Anti-beta Catenin antibody (Ab16051)
CTNNB, OK/SW-cl.35, PRO2286, CTNNB1, Catenin beta-1, Beta-catenin
详细信息
背景信息
What is this antibody validated in? Anti-beta Catenin antibody (ab16051) is a rabbit polyclonal antibody and is validated for use in Western Blot (WB), Immunohistochemistry (IHC-P), Immunocytochemistry/immunofluorescence (ICC/IF), ELISA in Human samples. What is the molecular weight of beta Catenin? Anti-beta Catenin (ab16051) specifically detects a band for beta Catenin (UniProt: P35222) at a molecular weight of 94kDa. Trusted by the scientific community Anti-beta Catenin (ab16051) was first used in a scientific publication in 2005 and has been cited over 330 times in peer-reviewed journals. Reviewed by scientists Anti-beta Catenin (ab16051) has over 30 independent reviews from customers. Specificity confirmed The specificity of Anti-beta Catenin antibody (ab16051) has been confirmed by Western blot testing in CTNNB1 Knockout HAP1 cells.
文献引用 (315)
MiR-378 mediates the ovariectomy induced bone loss via exaggerating osteoclastogenesis and transforming growth factor beta impaired osteogenesis.
miR-378 通过加剧破骨细胞生成和转化生长因子β损害成骨作用来介导卵巢切除术引起的骨丢失。
Inhibiting L-type calcium channel promotes cardiomyocyte proliferation through activating the canonical Wnt signaling pathway.
抑制 L 型钙通道可通过激活经典的 Wnt 信号通路促进心肌细胞增殖。
HBx hijacks the miR-19a-3p/BAMBI/TGF-β1 axis to impair the anti-tumour activity of CD4(+) T cells in diffuse large B-cell lymphoma.
HBx 劫持 miR-19a-3p/BAMBI/TGF-β1 轴，从而削弱弥漫性大 B 细胞淋巴瘤中 CD4(+) T 细胞的抗肿瘤活性。
Acute myeloid leukemia (AML)-derived bone mesenchymal stem cell exosomal METTL14 promotes AML cell growth and glycolysis by HOXA3/WNT7B axis.
急性髓系白血病 (AML) 来源的骨髓间充质干细胞外泌体 METTL14 通过 HOXA3/WNT7B 轴促进 AML 细胞生长和糖酵解。
Andrographolide targets syndecan4 to impair its interaction with syntenin and inhibits the biogenesis of small extracellular vesicles.
穿心莲内酯靶向 syndecan4，削弱其与 syntenin 的相互作用，并抑制小细胞外囊泡的生物发生。
Wnt-directed CXCL12-expressing apical papilla progenitor cells drive tooth root formation
Wnt信号通路调控的表达CXCL12的牙根尖乳头祖细胞驱动牙根形成
PB2 and NP of North American H5N1 virus drive immune cell replication and systemic infections.
北美H5N1病毒的PB2和NP蛋白驱动免疫细胞复制和全身感染。
Chronic sublethal exposure to methylmercury lengthens telomeres in developing zebra finches.
长期亚致死剂量接触甲基汞会延长发育中的斑胸草雀的端粒。
Evaluation of Tideglusib as a Disease Modifying Therapy in Murine Models of Arrhythmogenic Cardiomyopathy.
在致心律失常性心肌病小鼠模型中评估 Tideglusib 作为疾病修饰疗法的疗效
Activated mTOR Signaling in the RPE Drives EMT, Autophagy, and Metabolic Disruption, Resulting in AMD-Like Pathology in Mice.
RPE 中 mTOR 信号激活驱动 EMT、自噬和代谢紊乱，导致小鼠出现类似 AMD 的病理
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