产品形式Liquid
储存缓冲液pH: 7.5 Preservative: 0.02% Sodium azide Constituents: HEPES buffered saline
储存条件运输条件: Blue Ice 推荐的短期储存条件: +4°C 推荐的长期储存条件: +4°C
无载体蛋白No

背景信息

Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) is a mouse monoclonal antibody and is validated for use in Flow Cyt, ICC/IF, IHC-P, WB in human, mouse, rat samples. Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) has been cited over 553 times in peer reviewed journals and is trusted by the scientific community. Abcam's high quality validation processes ensure Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) has high sensitivity and specificity. Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) has 25 independent reviews from customers. Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) specifically detects ATP5A (UniProt ID: P25705; Molecular weight: 55kDa) and is sold in 100 µg selling sizes. Antibody clone 15H4C4 is also available pre-conjugated to FITC for your convenience ( ab119688 ). ATP5A (ATP Synthase F1 Subunit Alpha) is an essential protein for ATP production in mitochondria, forming part of the ATP synthase complex. This complex is crucial for oxidative phosphorylation, the process by which cells generate energy. Mutations in the ATP5A gene are linked to mitochondrial disorders, including combined oxidative phosphorylation deficiency 22 (COXPD22), which manifests as severe metabolic and neurological issues, emphasizing the protein's critical role in energy metabolism. Want a custom formulation? This antibody clone is manufactured by Abcam. If you require a custom buffer formulation or conjugation for your experiments, please contact orders@abcam.com

文献引用 (594)

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ALKBH1通过tRNA解码重编程和密码子偏向性翻译驱动肿瘤发生和耐药性

Chao Shen # ,Yuan Che # ,Keren Zhou # ,Kitty Wang # ,Wei Li # ,Diyuan Xue # ,Tong Wu ,Lu Yang ,Meiling Chen ,Yue Sheng ,Chengwan Zhang ,Sean Robinson ,Huiying Chen ,Lillian Sau ,Zhenhua Chen ,Mark Wunderlich ,Li Han ,Tingting Tang ,Ying Qing ,Dong Wu ,Miao Sun ,Keith Leung ,Katarzyna Dąbrowska ,Patrick Pirrotte ,Yueh-Chiang Hu ,Lucy Y Ghoda ,Bin Zhang ,James C Mulloy ,Minjie Wei ,Guido Marcucci ,Chuan He ,Xiaolan Deng ,Jianjun Chen

Copper Drives Remodeling of Metabolic State and Progression of Clear Cell Renal Cell Carcinoma

铜驱动代谢状态重塑和透明细胞肾细胞癌的进展

Megan E Bischoff # ,Behrouz Shamsaei # ,Juechen Yang # ,Dina Secic # ,Bhargav Vemuri ,Julie A Reisz ,Angelo D'Alessandro ,Caterina Bartolacci ,Rafal Adamczak ,Lucas Schmidt ,Jiang Wang ,Amelia Martines ,Jahnavi Venkat ,Vanina Toffessi Tcheuyap ,Jacek Biesiada ,Catherine A Behrmann ,Katherine E Vest ,James Brugarolas ,Pier Paolo Scaglioni ,David R Plas ,Krushna C Patra ,Shuchi Gulati ,Julio A Landero Figueroa ,Jarek Meller ,John T Cunningham ,Maria F Czyzyk-Krzeska

2-hydroxyglutarate mediates whitening of brown adipocytes coupled to nuclear softening upon mitochondrial dysfunction.

2-羟基戊二酸介导棕色脂肪细胞变白,并伴随线粒体功能障碍引起的细胞核软化

Kaul Harshita, Isermann Lea, Senft Katharina, Popovic Milica, Georgomanolis Theodoros, Baumann Linda, Sivanesan Pujyanathan, Pouikli Andromachi, Nolte Hendrik, Lucic Bojana, Hildebrandt Ximena, Seidel Katrin, Gnad Thorsten, Gaedke Felix, Göbel Ulrike, Peters Franziska, Cherevatenko Maksym, Park Joo Hyun, Schauss Astrid, Peltzer Nieves, Brüning Jens Claus, Kornfeld Jan-Wilhelm, Pfeifer Alexander, Langer Thomas, Lusic Marina, Wickström Sara A, Frezza Christian, Trifunovic Aleksandra

In situ architecture of the human prohibitin complex.

