Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (Ab14748)
ATP5A, ATP5A1, ATP5AL2, ATPM, ATP5F1A, ATP synthase F1 subunit alpha
详细信息
背景信息
Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) is a mouse monoclonal antibody and is validated for use in Flow Cyt, ICC/IF, IHC-P, WB in human, mouse, rat samples. Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) has been cited over 553 times in peer reviewed journals and is trusted by the scientific community. Abcam's high quality validation processes ensure Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) has high sensitivity and specificity. Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) has 25 independent reviews from customers. Anti-ATP5A antibody [15H4C4] - Mitochondrial Marker (ab14748) specifically detects ATP5A (UniProt ID: P25705; Molecular weight: 55kDa) and is sold in 100 µg selling sizes. Antibody clone 15H4C4 is also available pre-conjugated to FITC for your convenience ( ab119688 ). ATP5A (ATP Synthase F1 Subunit Alpha) is an essential protein for ATP production in mitochondria, forming part of the ATP synthase complex. This complex is crucial for oxidative phosphorylation, the process by which cells generate energy. Mutations in the ATP5A gene are linked to mitochondrial disorders, including combined oxidative phosphorylation deficiency 22 (COXPD22), which manifests as severe metabolic and neurological issues, emphasizing the protein's critical role in energy metabolism. Want a custom formulation? This antibody clone is manufactured by Abcam. If you require a custom buffer formulation or conjugation for your experiments, please contact orders@abcam.com
文献引用 (594)
ALKBH1 Drives Tumorigenesis and Drug Resistance via tRNA-decoding Reprogramming and Codon-biased Translation
ALKBH1通过tRNA解码重编程和密码子偏向性翻译驱动肿瘤发生和耐药性
Copper Drives Remodeling of Metabolic State and Progression of Clear Cell Renal Cell Carcinoma
铜驱动代谢状态重塑和透明细胞肾细胞癌的进展
2-hydroxyglutarate mediates whitening of brown adipocytes coupled to nuclear softening upon mitochondrial dysfunction.
2-羟基戊二酸介导棕色脂肪细胞变白,并伴随线粒体功能障碍引起的细胞核软化
In situ architecture of the human prohibitin complex.
人类抑制蛋白复合物的原位结构
Cardiomyocyte OTUD1 drives diabetic cardiomyopathy via directly deubiquitinating AMPKα2 and inducing mitochondrial dysfunction.
心肌细胞 OTUD1 通过直接去泛素化 AMPKα2 并诱导线粒体功能障碍来驱动糖尿病心肌病
TANGO2 binds crystallin alpha B and its loss causes desminopathy.
TANGO2 与晶状体蛋白 α B 结合,其缺失会导致结蛋白病
Tumour initiated purinergic signalling promotes cardiomyocyte RBFOX1 degradation and cardiotoxicity from DNA damaging anticancer agents.
肿瘤引发的嘌呤能信号传导促进心肌细胞 RBFOX1 降解,并导致 DNA 损伤性抗癌药物产生心脏毒性
ROMO1 overexpression protects the mitochondrial cysteinome from oxidations in aging.
ROMO1 过表达可保护线粒体半胱氨酸组免受衰老过程中的氧化损伤
Nuclear receptor signaling regulates compartmentalized phosphatidylcholine remodeling to facilitate thermosensitive lipid droplet fusion
核受体信号传导调节区室化磷脂酰胆碱重塑,从而促进热敏性脂滴融合。
SLC25A38 is required for mitochondrial pyridoxal 5'-phosphate (PLP) accumulation
SLC25A38是线粒体吡哆醛5'-磷酸(PLP)积累所必需的。
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