人类抑制蛋白复合物的原位结构

Lange Felix, Ratz Michael, Dohrke Jan-Niklas, Le Vasseur Maxence, Wenzel Dirk, Ilgen Peter, Riedel Dietmar, Jakobs Stefan

Cardiomyocyte OTUD1 drives diabetic cardiomyopathy via directly deubiquitinating AMPKα2 and inducing mitochondrial dysfunction.

心肌细胞 OTUD1 通过直接去泛素化 AMPKα2 并诱导线粒体功能障碍来驱动糖尿病心肌病

Han Xue, Zheng Ruyi, Zhang Jiajia, Liu Yanan, Li Ze, Liu Guoxuan, Zheng Jianing, Li Weiqi, Liang Zijun, Wang Mengyang, Yu Jie, Shi Qiaojuan, Ying Huazhong, Liang Guang

TANGO2 binds crystallin alpha B and its loss causes desminopathy.

TANGO2 与晶状体蛋白 α B 结合,其缺失会导致结蛋白病

Stentenbach Maike, Hughes Laetitia A, Fagan Samuel V, Payne Blake, Rudler Danielle L, Siira Stefan J, McCubbin Tim, Chopin Anaëlle, Perks Kara L, Ermer Judith A, Hendry James, Er Teagan S, Balasubramaniam Shanti, Eliades Joel A, Hool Livia C, Packer Nicolle H, Moh Edward S X, Padman Benjamin S, Rackham Oliver, Filipovska Aleksandra

Tumour initiated purinergic signalling promotes cardiomyocyte RBFOX1 degradation and cardiotoxicity from DNA damaging anticancer agents.

肿瘤引发的嘌呤能信号传导促进心肌细胞 RBFOX1 降解,并导致 DNA 损伤性抗癌药物产生心脏毒性

Tejay Saymon, Lorenzana-Carrillo Maria Areli, Huang Guocheng, Tabatabaei Dakhili Seyed Amirhossein, Zhao Yuan -Yuan, Eaton Farah, Mendiola Pla Michelle, Bowles Dawn E, Kinnaird Adam, Paterson D Ian, Pituskin Edith, Ussher John R, Michelakis Evangelos D, Sutendra Gopinath

ROMO1 overexpression protects the mitochondrial cysteinome from oxidations in aging.

ROMO1 过表达可保护线粒体半胱氨酸组免受衰老过程中的氧化损伤

Xu Fengli, Huang Haipeng, Peng Kun, Jian Chongshu, Wu Hao, Jing Zhiwen, Qiu Shan, Chen Ying, Liu Keke, Fu Ling, Wang Yanru, Yang Jing, Duan Xiaotao, Wang Chu, Cheng Heping, Wang Xianhua

Nuclear receptor signaling regulates compartmentalized phosphatidylcholine remodeling to facilitate thermosensitive lipid droplet fusion

核受体信号传导调节区室化磷脂酰胆碱重塑,从而促进热敏性脂滴融合。

Qi Li,Xiaofang Zhou,Xiaocong Zhang,Chuqi Zhang,Shaobing O Zhang

SLC25A38 is required for mitochondrial pyridoxal 5'-phosphate (PLP) accumulation

SLC25A38是线粒体吡哆醛5'-磷酸(PLP)积累所必需的。

Izabella A Pena,Jeffrey S Shi,Sarah M Chang,Jason Yang,Samuel Block,Charles H Adelmann,Heather R Keys,Preston Ge,Shveta Bathla,Isabella H Witham,Grzegorz Sienski,Angus C Nairn,David M Sabatini,Caroline A Lewis,Nora Kory,Matthew G Vander Heiden,Myriam Heiman
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